Several mechanisms can account for defective E alpha gene expression in different mouse haplotypes.
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Role for Msh5 in the regulation of Ig class switch recombinationTransport of Streptococcus pneumoniae capsular polysaccharide in MHC Class II tubulesImmunity to retroviral infection: the Friend virus modelAnalysis of the transcriptional networks underpinning the activation of murine macrophages by inflammatory mediatorsA genetic and functional relationship between T cells and cellular proliferation in the adult hippocampusIdentification of murine H2-Dd- and H2-Ab-restricted T-cell epitopes on a novel protective antigen, MPT51, of Mycobacterium tuberculosis.Altered I-J phenotype in E alpha transgenic mice.CD207+ CD103+ dermal dendritic cells cross-present keratinocyte-derived antigens irrespective of the presence of Langerhans cellsComparative genome organization of the major histocompatibility complex: lessons from the Felidae.Commercially available outbred mice for genome-wide association studies.Mice of the resistant H-2(b) haplotype mount broad CD4(+) T cell responses against 9 distinct Friend virus epitopes.Determinants of the B-cell response against a transgenic autoantigenInbred and wild mice carry identical deletions in their E alpha MHC genes.Tissue-specific, inducible and functional expression of the E alpha d MHC class II gene in transgenic mice.Eo: a history of a mutation.Detection of new MHC mutations in mice by skin grafting, tumor transplantation and monoclonal antibodies: a comparisonLessons from BXSB and related mouse models.Requirement for the coexpression of T3 and the T cell antigen receptor on a malignant human T cell line.Both positive and negative effects on immune responses by expression of a second class II MHC molecule.Major histocompatibility complex-linked diabetogenic gene of the nonobese diabetic mouse. Analysis of genomic DNA amplified by the polymerase chain reaction.Localization of a critical restriction site on the I-A beta chain that determines susceptibility to collagen-induced arthritis in mice.Major histocompatibility complex gene organization in the mole rat Spalax ehrenbergi: evidence for transfer of function between class II genes.Conserved major histocompatibility complex class II boxes--X and Y--are transcriptional control elements and specifically bind nuclear proteins.Characterization of a conserved helper-T-cell epitope from group A Streptococcal M proteinsMHC class II-alpha chain knockout mice support increased viral replication that is independent of their lack of MHC class II cell surface expression and associated immune function deficiencies.Far upstream regions of class II MHC Ea are necessary for position-independent, copy-dependent expression of Ea transgene.Cell surface molecules involved in early events in T-cell mitogenic stimulation by staphylococcal enterotoxins.The complete nucleotide sequence of the I-E alpha d immune response gene.Mapping of TH1 helper T-cell epitopes on major secreted mycobacterial antigen 85A in mice infected with live Mycobacterium bovis BCG.DNase I hypersensitive sites flank the mouse class II major histocompatibility complex during B cell development.Identification of 2 major loci linked to autoimmune hemolytic anemia in NZB miceVariable hematopoietic graft rejection and graft-versus-host disease in MHC-matched strains of mice.The T cell receptor repertoire influences V beta element usage in response to myoglobin.Presentation of antigen by mixed isotype class II molecules in normal H-2d mice.Identification of immunodominant T cell epitopes of human glutamic acid decarboxylase 65 by using HLA-DR(alpha1*0101,beta1*0401) transgenic mice.Dual signaling by innate and adaptive immune receptors is required for TLR7-induced B-cell-mediated autoimmunity.Recombination within the HLA-D region. Correlation of molecular genotyping with functional dataHLA class II regulation and structure. Analysis with HLA-DR3 and HLA-DP point mutantsMapping of a second recombination hot spot within the I-E region of the mouse H-2 gene complex.Genetic control of diabetes and insulitis in the nonobese diabetic (NOD) mouse
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P2860
Several mechanisms can account for defective E alpha gene expression in different mouse haplotypes.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on January 1983
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Several mechanisms can account ...... in different mouse haplotypes.
@en
Several mechanisms can account ...... in different mouse haplotypes.
@nl
type
label
Several mechanisms can account ...... in different mouse haplotypes.
@en
Several mechanisms can account ...... in different mouse haplotypes.
@nl
prefLabel
Several mechanisms can account ...... in different mouse haplotypes.
@en
Several mechanisms can account ...... in different mouse haplotypes.
@nl
P2093
P2860
P356
P1476
Several mechanisms can account ...... in different mouse haplotypes.
@en
P2093
D J Mathis
H O McDevitt
V E Williams
P2860
P304
P356
10.1073/PNAS.80.1.273
P407
P577
1983-01-01T00:00:00Z