The monocyte chemoattractant protein-1/CCR2 loop, inducible by TGF-beta, increases podocyte motility and albumin permeability.
about
Effects of Tumor Necrosis Factor-α on Podocyte Expression of Monocyte Chemoattractant Protein-1 and in Diabetic NephropathyNeurogenesis-Promoting Natural Product α-Asarone Modulates Morphological Dynamics of Activated Microglia.The MCP-1/CCR2 axis in podocytes is involved in apoptosis induced by diabetic conditions.Trophoblast glycoprotein: possible candidate mediating podocyte injuries in glomerulonephritisAlbumin-induced podocyte injury and protection are associated with regulation of COX-2.Targeted GAS6 delivery to the CNS protects axons from damage during experimental autoimmune encephalomyelitisTNFR2 interposes the proliferative and NF-κB-mediated inflammatory response by podocytes to TNF-α.Sesquiterpene lactones inhibit advanced oxidation protein product-induced MCP-1 expression in podocytes via an IKK/NF-κB-dependent mechanism.TWEAK/Fn14 system and crescent formation in IgA nephropathy.AMPK mediates the initiation of kidney disease induced by a high-fat dietMechanisms and consequences of TGF-ß overexpression by podocytes in progressive podocyte disease.Lipopolysaccharide induces inducible nitric oxide synthase-dependent podocyte dysfunction via a hypoxia-inducible factor 1α and cell division control protein 42 and Ras-related C3 botulinum toxin substrate 1 pathway.Renal Lipotoxicity-Associated Inflammation and Insulin Resistance Affects Actin Cytoskeleton Organization in PodocytesPentoxifylline Attenuates Proteinuria in Anti-Thy1 Glomerulonephritis via Downregulation of Nuclear Factor-κB and Smad2/3 Signaling.Accelerated renal disease is associated with the development of metabolic syndrome in a glucolipotoxic mouse model.Visualizing the mouse podocyte with multiphoton microscopy.Transforming growth factor-β1 and diabetic nephropathyHaptoglobin and CCR2 receptor expression in ovarian cancer cells that were exposed to ascitic fluid: exploring a new role of haptoglobin in the tumoral microenvironmentTriptolide markedly attenuates albuminuria and podocyte injury in an animal model of diabetic nephropathy.The proteoglycan syndecan 4 regulates transient receptor potential canonical 6 channels via RhoA/Rho-associated protein kinase signaling.Macrophages directly mediate diabetic renal injury.Abnormalities in signaling pathways in diabetic nephropathy.Interleukin-20 targets podocytes and is upregulated in experimental murine diabetic nephropathy.Targeting chemokines in proteinuria-induced renal disease.Role of chemokines in proteinuric kidney disorders.Inflammation in diabetic nephropathy: moving toward clinical biomarkers and targets for treatment.Absence of miR-146a in Podocytes Increases Risk of Diabetic Glomerulopathy via Up-regulation of ErbB4 and Notch-1.When the endothelium scores an own goal: endothelial cells actively augment metastatic extravasation through endothelial-mesenchymal transition.Immunity and inflammation in diabetic kidney disease: translating mechanisms to biomarkers and treatment targets.Crosstalk between protective autophagy and NF-κB signal in high glucose-induced podocytes.Effects of ferulic acid on diabetic nephropathy in a rat model of type 2 diabetes.Effects of losartan on expression of monocyte chemoattractant protein-1 (MCP-1) in hyperuricemic nephropathy rats.TGF-beta in the pathogenesis and prevention of disease: a matter of aneurysmic proportions.Statins reverse renal inflammation and endothelial dysfunction induced by chronic high salt intake.Inhibition of type I interferon signalling prevents TLR ligand-mediated proteinuria.Blockade of CCL2/CCR2 signalling ameliorates diabetic nephropathy in db/db mice.Valproate hampers podocyte acquisition of immune phenotypes via intercepting the GSK3β facilitated NFkB activation.CONTRIBUTION OF MYO-INOSITOL OXYGENASE IN AGE: RAGE MEDIATED RENAL TUBULO-INTERSTITIAL INJURY IN THE CONTEXT OF DIABETIC NEPHROPATHY.Combination of cilostazol and probucol protected podocytes from lipopolysaccharide-induced injury by both anti-inflammatory and anti-oxidative mechanisms.High content screening assay based discovery of paullones as novel podocyte protective agents.
