Potent induction of IFN-alpha and chemokines by autoantibodies in the cerebrospinal fluid of patients with neuropsychiatric lupus. - Wikidata - wikimedia - TriplyDB

Potent induction of IFN-alpha and chemokines by autoantibodies in the cerebrospinal fluid of patients with neuropsychiatric lupus.

Potent induction of IFN-alpha and chemokines by autoantibodies in the cerebrospinal fluid of patients with neuropsychiatric lupus.

http://www.wikidata.org/entity/Q37351240

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Management of Neuropsychiatric Systemic Lupus Erythematosus: Current Approaches and Future Perspectives Biomarkers for CNS involvement in pediatric lupus Pathogenic Inflammation and Its Therapeutic Targeting in Systemic Lupus Erythematosus Lupus brain fog: a biologic perspective on cognitive impairment, depression, and fatigue in systemic lupus erythematosus Anti-U1 RNP antibodies in cerebrospinal fluid are associated with central neuropsychiatric manifestations in systemic lupus erythematosus and mixed connective tissue disease Neuropsychiatric Lupus, the Blood Brain Barrier, and the TWEAK/Fn14 Pathway.Cytokines and chemokines in neuropsychiatric syndromes of systemic lupus erythematosus.Glutamate receptor biology and its clinical significance in neuropsychiatric systemic lupus erythematosus.Early, transient depletion of plasmacytoid dendritic cells ameliorates autoimmunity in a lupus model.Activation of type I interferon pathway in systemic lupus erythematosus: association with distinct clinical phenotypes.A candidate gene study of the type I interferon pathway implicates IKBKE and IL8 as risk loci for SLE.C1q deficiency leads to the defective suppression of IFN-alpha in response to nucleoprotein containing immune complexes.Association of IRF5 polymorphisms with activation of the interferon alpha pathway Neuroimaging evidence of white matter inflammation in newly diagnosed systemic lupus erythematosus.The type I interferon system in the etiopathogenesis of autoimmune diseases.Type I interferon and systemic lupus erythematosus.Cutting edge: Antimalarial drugs inhibit IFN-β production through blockade of cyclic GMP-AMP synthase-DNA interaction.Plasmacytoid dendritic cells and C1q differentially regulate inflammatory gene induction by lupus immune complexes.Sensors of the innate immune system: their link to rheumatic diseases.TNF-α and TGF-β counter-regulate PD-L1 expression on monocytes in systemic lupus erythematosus.Roles of pro- and anti-inflammatory cytokines in the pathogenesis of SLE Behavioral Deficits Are Accompanied by Immunological and Neurochemical Changes in a Mouse Model for Neuropsychiatric Lupus (NP-SLE).Neuropsychiatric disease in murine lupus is dependent on the TWEAK/Fn14 pathway.Biomarkers in systemic lupus erythematosus: challenges and prospects for the future.Developments in the clinical understanding of lupus The impact and implications of neuropsychiatric systemic lupus erythematosus in adolescents.Genetic manipulation of the ApoF/Stat2 locus supports an important role for type I interferon signaling in atherosclerosis.Neuropsychiatric Systemic Lupus Erythematosus Involvement: Towards a Tailored Approach to Our Patients?Potential role of IFNα in adult lupus.Cytokines and their roles in the pathogenesis of systemic lupus erythematosus: from basics to recent advances.The role of cytokines in the pathogenesis of systemic lupus erythematosus - from bench to bedside.Immune dysregulation and autoimmunity in bipolar disorder: Synthesis of the evidence and its clinical application.Diagnosis and management of neuropsychiatric SLE.Interferons in the central nervous system: a few instruments play many tunes.Complement, c1q, and c1q-related molecules regulate macrophage polarization.Neuropsychiatric systemic lupus erythematosus: pathogenesis and biomarkers.A Plasmacytoid Dendritic Cells-Type I Interferon Axis Is Critically Implicated in the Pathogenesis of Systemic Lupus Erythematosus.Microglia-dependent synapse loss in type I interferon-mediated lupus.Modulation of Neuroinflammation in the Central Nervous System: Role of Chemokines and Sphingolipids.Role of IL-1β, IL-6, IL-8 and IFN-γ in pathogenesis of central nervous system neuropsychiatric systemic lupus erythematous.

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Potent induction of IFN-alpha and chemokines by autoantibodies in the cerebrospinal fluid of patients with neuropsychiatric lupus.

http://www.wikidata.org/entity/Q37351240

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on January 2009

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Potent induction of IFN-alpha ...... s with neuropsychiatric lupus.

@en

Potent induction of IFN-alpha ...... s with neuropsychiatric lupus.

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type

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Potent induction of IFN-alpha ...... s with neuropsychiatric lupus.

@en

Potent induction of IFN-alpha ...... s with neuropsychiatric lupus.

@nl

prefLabel

Potent induction of IFN-alpha ...... s with neuropsychiatric lupus.

@en

Potent induction of IFN-alpha ...... s with neuropsychiatric lupus.

@nl

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JIMMUNOL.182.2.1192

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Journal of Immunology

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Potent induction of IFN-alpha ...... s with neuropsychiatric lupus.

@en

P2093

Deanna M Santer

http://www.w3.org/2001/XMLSchema#string

Keith B Elkon

http://www.w3.org/2001/XMLSchema#string

Seiji Minota

http://www.w3.org/2001/XMLSchema#string

Taku Yoshio

http://www.w3.org/2001/XMLSchema#string

Thomas Möller

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P2860

BDCA-2, a novel plasmacytoid dendritic cell-specific type II C-type lectin, mediates antigen capture and is a potent inhibitor of interferon alpha/beta induction

The antiinflammatory activity of IgG: the intravenous IgG paradox

Mutations in the gene encoding the 3'-5' DNA exonuclease TREX1 cause Aicardi-Goutières syndrome at the AGS1 locus

B-cell depletion with rituximab in relapsing-remitting multiple sclerosis

Anti-inflammatory actions of intravenous immunoglobulin

The 1982 revised criteria for the classification of systemic lupus erythematosus

A study of 20 SLE patients with intravenous immunoglobulin--clinical and serologic response.

Structural and functional neuropathology in transgenic mice with CNS expression of IFN-alpha.

Mutations in genes encoding ribonuclease H2 subunits cause Aicardi-Goutières syndrome and mimic congenital viral brain infection.

Toll-like receptor 7 and TLR9 dictate autoantibody specificity and have opposing inflammatory and regulatory roles in a murine model of lupus.

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1192-1201

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scholarly article

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10.4049/JIMMUNOL.182.2.1192

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2009-01-01T00:00:00Z

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23759805

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19124763

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cerebrospinal fluid

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