Central inhibition of interleukin-1beta ameliorates sickness behavior in aged mice.
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Microglial priming and enhanced reactivity to secondary insult in aging, and traumatic CNS injury, and neurodegenerative diseaseIL-1 receptor antagonist attenuates neonatal lipopolysaccharide-induced long-lasting learning impairment and hippocampal injury in adult rats.Neuroinflammation in the normal aging hippocampusAn in vivo animal study assessing long-term changes in hypothalamic cytokines following perinatal exposure to a chemical mixture based on Arctic maternal body burdenInterleukin-1 inhibition and fatigue in primary Sjögren's syndrome--a double blind, randomised clinical trialThe relationship between interleukin-1 receptor antagonist and cognitive function in older adults with bipolar disorder.IL-1RA blocks E. coli-induced suppression of Arc and long-term memory in aged F344xBN F1 ratsDifferential sensitivity to endotoxin exposure in young and middle-age miceTGFβ produced by IL-10 redirected astrocytes attenuates microglial activationConsuming a diet supplemented with resveratrol reduced infection-related neuroinflammation and deficits in working memory in aged miceAging sensitizes rapidly isolated hippocampal microglia to LPS ex vivo.Neurobiological studies of fatigue.Memory impairments in healthy aging: Role of aging-induced microglial sensitization.Fractalkine receptor (CX3CR1) deficiency sensitizes mice to the behavioral changes induced by lipopolysaccharideCerebrospinal fluid markers of neuroinflammation in delirium: a role for interleukin-1β in delirium after hip fractureInhibition of interleukin-6 trans-signaling in the brain facilitates recovery from lipopolysaccharide-induced sickness behaviorMacrophages make me sick: how macrophage activation states influence sickness behaviorEffects of paeonol on anti-neuroinflammatory responses in microglial cellsDietary broccoli mildly improves neuroinflammation in aged mice but does not reduce lipopolysaccharide-induced sickness behaviorPeripheral inflammatory disease associated with centrally activated IL-1 system in humans and miceMicroglial aging in the healthy CNS: phenotypes, drivers, and rejuvenationEffects of voluntary wheel running on LPS-induced sickness behavior in aged mice.Aging leads to prolonged duration of inflammation-induced depression-like behavior caused by Bacillus Calmette-Guérin.Interleukin-6 trans-signaling in the senescent mouse brain is involved in infection-related deficits in contextual fear conditioning.Controlled progressive innate immune stimulation regimen prevents the induction of sickness behavior in the open field test.Influence of age on behavioral, immune and endocrine responses to the T-cell superantigen staphylococcal enterotoxin A.Progress and prospects: Immunobiology of gene therapy for neurodegenerative disease: prospects and risks.Review: microglia of the aged brain: primed to be activated and resistant to regulation.Feeding the beast: can microglia in the senescent brain be regulated by diet?Neuroinflammation: the devil is in the details.Luteolin inhibits microglia and alters hippocampal-dependent spatial working memory in aged mice.Anthocyanins control neuroinflammation and consequent memory dysfunction in mice exposed to lipopolysaccharide.Spatial Learning Responses to Lipopolysaccharide in Adult and Aged Rats.Central inhibition of interleukin-6 trans-signaling during peripheral infection reduced neuroinflammation and sickness in aged mice.Effects of low-dose lipopolysaccharide and age on spatial learning in different Morris water maze protocols.Dietary Luteolin Reduces Proinflammatory Microglia in the Brain of Senescent MiceIL-1RA injected intra-cisterna magna confers extended prophylaxis against lipopolysaccharide-induced neuroinflammatory and sickness responses.Immune and behavioral consequences of microglial reactivity in the aged brain.Differential response to intrahippocampal interleukin-4/interleukin-13 in aged and exercise mice.Different Sickness Responses in Adult and Aged Rats Following Lipopolysaccharide Administration.
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P2860
Central inhibition of interleukin-1beta ameliorates sickness behavior in aged mice.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 31 December 2008
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vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
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name
Central inhibition of interleukin-1beta ameliorates sickness behavior in aged mice.
@en
Central inhibition of interleukin-1beta ameliorates sickness behavior in aged mice.
@nl
type
label
Central inhibition of interleukin-1beta ameliorates sickness behavior in aged mice.
@en
Central inhibition of interleukin-1beta ameliorates sickness behavior in aged mice.
@nl
prefLabel
Central inhibition of interleukin-1beta ameliorates sickness behavior in aged mice.
@en
Central inhibition of interleukin-1beta ameliorates sickness behavior in aged mice.
@nl
P2860
P1476
Central inhibition of interleukin-1beta ameliorates sickness behavior in aged mice
@en
P2093
Jayne Abraham
P2860
P304
P356
10.1016/J.BBI.2008.12.008
P577
2008-12-31T00:00:00Z