Alterations in ionotropic glutamate receptor subunits during binge cocaine self-administration and withdrawal in rats.
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Glutamatergic substrates of drug addiction and alcoholismInhibition of cyclooxygenase-2 by NS398 attenuates noise-induced hearing loss in mice.Animal models of substance abuse and addiction: implications for science, animal welfare, and societyStability of surface NMDA receptors controls synaptic and behavioral adaptations to amphetamine.Integrative proteomic analysis of the nucleus accumbens in rhesus monkeys following cocaine self-administrationCytosolic proteomic alterations in the nucleus accumbens of cocaine overdose victimsCocaine-induced neuroadaptations in glutamate transmission: potential therapeutic targets for craving and addictionUpregulation of Arc mRNA expression in the prefrontal cortex following cue-induced reinstatement of extinguished cocaine-seeking behavior.Assessment of genome and proteome profiles in cocaine abuseAMPA receptor plasticity in the nucleus accumbens after repeated exposure to cocaine.Bidirectional effects of inhibiting or potentiating NMDA receptors on extinction after cocaine self-administration in ratsDifferential glutamate AMPA-receptor plasticity in subpopulations of VTA neurons in the presence or absence of residual cocaine: implications for the development of addiction.Suppression of activity-regulated cytoskeleton-associated gene expression in the dorsal striatum attenuates extinction of cocaine-seekingChanges in levels of D1, D2, or NMDA receptors during withdrawal from brief or extended daily access to IV cocaineCocainomics: new insights into the molecular basis of cocaine addiction.Ethanol self-administration modulation of NMDA receptor subunit and related synaptic protein mRNA expression in prefrontal cortical fields in cynomolgus monkeysFos and glutamate AMPA receptor subunit coexpression associated with cue-elicited cocaine-seeking behavior in abstinent rats.Extinction training regulates neuroadaptive responses to withdrawal from chronic cocaine self-administration.Extinction-induced neuroplasticity attenuates stress-induced cocaine seeking: a state-dependent learning hypothesis.Dopamine and glutamate release in the dorsolateral caudate putamen following withdrawal from cocaine self-administration in rats.GDNF and addiction.Region-specific involvement of AMPA/Kainate receptors in Fos protein expression induced by cocaine-conditioned cuesCocaine but not natural reward self-administration nor passive cocaine infusion produces persistent LTP in the VTA.Phosphorylation-dependent trafficking of GluR2-containing AMPA receptors in the nucleus accumbens plays a critical role in the reinstatement of cocaine seeking.Deficits in ventromedial prefrontal cortex group 1 metabotropic glutamate receptor function mediate resistance to extinction during protracted withdrawal from an extensive history of cocaine self-administrationContribution of ventral tegmental GABA receptors to cocaine self-administration in ratsNeuroplasticity in the mesolimbic dopamine system and cocaine addictionShort and long access to cocaine self-administration activates tyrosine phosphatase STEP and attenuates GluN expression but differentially regulates GluA expression in the prefrontal cortex.Persistent proteomic alterations in the medial prefrontal cortex with abstinence from cocaine self-administration.Differential regulation of ionotropic glutamate receptor subunits following cocaine self-administration.Continuous exposure to dizocilpine facilitates escalation of cocaine consumption in male Sprague-Dawley rats.Disruption of glutamate receptor-interacting protein in nucleus accumbens enhances vulnerability to cocaine relapse.Cocaine-induced metaplasticity in the nucleus accumbens: silent synapse and beyond.Glutamatergic synaptic plasticity in the mesocorticolimbic system in addictionGlutamatergic plasticity in medial prefrontal cortex and ventral tegmental area following extended-access cocaine self-administration.Extended daily access to cocaine results in distinct alterations in Homer 1b/c and NMDA receptor subunit expression within the medial prefrontal cortex.Contributions of prolonged contingent and non-contingent cocaine exposure to escalation of cocaine intake and glutamatergic gene expression.Disrupting GluA2 phosphorylation potentiates reinstatement of cocaine seeking.AMPA-receptor GluR1 subunits are involved in the control over behavior by cocaine-paired cues.Increased corticolimbic connectivity in cocaine dependence versus pathological gambling is associated with drug severity and emotion-related impulsivity.
P2860
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P2860
Alterations in ionotropic glutamate receptor subunits during binge cocaine self-administration and withdrawal in rats.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on May 2004
@en
vedecký článok
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vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
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name
Alterations in ionotropic glut ...... ration and withdrawal in rats.
@en
Alterations in ionotropic glut ...... ration and withdrawal in rats.
@nl
type
label
Alterations in ionotropic glut ...... ration and withdrawal in rats.
@en
Alterations in ionotropic glut ...... ration and withdrawal in rats.
@nl
prefLabel
Alterations in ionotropic glut ...... ration and withdrawal in rats.
@en
Alterations in ionotropic glut ...... ration and withdrawal in rats.
@nl
P2860
P50
P1476
Alterations in ionotropic glut ...... tration and withdrawal in rats
@en
P2093
Bill Liang
Wenxue Tang
P2860
P304
P356
10.1111/J.1471-4159.2004.02392.X
P407
P577
2004-05-01T00:00:00Z