The effects of NOS2 gene deletion on mice expressing mutated human AbetaPP.
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Innate immunity in Alzheimer's disease: a complex affairArginine deprivation and immune suppression in a mouse model of Alzheimer's diseaseCombined linkage and linkage disequilibrium analysis of a motor speech phenotype within families ascertained for autism risk loci.Triterpenoid CDDO-methylamide improves memory and decreases amyloid plaques in a transgenic mouse model of Alzheimer's disease.Current neuroimaging techniques in Alzheimer's disease and applications in animal modelsElevated copper in the amyloid plaques and iron in the cortex are observed in mouse models of Alzheimer's disease that exhibit neurodegenerationmNos2 deletion and human NOS2 replacement in Alzheimer disease modelsAccelerating drug discovery for Alzheimer's disease: best practices for preclinical animal studiesThe impact of human and mouse differences in NOS2 gene expression on the brain's redox and immune environment.Neuroprotective strategies involving ROS in Alzheimer disease.Introducing Human APOE into Aβ Transgenic Mouse Models.The classification of microglial activation phenotypes on neurodegeneration and regeneration in Alzheimer's disease brain.Exendin-4 protected against cognitive dysfunction in hyperglycemic mice receiving an intrahippocampal lipopolysaccharide injection.A transgenic Alzheimer rat with plaques, tau pathology, behavioral impairment, oligomeric aβ, and frank neuronal lossSunitinib enhances neuronal survival in vitro via NF-κB-mediated signaling and expression of cyclooxygenase-2 and inducible nitric oxide synthase.Endothelial nitric oxide deficiency promotes Alzheimer's disease pathologyEffects of aged garlic extract and FruArg on gene expression and signaling pathways in lipopolysaccharide-activated microglial cells.Longitudinal study of differential protein expression in an Alzheimer's mouse model lacking inducible nitric oxide synthase.The interplay between iron accumulation, mitochondrial dysfunction, and inflammation during the execution step of neurodegenerative disorders.TDP-43 expression influences amyloidβ plaque deposition and tau aggregation.Innate immunity in Alzheimer's disease: the relevance of animal models?Nitric oxide signaling is recruited as a compensatory mechanism for sustaining synaptic plasticity in Alzheimer's disease mice.Amyloid reduction by amyloid-beta vaccination also reduces mouse tau pathology and protects from neuron loss in two mouse models of Alzheimer's disease.Constructing an integrated gene similarity network for the identification of disease genes.Use of Eflornithine (DFMO) in the Treatment of Early Alzheimer's Disease: A Compassionate Use, Single-Case Study.
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P2860
The effects of NOS2 gene deletion on mice expressing mutated human AbetaPP.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on December 2008
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
The effects of NOS2 gene deletion on mice expressing mutated human AbetaPP.
@en
The effects of NOS2 gene deletion on mice expressing mutated human AbetaPP.
@nl
type
label
The effects of NOS2 gene deletion on mice expressing mutated human AbetaPP.
@en
The effects of NOS2 gene deletion on mice expressing mutated human AbetaPP.
@nl
prefLabel
The effects of NOS2 gene deletion on mice expressing mutated human AbetaPP.
@en
The effects of NOS2 gene deletion on mice expressing mutated human AbetaPP.
@nl
P2093
P2860
P1476
The effects of NOS2 gene deletion on mice expressing mutated human AbetaPP.
@en
P2093
David A Wink
Donna M Wilcock
Judianne Davis
Michael P Vitek
William E Van Nostrand
P2860
P304
P577
2008-12-01T00:00:00Z