Acute kidney injury and aberrant planar cell polarity induce cyst formation in mice lacking renal cilia. - Wikidata - wikimedia - TriplyDB

Acute kidney injury and aberrant planar cell polarity induce cyst formation in mice lacking renal cilia.

Acute kidney injury and aberrant planar cell polarity induce cyst formation in mice lacking renal cilia.

http://www.wikidata.org/entity/Q35148415

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Uromodulin is expressed in renal primary cilia and UMOD mutations result in decreased ciliary uromodulin expression The primary cilium as a cellular signaling center: lessons from disease Autosomal dominant polycystic kidney disease: the last 3 years Role of the Polycystins in Cell Migration, Polarity, and Tissue Morphogenesis Polycystin-1: a master regulator of intersecting cystic pathways The kidney and planar cell polarity.The role of the cilium in normal and abnormal cell cycles: emphasis on renal cystic pathologies Kif3a guides microtubular dynamics, migration and lumen formation of MDCK cells New frontiers: discovering cilia-independent functions of cilia proteins p120 catenin is required for normal renal tubulogenesis and glomerulogenesis Impaired Wnt-beta-catenin signaling disrupts adult renal homeostasis and leads to cystic kidney ciliopathy Role of epidermal primary cilia in the homeostasis of skin and hair follicles Wnt9b signaling regulates planar cell polarity and kidney tubule morphogenesis Polycystin-dependent fluid flow sensing targets histone deacetylase 5 to prevent the development of renal cysts Systems biology approach to identify transcriptome reprogramming and candidate microRNA targets during the progression of polycystic kidney disease Disruption of IFT complex A causes cystic kidneys without mitotic spindle misorientation A genetic interaction network of five genes for human polycystic kidney and liver diseases defines polycystin-1 as the central determinant of cyst formation Polycystin-2 and phosphodiesterase 4C are components of a ciliary A-kinase anchoring protein complex that is disrupted in cystic kidney diseases Deletion of IFT20 in the mouse kidney causes misorientation of the mitotic spindle and cystic kidney disease Planar cell polarity signaling: from fly development to human disease Ciliopathies The primary cilium as a complex signaling center Spindle pole cohesion requires glycosylation-mediated localization of NuMA.Dicer regulates the development of nephrogenic and ureteric compartments in the mammalian kidney.Loss of oriented cell division does not initiate cyst formation The zebrafish foxj1a transcription factor regulates cilia function in response to injury and epithelial stretch A mitotic transcriptional switch in polycystic kidney disease Carcinogens induce loss of the primary cilium in human renal proximal tubular epithelial cells independently of effects on the cell cycle Chemical modifier screen identifies HDAC inhibitors as suppressors of PKD models.Aberrant planar cell polarity induced by urinary tract obstruction.Pericentrin in cellular function and disease Macrophages promote polycystic kidney disease progression.Advances in the pathogenesis and treatment of polycystic kidney disease Functional Analysis of KIF3A and KIF3B during Spermiogenesis of Chinese Mitten Crab Eriocheir sinensis.Cyst formation in the PKD2 (1-703) transgenic rat precedes deregulation of proliferation-related pathways Molecular advances in autosomal dominant polycystic kidney disease.A pilot clinical study to evaluate changes in urine osmolality and urine cAMP in response to acute and chronic water loading in autosomal dominant polycystic kidney disease.Reduced Notch signaling leads to renal cysts and papillary microadenomas.Treatment strategies and clinical trial design in ADPKD Glomerulocystic kidney disease

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P2860

Acute kidney injury and aberrant planar cell polarity induce cyst formation in mice lacking renal cilia.

http://www.wikidata.org/entity/Q35148415

description

2008 nî lūn-bûn

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2008 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2008 թվականի փետրվարին հրատարակված գիտական հոդված

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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name

Acute kidney injury and aberra ...... n in mice lacking renal cilia.

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Acute kidney injury and aberra ...... n in mice lacking renal cilia.

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type

label

Acute kidney injury and aberra ...... n in mice lacking renal cilia.

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Acute kidney injury and aberra ...... n in mice lacking renal cilia.

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Acute kidney injury and aberra ...... n in mice lacking renal cilia.

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Acute kidney injury and aberra ...... n in mice lacking renal cilia.

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P1433

Human Molecular Genetics

P1476

Acute kidney injury and aberra ...... on in mice lacking renal cilia

@en

P2093

Fangming Lin

http://www.w3.org/2001/XMLSchema#string

Ling Li

http://www.w3.org/2001/XMLSchema#string

Patricia Cobo-Stark

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Stefan Somlo

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Xinli Shao

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P2860

The Ciliopathies: An Emerging Class of Human Genetic Disorders

Cre reporter strains produced by targeted insertion of EYFP and ECFP into the ROSA26 locus

Cilia and developmental signaling

Polycystins 1 and 2 mediate mechanosensation in the primary cilium of kidney cells

Left-right asymmetry and kinesin superfamily protein KIF3A: new insights in determination of laterality and mesoderm induction by kif3A-/- mice analysis

Situs inversus and embryonic ciliary morphogenesis defects in mouse mutants lacking the KIF3A subunit of kinesin-II

Mutation of hepatocyte nuclear factor-1beta inhibits Pkhd1 gene expression and produces renal cysts in mice

The Oak Ridge Polycystic Kidney (orpk) disease gene is required for left-right axis determination

Inversin, the gene product mutated in nephronophthisis type II, functions as a molecular switch between Wnt signaling pathways

Polyductin undergoes notch-like processing and regulated release from primary cilia

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1578-1590

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10.1093/HMG/DDN045

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11

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17

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2008-02-09T00:00:00Z

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5588044

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18263895

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3150596

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