Recurrent somatic mutations in POLR2A define a distinct subset of meningiomas.
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Whole-genome sequencing identifies new genetic alterations in meningiomasGenomic profile of human meningioma cell lines.Genomic landscape of high-grade meningiomas.Diagnostic Challenges in Meningioma.The Emerging Role of TRAF7 in Tumor Development.SMO mutation status defines a distinct and frequent molecular subgroup in olfactory groove meningiomas.Transcriptional Addiction in Cancer.BAP1 mutations in high-grade meningioma: implications for patient care.Osteoglycin promotes meningioma development through downregulation of NF2 and activation of mTOR signaling.Integrated genomic analyses of de novo pathways underlying atypical meningiomasDouble somatic SMARCB1 and NF2 mutations in sporadic spinal schwannoma.RNA-seq transcriptome analysis of formalin fixed, paraffin-embedded canine meningioma.Highlights from the Literature.Overexpression of eIF4F components in meningiomas and suppression of meningioma cell growth by inhibiting translation initiation.Progestin-associated shift of meningioma mutational landscape.Somatic Superenhancer Duplications and Hotspot Mutations Lead to Oncogenic Activation of the KLF5 Transcription Factor.A description of familial clustering of meningiomas in the Utah population.Diffuse midline skull base meningiomas: identification of a rare and aggressive subgroup of meningiomas.Frequent AKT1E17K mutations in skull base meningiomas are associated with mTOR and ERK1/2 activation and reduced time to tumor recurrence.Meningiomas induced by low-dose radiation carry structural variants of NF2 and a distinct mutational signature.A gene expression signature predicts recurrence-free survival in meningioma.SMO mutant olfactory groove meningiomas-the next in line for targeted therapy.Predicting hepatocellular carcinoma through cross-talk genes identified by risk pathways.An overview of meningiomasGenetic Alterations of TRAF Proteins in Human CancersAn integrated genomic analysis of anaplastic meningioma identifies prognostic molecular signaturesEmerging Medical Treatments for Meningioma in the Molecular Era
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P2860
Recurrent somatic mutations in POLR2A define a distinct subset of meningiomas.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 22 August 2016
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Recurrent somatic mutations in POLR2A define a distinct subset of meningiomas.
@en
Recurrent somatic mutations in POLR2A define a distinct subset of meningiomas.
@nl
type
label
Recurrent somatic mutations in POLR2A define a distinct subset of meningiomas.
@en
Recurrent somatic mutations in POLR2A define a distinct subset of meningiomas.
@nl
prefLabel
Recurrent somatic mutations in POLR2A define a distinct subset of meningiomas.
@en
Recurrent somatic mutations in POLR2A define a distinct subset of meningiomas.
@nl
P2093
P2860
P50
P356
P1433
P1476
Recurrent somatic mutations in POLR2A define a distinct subset of meningiomas.
@en
P2093
A Gulhan Ercan-Sencicek
Abraham S Weintraub
Akdes Serin Harmancı
Alexander O Vortmeyer
Boris Krischek
E Zeynep Erson-Omay
Geneive Carrión-Grant
Jacob F Baranoski
Jennifer Moliterno Günel
Johanna E Goldmann
P2860
P2888
P304
P356
10.1038/NG.3651
P407
P577
2016-08-22T00:00:00Z
P6179
1005736302