Microglial derived tumor necrosis factor-α drives Alzheimer's disease-related neuronal cell cycle events
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CX3CL1/CX3CR1 in Alzheimer's Disease: A Target for NeuroprotectionReactive microglia drive tau pathology and contribute to the spreading of pathological tau in the brain.Excess cerebral TNF causing glutamate excitotoxicity rationalizes treatment of neurodegenerative diseases and neurogenic pain by anti-TNF agentsSynaptopathies: synaptic dysfunction in neurological disorders - A review from students to studentsmTOR and neuronal cell cycle reentry: How impaired brain insulin signaling promotes Alzheimer's disease.T cells promote the regeneration of neural precursor cells in the hippocampus of Alzheimer's disease mice.The evolving biology of microglia in Alzheimer's disease.Non-Neuronal Cells Are Required to Mediate the Effects of Neuroinflammation: Results from a Neuron-Enriched Culture System.Subventricular zone neural progenitors reverse TNF-alpha effects in cortical neurons.Mathematical model on Alzheimer's diseaseComparative Histological Study on the Therapeutic Effect of Green Tea and Stem Cells in Alzheimer's Disease Complicating Experimentally Induced Diabetes.The neuropathology and cerebrovascular mechanisms of dementiaDefining the Microglia Response during the Time Course of Chronic NeurodegenerationPotential role of PCTAIRE-2, PCTAIRE-3 and P-Histone H4 in amyloid precursor protein-dependent Alzheimer pathology.Altered Neuroinflammation and Behavior after Traumatic Brain Injury in a Mouse Model of Alzheimer's Disease.Environmental Enrichment Potently Prevents Microglia-Mediated Neuroinflammation by Human Amyloid β-Protein OligomersNeuronally-directed effects of RXR activation in a mouse model of Alzheimer's disease.Effects of environmental enrichment on white matter glial responses in a mouse model of chronic cerebral hypoperfusion.Oxidative Stress and Aberrant Cell Cycle in Alzheimer's Disease Lymphocytes: Diagnostic Prospects.Central nervous system myeloid cells as drug targets: current status and translational challenges.Posttranslational modification impact on the mechanism by which amyloid-β induces synaptic dysfunction.Fas apoptosis inhibitory molecules: more than death-receptor antagonists in the nervous system.Re-imagining Alzheimer's disease - the diminishing importance of amyloid and a glimpse of what lies ahead.Targeting neuroinflammation in Alzheimer's disease: evidence for NSAIDs and novel therapeutics.Individual Cytokines Modulate the Neurological Symptoms of ATM Deficiency in a Region Specific Manner(1,2,3)An integrated proteomics approach shows synaptic plasticity changes in an APP/PS1 Alzheimer's mouse model.TNFα affects CREB-mediated neuroprotective signaling pathways of synaptic plasticity in neurons as revealed by proteomics and phospho-proteomics.PPARγ coactivator-1α (PGC-1α) protects neuroblastoma cells against amyloid-beta (Aβ) induced cell death and neuroinflammation via NF-κB pathwayCharacterisation of extracellular vesicle-subsets derived from brain endothelial cells and analysis of their protein cargo modulation after TNF exposure.Selenofuranoside Ameliorates Memory Loss in Alzheimer-Like Sporadic Dementia: AChE Activity, Oxidative Stress, and Inflammation Involvement.Reduced brain insulin signaling: A seminal process in Alzheimer's disease pathogenesis.The Cholinergic System Modulates Memory and Hippocampal Plasticity via Its Interactions with Non-Neuronal Cells.Tempol and perindopril protect against lipopolysaccharide-induced cognition impairment and amyloidogenesis by modulating brain-derived neurotropic factor, neuroinflammation and oxido-nitrosative stress.Prostaglandin I2 is responsible for ameliorating prostaglandin E2 stress in stimulating the expression of tumor necrosis factor α in a β-amyloid protein -dependent mechanism.DNA damage-associated oligodendrocyte degeneration precedes amyloid pathology and contributes to Alzheimer's and dementia.Angelica tenuissima Nakai ameliorates cognitive impairment and promotes neurogenesis in mouse model of Alzheimer's disease.Androgen alleviates neurotoxicity of β-amyloid peptide (Aβ) by promoting microglial clearance of Aβ and inhibiting microglial inflammatory response to Aβ.U1 snRNP Alteration and Neuronal Cell Cycle Reentry in Alzheimer Disease.A Novel Association of Polymorphism in the ITGA4 Gene Encoding the VLA-4 α4 Subunit with Increased Risk of Alzheimer's Disease.Polyphenols Derived from Lychee Seed Suppress Aβ (1-42)-Induced Neuroinflammation
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P2860
Microglial derived tumor necrosis factor-α drives Alzheimer's disease-related neuronal cell cycle events
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on 17 October 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Microglial derived tumor necro ...... ted neuronal cell cycle events
@en
Microglial derived tumor necro ...... ed neuronal cell cycle events.
@nl
type
label
Microglial derived tumor necro ...... ted neuronal cell cycle events
@en
Microglial derived tumor necro ...... ed neuronal cell cycle events.
@nl
prefLabel
Microglial derived tumor necro ...... ted neuronal cell cycle events
@en
Microglial derived tumor necro ...... ed neuronal cell cycle events.
@nl
P2093
P2860
P1476
Microglial derived tumor necro ...... ted neuronal cell cycle events
@en
P2093
Astrid Cardona
Bruce T Lamb
Guixiang Xu
Jason P Weick
Karl Herrup
Kiran Bhaskar
Nicole Maphis
Olga N Kokiko-Cochran
Richard M Ransohoff
Susan M Staugaitis
P2860
P304
P356
10.1016/J.NBD.2013.10.007
P577
2013-10-17T00:00:00Z