Hypoxia enhances stimulus-dependent induction of E-selectin on aortic endothelial cells. - Wikidata - wikimedia - TriplyDB

Hypoxia enhances stimulus-dependent induction of E-selectin on aortic endothelial cells.

Hypoxia enhances stimulus-dependent induction of E-selectin on aortic endothelial cells.

http://www.wikidata.org/entity/Q37486712

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Identification of tumor associated single-chain Fv by panning and screening antibody phage library using tumor cells.Ecto-5'-nucleotidase (CD73) regulation by hypoxia-inducible factor-1 mediates permeability changes in intestinal epithelia.Hypoxia-inducible factor 1-dependent induction of intestinal trefoil factor protects barrier function during hypoxia The transcription factor DEC1 (stra13, SHARP2) is associated with the hypoxic response and high tumour grade in human breast cancers Exercise-induced stem cell activation and its implication for cardiovascular and skeletal muscle regeneration.Activated endothelial cells elicit paracrine induction of epithelial chloride secretion. 6-Keto-PGF1alpha is an epithelial secretagogue Effects of acute hypoxia and lipopolysaccharide on nitric oxide synthase-2 expression in acute lung injury.Monocyte adhesion and spreading on human endothelial cells is dependent on Rho-regulated receptor clustering.Mechanisms of hypoxia-induced endothelial cell death. Role of p53 in apoptosis.Hyperbaric oxygen treatment reduces neutrophil-endothelial adhesion in chronic wound conditions through S-nitrosation.

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Hypoxia enhances stimulus-dependent induction of E-selectin on aortic endothelial cells.

http://www.wikidata.org/entity/Q37486712

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on July 1996

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Hypoxia enhances stimulus-dependent induction of E-selectin on aortic endothelial cells.

@en

Hypoxia enhances stimulus-dependent induction of E-selectin on aortic endothelial cells.

@nl

type

label

Hypoxia enhances stimulus-dependent induction of E-selectin on aortic endothelial cells.

@en

Hypoxia enhances stimulus-dependent induction of E-selectin on aortic endothelial cells.

@nl

prefLabel

Hypoxia enhances stimulus-dependent induction of E-selectin on aortic endothelial cells.

@en

Hypoxia enhances stimulus-dependent induction of E-selectin on aortic endothelial cells.

@nl

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PNAS.93.14.7075

P1433

Proceedings of the National Academy of Sciences of the United States of America

P1476

Hypoxia enhances stimulus-dependent induction of E-selectin on aortic endothelial cells

@en

P2093

A L Dzus

http://www.w3.org/2001/XMLSchema#string

D P Nelson

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E J Neufeld

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G Zünd

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J E Mayer

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S P Colgan

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Endothelial cell tolerance to hypoxia. Potential role of purine nucleotide phosphates.

Vectorial targeting of apical and basolateral plasma membrane proteins in a human adenocarcinoma epithelial cell line.

Identification of an inducible endothelial-leukocyte adhesion molecule.

In vivo neutralization of P-selectin protects feline heart and endothelium in myocardial ischemia and reperfusion injury.

Pathophysiology of ischaemia reperfusion injury: central role of the neutrophil.

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Induction of interleukin 6 (IL-6) by hypoxia in vascular cells. Central role of the binding site for nuclear factor-IL-6.

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7075-7080

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scholarly article

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10.1073/PNAS.93.14.7075

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14509204

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