Transgenic expression of replication-restricted enteroviral genomes in heart muscle induces defective excitation-contraction coupling and dilated cardiomyopathy.
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Molecular mimicry, bystander activation, or viral persistence: infections and autoimmune disease.c-FLIP-Short reduces type I interferon production and increases viremia with coxsackievirus B3Viral myocarditis and dilated cardiomyopathy: mechanisms, manifestations, and managementPersistent RNA virus infections: do PAMPS drive chronic disease?Structural interplay of KV7.1 and KCNE1 is essential for normal repolarization and is compromised in short QT syndrome 2 (KV7.1-A287T).5'-Terminal deletions occur in coxsackievirus B3 during replication in murine hearts and cardiac myocyte cultures and correlate with encapsidation of negative-strand viral RNA.Molecular mechanisms of coxsackievirus persistence in chronic inflammatory myopathy: viral RNA persists through formation of a double-stranded complex without associated genomic mutations or evolutionElectrophysiological alterations in a murine model of chronic coxsackievirus B3 myocarditisExpression of immunoregulatory cytokines by recombinant coxsackievirus B3 variants confers protection against virus-caused myocarditisMeeting Koch's postulates for calcium signaling in cardiac hypertrophy.Cellular and functional defects in a mouse model of heart failure.Activation of NF-kappaB by double-stranded RNA (dsRNA) in the absence of protein kinase R and RNase L demonstrates the existence of two separate dsRNA-triggered antiviral programsCardiac hypertrophy and failure: lessons learned from genetically engineered mice.Animal models of cardiac disease and stem cell therapy.Cardiac dysfunction in pauci symptomatic human immunodeficiency virus patients: a meta-analysis in the highly active antiretroviral therapy era.Type B coxsackieviruses and their interactions with the innate and adaptive immune systems.High prevalence of enteroviral genomic sequences in myocardium from cases of endemic cardiomyopathy (Keshan disease) in China.Myocarditis, microbes and autoimmunity.Recent progress in understanding coxsackievirus replication, dissemination, and pathogenesis.Host and virus determinants of picornavirus pathogenesis and tropism.Inflammation, ECG changes and pericardial effusion: whom to biopsy in suspected myocarditis?Viral persistence and chronic immunopathology in the adult central nervous system following Coxsackievirus infection during the neonatal period.Abnormalities of calcium metabolism and myocardial contractility depression in the failing heart.Viral myocarditis: potential defense mechanisms within the cardiomyocyte against virus infection.Viral myocarditis: from experimental models to molecular diagnosis in patients.Management of myopericarditis.A distinct subgroup of cardiomyopathy patients characterized by transcriptionally active cardiotropic erythrovirus and altered cardiac gene expression.5' terminal deletions in the genome of a coxsackievirus B2 strain occurred naturally in human heart.Vgamma4(+) T cells promote autoimmune CD8(+) cytolytic T-lymphocyte activation in coxsackievirus B3-induced myocarditis in mice: role for CD4(+) Th1 cells.Cell cycle status affects coxsackievirus replication, persistence, and reactivation in vitroCardiac Autoimmunity: Myocarditis.A phosphorothioate antisense oligodeoxynucleotide specifically inhibits coxsackievirus B3 replication in cardiomyocytes and mouse hearts.Major Persistent 5' Terminally Deleted Coxsackievirus B3 Populations in Human Endomyocardial Tissues.Enteroviral protease 2A directly cleaves dystrophin and is inhibited by a dystrophin-based substrate analogue.Immunological and pathological consequences of coxsackievirus RNA persistence in the heart.Differential interferon responses enhance viral epitope generation by myocardial immunoproteasomes in murine enterovirus myocarditis.Defective intracellular Ca(2+) signaling contributes to cardiomyopathy in Type 1 diabetic rats.Modulation of CICR has no maintained effect on systolic Ca2+: simultaneous measurements of sarcoplasmic reticulum and sarcolemmal Ca2+ fluxes in rat ventricular myocytes.Sarcoplasmic reticulum calcium defect in Ras-induced hypertrophic cardiomyopathy heart.Antigen-presentation capacity of dendritic cells is impaired in ongoing enterovirus myocarditis.
P2860
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P2860
Transgenic expression of replication-restricted enteroviral genomes in heart muscle induces defective excitation-contraction coupling and dilated cardiomyopathy.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on October 1998
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Transgenic expression of repli ...... ng and dilated cardiomyopathy.
@en
Transgenic expression of repli ...... ng and dilated cardiomyopathy.
@nl
type
label
Transgenic expression of repli ...... ng and dilated cardiomyopathy.
@en
Transgenic expression of repli ...... ng and dilated cardiomyopathy.
@nl
prefLabel
Transgenic expression of repli ...... ng and dilated cardiomyopathy.
@en
Transgenic expression of repli ...... ng and dilated cardiomyopathy.
@nl
P2093
P2860
P356
P1476
Transgenic expression of repli ...... ng and dilated cardiomyopathy.
@en
P2093
K U Knowlton
L F Santana
W Jonathan Lederer
P2860
P304
P356
10.1172/JCI1972
P407
P577
1998-10-01T00:00:00Z