Diabetes and arterial extracellular matrix changes in a porcine model of atherosclerosis.
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Regulated Hyaluronan Synthesis by Vascular CellsRegulation of hyaluronan synthesis in vascular diseases and diabetesOxidative stress and diabetic complicationsDiabetic nephropathy and antioxidants.Diabetes-induced alterations in the extracellular matrix and their impact on myocardial function.Roles of polyuria and hyperglycemia in bladder dysfunction in diabetesIncrease in GLUT1 in smooth muscle alters vascular contractility and increases inflammation in response to vascular injuryHyaluronan as an immune regulator in human diseases.Fluorescence resonance energy transfer (FRET) and proximity ligation assays reveal functionally relevant homo- and heteromeric complexes among hyaluronan synthases HAS1, HAS2, and HAS3Valsartan blocks thrombospondin/transforming growth factor/Smads to inhibit aortic remodeling in diabetic ratsMonocyte-to-macrophage differentiation: synthesis and secretion of a complex extracellular matrix.How to best manage glycemia and non-glycemia during the time of acute myocardial infarction.Obstruction-induced pulmonary vascular remodeling.Arterial retention of remnant lipoproteins ex vivo is increased in insulin resistance because of increased arterial biglycan and production of cholesterol-rich atherogenic particles that can be improved by ezetimibe in the JCR:LA-cp ratClinical Update: Cardiovascular Disease in Diabetes Mellitus: Atherosclerotic Cardiovascular Disease and Heart Failure in Type 2 Diabetes Mellitus - Mechanisms, Management, and Clinical ConsiderationsCardiovascular disease risk in type 2 diabetes mellitus: insights from mechanistic studies.Molecular biology of atherosclerosis.Epigenetics in extracellular matrix remodeling and hyaluronan metabolism.Biology and biotechnology of hyaluronan.Role of UDP-N-acetylglucosamine (GlcNAc) and O-GlcNAcylation of hyaluronan synthase 2 in the control of chondroitin sulfate and hyaluronan synthesis.Histomorphological and biochemical properties of plantar soft tissue in diabetes.Elevated circulating TGF-β is not the cause of increased atherosclerosis development in biglycan deficient mice.Activation of Nrf2 signaling by natural products-can it alleviate diabetes?Increased risk of cardiovascular disease in Type 1 diabetes: arterial exposure to remnant lipoproteins leads to enhanced deposition of cholesterol and binding to glycated extracellular matrix proteoglycans.
P2860
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P2860
Diabetes and arterial extracellular matrix changes in a porcine model of atherosclerosis.
description
article científic
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article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 24 July 2007
@en
vedecký článok
@sk
vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Diabetes and arterial extracellular matrix changes in a porcine model of atherosclerosis.
@en
Diabetes and arterial extracellular matrix changes in a porcine model of atherosclerosis.
@nl
type
label
Diabetes and arterial extracellular matrix changes in a porcine model of atherosclerosis.
@en
Diabetes and arterial extracellular matrix changes in a porcine model of atherosclerosis.
@nl
prefLabel
Diabetes and arterial extracellular matrix changes in a porcine model of atherosclerosis.
@en
Diabetes and arterial extracellular matrix changes in a porcine model of atherosclerosis.
@nl
P2093
P2860
P356
P1476
Diabetes and arterial extracellular matrix changes in a porcine model of atherosclerosis.
@en
P2093
Alan Chait
Christy Jen
Hao-Ji Chen
Kathleen Wark
Kevin D O'Brien
Ross G Gerrity
Thomas N Wight
Thomas O McDonald
P2860
P304
P356
10.1369/JHC.7A7221.2007
P577
2007-07-24T00:00:00Z