Identification of a rare Epstein-Barr virus variant that enhances early antigen expression in Raji cells.
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EBNA2 and activated Notch induce expression of BATFGenome rearrangements activate the Epstein-Barr virus gene whose product disrupts latency.Epstein-Barr virus WZhet DNA can induce lytic replication in epithelial cells in vitro, although WZhet is not detectable in many human tissues in vivo.Autostimulation of the Epstein-Barr virus BRLF1 promoter is mediated through consensus Sp1 and Sp3 binding sites.Both Epstein-Barr virus (EBV)-encoded trans-acting factors, EB1 and EB2, are required to activate transcription from an EBV early promoter.BHRF1 of Epstein-Barr virus, which is homologous to human proto-oncogene bcl2, is not essential for transformation of B cells or for virus replication in vitro.Immortalization of human primary B lymphocytes in vitro with DNA.Protein kinase C-independent activation of the Epstein-Barr virus lytic cycle.Latent membrane protein 1 inhibits Epstein-Barr virus lytic cycle induction and progress via different mechanisms.Transfection of a rearranged viral DNA fragment, WZhet, stably converts latent Epstein-Barr viral infection to productive infection in lymphoid cells.Epithelial cell retention of transcriptionally active, P3HR-1-derived heterogeneous Epstein-Barr virus DNA with concurrent loss of parental virus.Autoantigenic proteins that bind recombinogenic sequences in Epstein-Barr virus and cellular DNAA defective, rearranged Epstein-Barr virus genome in EBER-negative and EBER-positive Hodgkin's disease.Immortalization of human B lymphocytes by a plasmid containing 71 kilobase pairs of Epstein-Barr virus DNA.Selective switch between latency and lytic replication of Kaposi's sarcoma herpesvirus and Epstein-Barr virus in dually infected body cavity lymphoma cells.Identification, expression, and immunogenicity of Kaposi's sarcoma-associated herpesvirus-encoded small viral capsid antigen.Late gene expression from the Epstein-Barr virus BcLF1 and BFRF3 promoters does not require DNA replication in cis.An oriP expression vector containing the HIV-1 Tat/TAR transactivation axis produces high levels of protein expression in mammalian cellsMarker rescue of a transformation-negative Epstein-Barr virus recombinant from an infected Burkitt lymphoma cell line: a method useful for analysis of genes essential for transformationLatent membrane protein of Epstein-Barr virus induces cellular phenotypes independently of expression of Bcl-2.Second-site homologous recombination in Epstein-Barr virus: insertion of type 1 EBNA 3 genes in place of type 2 has no effect on in vitro infection.Epstein-Barr virus (EBV)-negative B-lymphoma cell lines for clonal isolation and replication of EBV recombinants.Viral proteins associated with the Epstein-Barr virus transactivator, ZEBRA.Influence of Burkitt's lymphoma and primary B cells on latent gene expression by the nonimmortalizing P3J-HR-1 strain of Epstein-Barr virus.Expression of the BZLF1 latency-disrupting gene differs in standard and defective Epstein-Barr viruses.Defective viral DNA in Epstein-Barr virus-associated oral hairy leukoplakia.Identification of phorbol ester response elements in the promoter of Epstein-Barr virus putative lytic switch gene BZLF1The level of c-fgr RNA is increased by EBNA-2, an Epstein-Barr virus gene required for B-cell immortalization.Epstein-Barr virus nuclear antigen 2 transactivates latent membrane protein LMP1.The spliced BZLF1 gene of Epstein-Barr virus (EBV) transactivates an early EBV promoter and induces the virus productive cycle.Mapping of genes in BamHI fragment M of Epstein-Barr virus DNA that may determine the fate of viral infection.Palindromic structure and polypeptide expression of 36 kilobase pairs of heterogeneous Epstein-Barr virus (P3HR-1) DNA.Amplification of Epstein-Barr virus (EBV) DNA by superinfection with a strain of EBV derived from nasopharyngeal carcinoma.Promiscuous trans activation of gene expression by an Epstein-Barr virus-encoded early nuclear proteinSequences of the Epstein-Barr Virus (EBV) large internal repeat form the center of a 16-kilobase-pair palindrome of EBV (P3HR-1) heterogeneous DNA.Two strains of Epstein-Barr virus (B95-8 and a P3HR-1 subclone) that lack defective genomes induce early antigen and cause abortive infection of Raji cells.P3HR-1 Epstein-Barr virus with heterogeneous DNA is an independent replicon maintained by cell-to-cell spread.Latent and viral replicative transcription in vivo from the BamHI K fragment of Epstein-Barr virus DNA.Constitutive expression of Epstein-Barr virus-encoded RNAs and nuclear antigen during latency and after induction of Epstein-Barr virus replication.Epstein-Barr virus with heterogeneous DNA disrupts latency
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P2860
Identification of a rare Epstein-Barr virus variant that enhances early antigen expression in Raji cells.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on May 1983
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Identification of a rare Epste ...... igen expression in Raji cells.
@en
Identification of a rare Epste ...... igen expression in Raji cells.
@nl
type
label
Identification of a rare Epste ...... igen expression in Raji cells.
@en
Identification of a rare Epste ...... igen expression in Raji cells.
@nl
prefLabel
Identification of a rare Epste ...... igen expression in Raji cells.
@en
Identification of a rare Epste ...... igen expression in Raji cells.
@nl
P2093
P2860
P4510
P356
P1476
Identification of a rare Epste ...... igen expression in Raji cells.
@en
P2093
P2860
P304
P356
10.1073/PNAS.80.9.2762
P407
P577
1983-05-01T00:00:00Z