CDK4/6 inhibition induces epithelial cell cycle arrest and ameliorates acute kidney injury.
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DNA Damage in Chronic Kidney Disease: Evaluation of Clinical BiomarkersTargeting Endogenous Repair Pathways after AKI.Progression after AKI: Understanding Maladaptive Repair Processes to Predict and Identify Therapeutic Treatments.Fluorescence microangiography for quantitative assessment of peritubular capillary changes after AKI in miceNew targeted therapies for breast cancer: A focus on tumor microenvironmental signals and chemoresistant breast cancersMitigation of acute kidney injury by cell-cycle inhibitors that suppress both CDK4/6 and OCT2 functionsIdentification of deregulation of apoptosis and cell cycle in neuroendocrine tumors of the lung via NanoString nCounter expression analysis.Immune system modulation of kidney regeneration--mechanisms and implications.Cell cycle control in the kidney.Mechanisms of maladaptive repair after AKI leading to accelerated kidney ageing and CKD.Necroinflammation in Kidney Disease.Stop adding insult to injury-identifying and managing risk factors for the progression of acute kidney injury in children.To clear, or not to clear (senescent cells)? That is the question.DNA repair in ischemic acute kidney injury.Cellular senescence in renal ageing and disease.Sav1 Loss Induces Senescence and Stat3 Activation Coinciding with Tubulointerstitial Fibrosis.Evaluation of biomarkers of cell cycle arrest and inflammation in prediction of dialysis or recovery after kidney transplantation.MicroRNA-H4-5p encoded by HSV-1 latency-associated transcript promotes cell proliferation, invasion and cell cycle progression via p16-mediated PI3K-Akt signaling pathway in SHSY5Y cells.Renal fibrosis: Primacy of the proximal tubule.Meis1 is specifically upregulated in kidney myofibroblasts during aging and injury but is not required for kidney homeostasis or fibrotic response.Acquired resistance to rechallenge injury after acute kidney injury in rats is associated with cell cycle arrest in proximal tubule cells.Immune mechanisms in the different phases of acute tubular necrosis
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CDK4/6 inhibition induces epithelial cell cycle arrest and ameliorates acute kidney injury.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 11 December 2013
@en
vedecký článok
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vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
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name
CDK4/6 inhibition induces epit ...... eliorates acute kidney injury.
@en
CDK4/6 inhibition induces epit ...... eliorates acute kidney injury.
@nl
type
label
CDK4/6 inhibition induces epit ...... eliorates acute kidney injury.
@en
CDK4/6 inhibition induces epit ...... eliorates acute kidney injury.
@nl
prefLabel
CDK4/6 inhibition induces epit ...... eliorates acute kidney injury.
@en
CDK4/6 inhibition induces epit ...... eliorates acute kidney injury.
@nl
P2093
P2860
P1476
CDK4/6 inhibition induces epit ...... meliorates acute kidney injury
@en
P2093
Benjamin D Humphreys
Derek P DiRocco
Patrick Roberts
P2860
P304
P356
10.1152/AJPRENAL.00475.2013
P577
2013-12-11T00:00:00Z