Requirement for Ca2+/calmodulin-dependent kinase II in the transition from pressure overload-induced cardiac hypertrophy to heart failure in mice. - Wikidata - wikimedia - TriplyDB

Requirement for Ca2+/calmodulin-dependent kinase II in the transition from pressure overload-induced cardiac hypertrophy to heart failure in mice.

Requirement for Ca2+/calmodulin-dependent kinase II in the transition from pressure overload-induced cardiac hypertrophy to heart failure in mice.

http://www.wikidata.org/entity/Q37170829

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Dysfunctional ryanodine receptors in the heart: new insights into complex cardiovascular diseases Histone deacetylase signaling in cardioprotection Review of novel therapeutic targets for improving heart failure treatment based on experimental and clinical studies Regulation of CaMKII signaling in cardiovascular disease Signaling effectors underlying pathologic growth and remodeling of the heart CaMKII regulation of cardiac K channels Calmodulin-dependent protein kinase II: linking heart failure and arrhythmias Mechanisms of altered Ca²⁺ handling in heart failure New therapeutic targets in cardiology: arrhythmias and Ca2+/calmodulin-dependent kinase II (CaMKII)CaMKIIdelta subtypes: localization and function Targets for therapy in sarcomeric cardiomyopathies Role of sodium and calcium dysregulation in tachyarrhythmias in sudden cardiac death.miR-185 plays an anti-hypertrophic role in the heart via multiple targets in the calcium-signaling pathways AAV-mediated knock-down of HRC exacerbates transverse aorta constriction-induced heart failure Repression of miR-142 by p300 and MAPK is required for survival signalling via gp130 during adaptive hypertrophy The δA isoform of calmodulin kinase II mediates pathological cardiac hypertrophy by interfering with the HDAC4-MEF2 signaling pathway Location matters: clarifying the concept of nuclear and cytosolic CaMKII subtypes CaMKIIδB mediates aberrant NCX1 expression and the imbalance of NCX1/SERCA in transverse aortic constriction-induced failing heart Role of CaMKIIdelta phosphorylation of the cardiac ryanodine receptor in the force frequency relationship and heart failure Diabetes increases mortality after myocardial infarction by oxidizing CaMKII Voltage-Gated Sodium Channel Phosphorylation at Ser571 Regulates Late Current, Arrhythmia, and Cardiac Function In Vivo.Oxidized calmodulin kinase II regulates conduction following myocardial infarction: a computational analysis.Role of RyR2 phosphorylation in heart failure and arrhythmias: Controversies around ryanodine receptor phosphorylation in cardiac disease.Protein Kinase Signaling at the Crossroads of Myocyte Life and Death in Ischemic Heart Disease.Phospholamban ablation rescues sarcoplasmic reticulum Ca(2+) handling but exacerbates cardiac dysfunction in CaMKIIdelta(C) transgenic mice Cardiac Protection of Valsartan on Juvenile Rats with Heart Failure by Inhibiting Activity of CaMKII via Attenuating Phosphorylation.Epac1-dependent phospholamban phosphorylation mediates the cardiac response to stresses CaV1.2 beta-subunit coordinates CaMKII-triggered cardiomyocyte death and afterdepolarizations Electrical remodeling in the failing heart.β-Adrenergic receptor subtype signaling in heart: from bench to bedside.beta-Arrestin-dependent activation of Ca(2+)/calmodulin kinase II after beta(1)-adrenergic receptor stimulation.Aging and CaMKII alter intracellular Ca2+ transients and heart rhythm in Drosophila melanogaster.Intracellular translocation of calmodulin and Ca2+/calmodulin-dependent protein kinase II during the development of hypertrophy in neonatal cardiomyocytes.Loss of MD1 exacerbates pressure overload-induced left ventricular structural and electrical remodelling.Inhibition of CaMKII does not attenuate cardiac hypertrophy in mice with dysfunctional ryanodine receptor CaMKII-dependent phosphorylation of cardiac ryanodine receptors regulates cell death in cardiac ischemia/reperfusion injury.Differential cardiac remodeling in preload versus afterload.Left ventricular and aortic dysfunction in cystic fibrosis mice.Epac2 mediates cardiac β1-adrenergic-dependent sarcoplasmic reticulum Ca2+ leak and arrhythmia.Long-term simulated microgravity causes cardiac RyR2 phosphorylation and arrhythmias in mice.

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Requirement for Ca2+/calmodulin-dependent kinase II in the transition from pressure overload-induced cardiac hypertrophy to heart failure in mice.

http://www.wikidata.org/entity/Q37170829

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 20 April 2009

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Requirement for Ca2+/calmoduli ...... ophy to heart failure in mice.

@en

Requirement for Ca2+/calmoduli ...... ophy to heart failure in mice.

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type

label

Requirement for Ca2+/calmoduli ...... ophy to heart failure in mice.

@en

Requirement for Ca2+/calmoduli ...... ophy to heart failure in mice.

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prefLabel

Requirement for Ca2+/calmoduli ...... ophy to heart failure in mice.

@en

Requirement for Ca2+/calmoduli ...... ophy to heart failure in mice.

@nl

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Journal of Clinical Investigation

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Requirement for Ca2+/calmoduli ...... rophy to heart failure in mice

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Christopher Kable Means

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Haiyun Ling

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Joan Heller Brown

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Ju Chen

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Kirk L Peterson

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Nancy D Dalton

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Tong Zhang

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Yusu Gu

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P2860

PKA phosphorylation dissociates FKBP12.6 from the calcium release channel (ryanodine receptor): defective regulation in failing hearts

CaM kinase II selectively signals to histone deacetylase 4 during cardiomyocyte hypertrophy

Transgenic CaMKIIdeltaC overexpression uniquely alters cardiac myocyte Ca2+ handling: reduced SR Ca2+ load and activated SR Ca2+ release

The deltaC isoform of CaMKII is activated in cardiac hypertrophy and induces dilated cardiomyopathy and heart failure

Protein kinases C and D mediate agonist-dependent cardiac hypertrophy through nuclear export of histone deacetylase 5

Ca2+/calmodulin-dependent protein kinase IIdelta and protein kinase D overexpression reinforce the histone deacetylase 5 redistribution in heart failure.

Identification and expression of delta-isoforms of the multifunctional Ca2+/calmodulin-dependent protein kinase in failing and nonfailing human myocardium

Identification of novel isoforms of the delta subunit of Ca2+/calmodulin-dependent protein kinase II. Differential expression in rat brain and aorta

Ablation of Cypher, a PDZ-LIM domain Z-line protein, causes a severe form of congenital myopathy

Protamine-Cre recombinase transgenes efficiently recombine target sequences in the male germ line of mice, but not in embryonic stem cells

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1230-1240

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scholarly article

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10.1172/JCI38022

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5

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119

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Hongqiang Cheng

Laetitia Pereira

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2009-04-20T00:00:00Z

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19381018

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2673879

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