Modulation of CICR has no maintained effect on systolic Ca2+: simultaneous measurements of sarcoplasmic reticulum and sarcolemmal Ca2+ fluxes in rat ventricular myocytes.
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Dysfunctional ryanodine receptors in the heart: new insights into complex cardiovascular diseasesA network-oriented perspective on cardiac calcium signalingLate Na(+) current and protracted electrical recovery are critical determinants of the aging myopathyRyR2 mutations linked to ventricular tachycardia and sudden death reduce the threshold for store-overload-induced Ca2+ release (SOICR)Inherited dysfunction of sarcoplasmic reticulum Ca2+ handling and arrhythmogenesisPhosphodiesterase 4B in the cardiac L-type Ca²⁺ channel complex regulates Ca²⁺ current and protects against ventricular arrhythmias in miceGender differences in the modulation of cardiac gene expression by dietary conjugated linoleic acid isomersPhosphorylation of the ryanodine receptor mediates the cardiac fight or flight response in mice.FKBP12.6 overexpression decreases Ca2+ spark amplitude but enhances [Ca2+]i transient in rat cardiac myocytes.Functional coupling of calcineurin and protein kinase A in mouse ventricular myocytes.Regulation of Ca2+ sparks by Ca2+ and Mg2+ in mammalian and amphibian muscle. An RyR isoform-specific role in excitation-contraction coupling?Dysregulated sarcoplasmic reticulum calcium release: potential pharmacological target in cardiac disease.Flecainide inhibits arrhythmogenic Ca2+ waves by open state block of ryanodine receptor Ca2+ release channels and reduction of Ca2+ spark mass.Ca2+ blinks: rapid nanoscopic store calcium signalingCaveolae create local signalling domains through their distinct protein content, lipid profile and morphologyDirect measurements of SR free Ca reveal the mechanism underlying the transient effects of RyR potentiation under physiological conditions.Transmural heterogeneity of repolarization and Ca2+ handling in a model of mouse ventricular tissue.Regulation of cardiac sarcoplasmic reticulum Ca release by luminal [Ca] and altered gating assessed with a mathematical model.Spontaneous Ca2+ sparks and Ca2+ homeostasis in a minimal model of permeabilized ventricular myocytes.The ryanodine receptor in cardiac physiology and disease.Balanced changes in Ca buffering by SERCA and troponin contribute to Ca handling during β-adrenergic stimulation in cardiac myocytesRedox signaling in cardiac myocytes.Superresolution modeling of calcium release in the heartRestitution of Ca(2+) release and vulnerability to arrhythmias.Regulation of cardiac excitation-contraction coupling by action potential repolarization: role of the transient outward potassium current (I(to)).Heart failure -- a challenge to our current concepts of excitation-contraction coupling.SERCA2a gene transfer decreases sarcoplasmic reticulum calcium leak and reduces ventricular arrhythmias in a model of chronic heart failure.How does stochastic ryanodine receptor-mediated Ca leak fail to initiate a Ca spark?Role of substrate and triggers in the genesis of cardiac alternans, from the myocyte to the whole heart: implications for therapy.Stability and instability of regulation of intracellular calcium.CaMKIIδC slows [Ca]i decline in cardiac myocytes by promoting Ca sparks.Loss of luminal Ca2+ activation in the cardiac ryanodine receptor is associated with ventricular fibrillation and sudden deathAcidosis in models of cardiac ventricular myocytes.Up-regulation of sarcoplasmic reticulum Ca(2+) uptake leads to cardiac hypertrophy, contractile dysfunction and early mortality in mice deficient in CASQ2.Parameter sensitivity analysis in electrophysiological models using multivariable regressionBeta-adrenergic signaling accelerates and synchronizes cardiac ryanodine receptor response to a single L-type Ca2+ channelSERCA2a gene therapy in heart failure: an anti-arrhythmic positive inotrope.What is the purpose of the large sarcolemmal calcium flux on each heartbeat?From eggs to hearts: what is the link between cyclic ADP-ribose and ryanodine receptors?Dynamic local changes in sarcoplasmic reticulum calcium: physiological and pathophysiological roles.
P2860
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P2860
Modulation of CICR has no maintained effect on systolic Ca2+: simultaneous measurements of sarcoplasmic reticulum and sarcolemmal Ca2+ fluxes in rat ventricular myocytes.
description
2000 nî lūn-bûn
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2000年の論文
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2000年学术文章
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2000年学术文章
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name
Modulation of CICR has no main ...... s in rat ventricular myocytes.
@en
Modulation of CICR has no main ...... s in rat ventricular myocytes.
@nl
type
label
Modulation of CICR has no main ...... s in rat ventricular myocytes.
@en
Modulation of CICR has no main ...... s in rat ventricular myocytes.
@nl
prefLabel
Modulation of CICR has no main ...... s in rat ventricular myocytes.
@en
Modulation of CICR has no main ...... s in rat ventricular myocytes.
@nl
P2860
P1476
Modulation of CICR has no main ...... es in rat ventricular myocytes
@en
P2093
A W Trafford
G C Sibbring
P2860
P304
P356
10.1111/J.1469-7793.2000.T01-2-00259.X
P407
P478
P577
2000-01-01T00:00:00Z