P2860
Q28080888-234C9A46-DAC9-4FD2-BEEE-315CB2406A94Q30831348-3409A6EA-3F06-4EB6-B6D9-079AE172AEBFQ34050210-169EE57B-1D00-4DD1-9807-DB8D3C237A3CQ34354544-C22A75EE-2CB3-4735-8115-0AF60C1CF6FEQ34577309-76509F5E-3769-42C9-902F-67371C6431C9Q34614563-BC590FA9-DE4A-4B01-99D1-362944E23728Q34788604-E6F0BAF0-2BC1-4FF9-9B6F-BC0BD9910ACFQ35022648-209CEB0D-A123-467B-B4CB-D906E6344E0AQ35187418-123352F6-0A1E-49FB-B5BE-1A1F3968125BQ35272259-6715ED74-8819-4BEC-8676-B04A3B45DFBBQ35649310-BCEF9785-9ECE-4F45-B019-CAD57250E5DBQ35688862-7A410030-8FD7-4A34-9640-C79715EDA50FQ35834459-DA5F0279-975E-4E6C-9C60-49038DAFFC32Q35855665-B1CB0EBD-48DB-4D61-AB49-1FB1972D224DQ36179961-C389734C-1338-4C3E-B6A2-A51A6C223795Q36456525-2EFAA497-09C1-4D9E-8928-68E086CD65ADQ36810701-96ED2500-8009-4E31-8CA1-B3F3F8CFBB3EQ37113471-A4393C7A-080D-4CA2-852B-979037E92E82Q37207268-F58DE670-CE13-420F-862C-94ED00A7851DQ37436623-DCB3AC7D-F601-4F5C-8D4E-539C96C20FA1Q37440181-88FD680D-2C4B-435C-BE10-0337358A85A4Q37708046-E3AAEA62-BAC9-4ABA-8FF1-4D36164D5DD6Q37739475-133AD914-B2CF-42A3-B4CF-5E51BCE766C6Q38026076-295BF1E3-2E92-416A-B10F-8DFFAC1C5F93Q38188747-178E215C-00E6-4D28-8041-0FB91B664239Q38255902-8910754F-C2CD-419E-A948-4287CCE5C449Q38289926-12F97B15-EF3A-43D5-9507-B205C0DCA37CQ38780879-81B6252B-E879-46B4-AF9F-422D4501B082Q38936622-81D88775-84F3-47F4-8DBD-40EEC1C2FB70Q38981875-9369D742-90EC-4F66-8A2C-35BEF2BC8F1FQ40335091-FFE732C3-A34E-4275-938F-461C79023928Q41364208-DDD1ED3D-05C6-46EF-B93B-E30DEC76F0ABQ42059264-62D46DA9-37ED-4292-9172-C8282F519BFCQ42487652-FE9124DB-3414-4903-A9C7-D3311319E41EQ44301814-9B1A9D33-91F6-4CDE-BB10-D74FDFFD6427Q45232179-95AF5793-B0BC-47BF-807A-5FA2393ACF7DQ45385733-CDBBBF0E-09AE-4DBC-9D92-EEF69A2636EEQ46303824-B64B30C3-3828-4642-898F-2C54BE934C8CQ46441740-1AEDFC21-6058-4618-95E0-2DC1385D69CDQ47632212-B4C9EAA6-276F-4661-A9EF-AB77A13AF11E
P2860
The monocyte chemoattractant protein-1/CCR2 loop, inducible by TGF-beta, increases podocyte motility and albumin permeability.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 06 May 2009
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
The monocyte chemoattractant p ...... lity and albumin permeability.
@en
The monocyte chemoattractant p ...... lity and albumin permeability.
@nl
type
label
The monocyte chemoattractant p ...... lity and albumin permeability.
@en
The monocyte chemoattractant p ...... lity and albumin permeability.
@nl
prefLabel
The monocyte chemoattractant p ...... lity and albumin permeability.
@en
The monocyte chemoattractant p ...... lity and albumin permeability.
@nl
P2093
P2860
P1476
The monocyte chemoattractant p ...... ility and albumin permeability
@en
P2093
Choon Hee Chung
Eun Young Lee
Petr E Pyagay
Sheldon Chen
Tet Kin Yeo
P2860
P304
P356
10.1152/AJPRENAL.90642.2008
P577
2009-05-06T00:00:00Z