Pkd2 dosage influences cellular repair responses following ischemia-reperfusion injury. - Wikidata - wikimedia - TriplyDB

Pkd2 dosage influences cellular repair responses following ischemia-reperfusion injury.

Pkd2 dosage influences cellular repair responses following ischemia-reperfusion injury.

http://www.wikidata.org/entity/Q37362069

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A polycystin-centric view of cyst formation and disease: the polycystins revisited Regulation of STATs by polycystin-1 and their role in polycystic kidney disease Autophagy and regulation of cilia function and assembly Macrophages promote polycystic kidney disease progression.Cyst formation in the PKD2 (1-703) transgenic rat precedes deregulation of proliferation-related pathways The cleaved cytoplasmic tail of polycystin-1 regulates Src-dependent STAT3 activation.The cell biology of polycystic kidney disease Protein kinase D-mediated phosphorylation of polycystin-2 (TRPP2) is essential for its effects on cell growth and calcium channel activity.Kidney: polycystic kidney disease.Filamin-a increases the stability and plasma membrane expression of polycystin-2.Elevated expression of mechanosensory polycystins in human carotid atherosclerotic plaques: association with p53 activation and disease severity G-protein signaling modulator 1 deficiency accelerates cystic disease in an orthologous mouse model of autosomal dominant polycystic kidney disease.Pkd2 mesenteric vessels exhibit a primary defect in endothelium-dependent vasodilatation restored by rosiglitazone.ERK-mediated suppression of cilia in cisplatin-induced tubular cell apoptosis and acute kidney injury.Primary cilia and kidney injury: current research status and future perspectives Autocrine IL-10 activation of the STAT3 pathway is required for pathological macrophage differentiation in polycystic kidney disease Molecular and cellular pathogenesis of autosomal dominant polycystic kidney disease.Genetics and pathogenesis of autosomal dominant polycystic kidney disease: 20 years on.Inflammation and Fibrosis in Polycystic Kidney Disease.Variable Cyst Development in Autosomal Dominant Polycystic Kidney Disease: The Biologic Context.Phosphoinositide 3-Kinase-C2α Regulates Polycystin-2 Ciliary Entry and Protects against Kidney Cyst Formation.Acute Kidney Injury in ADPKD Patients with Pneumonia.Environmental hit on a genetic basis in polycystic kidney disease.Acute kidney injury induces hallmarks of polycystic kidney disease.Targeting new cellular disease pathways in autosomal dominant polycystic kidney disease.

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Pkd2 dosage influences cellular repair responses following ischemia-reperfusion injury.

http://www.wikidata.org/entity/Q37362069

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 03 September 2009

@en

vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

@da

vědecký článek

@cs

name

Pkd2 dosage influences cellular repair responses following ischemia-reperfusion injury.

@en

Pkd2 dosage influences cellular repair responses following ischemia-reperfusion injury.

@nl

type

label

Pkd2 dosage influences cellular repair responses following ischemia-reperfusion injury.

@en

Pkd2 dosage influences cellular repair responses following ischemia-reperfusion injury.

@nl

prefLabel

Pkd2 dosage influences cellular repair responses following ischemia-reperfusion injury.

@en

Pkd2 dosage influences cellular repair responses following ischemia-reperfusion injury.

@nl

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AJPATH.2009.090227

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The American Journal of Pathology

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Pkd2 dosage influences cellular repair responses following ischemia-reperfusion injury.

@en

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Albert Chee Meng Ong

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Frederick Wai Keung Tam

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John Lionel Haylor

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John Patrick McDaid

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Sony Prasad

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P2860

Renal cystic disease in tuberous sclerosis: role of the polycystic kidney disease 1 gene

Cardiac defects and renal failure in mice with targeted mutations in Pkd2

Polycystin-2 expression is increased following experimental ischaemic renal injury

PKD1 induces p21(waf1) and regulation of the cell cycle via direct activation of the JAK-STAT signaling pathway in a process requiring PKD2

Pkd2 haploinsufficiency alters intracellular calcium regulation in vascular smooth muscle cells

CXCR2 agonists in ADPKD liver cyst fluids promote cell proliferation.

Progressive loss of renal function is an age-dependent heritable trait in type 1 autosomal dominant polycystic kidney disease.

Modifier genes play a significant role in the phenotypic expression of PKD1.

Defining a link with autosomal-dominant polycystic kidney disease in mice with congenitally low expression of Pkd1

Acute kidney injury and aberrant planar cell polarity induce cyst formation in mice lacking renal cilia.

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1493-1503

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scholarly article

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10.2353/AJPATH.2009.090227

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2009-09-03T00:00:00Z

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26788693

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19729489

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reperfusion injury

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2751546

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