The dysregulation of intracellular calcium in Alzheimer disease.
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Receptor for advanced glycation end products and its involvement in inflammatory diseasesLevetiracetam suppresses neuronal network dysfunction and reverses synaptic and cognitive deficits in an Alzheimer's disease model.Research progress on flavonoids isolated from traditional Chinese medicine in treatment of Alzheimer's diseaseThe synaptic maintenance problem: membrane recycling, Ca2+ homeostasis and late onset degenerationAmyloid-β oligomers induce tau-independent disruption of BDNF axonal transport via calcineurin activation in cultured hippocampal neuronsThe role of ryanodine receptor type 3 in a mouse model of Alzheimer diseaseCalcium Dynamics Mediated by the Endoplasmic/Sarcoplasmic Reticulum and Related Diseases.Mutagenesis mapping of the presenilin 1 calcium leak conductance pore.Role of presenilins in neuronal calcium homeostasisProlonged Abeta treatment leads to impairment in the ability of primary cortical neurons to maintain K+ and Ca2+ homeostasis.PSAPP mice exhibit regionally selective reductions in gliosis and plaque deposition in response to S100B ablation.Disturbed Ca2+ homeostasis increases glutaminyl cyclase expression; connecting two early pathogenic events in Alzheimer's disease in vitro.Non-selective calcium channel blocker bepridil decreases secondary pathology in mice after photothrombotic cortical lesionMembrane Incorporation, Channel Formation, and Disruption of Calcium Homeostasis by Alzheimer's β-Amyloid Protein.Spinocerebellar ataxia type 13 mutant potassium channel alters neuronal excitability and causes locomotor deficits in zebrafish.Preventing effect of L-type calcium channel blockade on electrophysiological alterations in dentate gyrus granule cells induced by entorhinal amyloid pathologyBenzodiazepinone derivatives protect against endoplasmic reticulum stress-mediated cell death in human neuronal cell linesA translational continuum of model systems for evaluating treatment strategies in Alzheimer's disease: isradipine as a candidate drug.CALHM1 P86L polymorphism modulates CSF Aβ levels in cognitively healthy individuals at risk for Alzheimer's disease.Identification of tetrahydrocarbazoles as novel multifactorial drug candidates for treatment of Alzheimer's disease.Hypoxia-inducible factor-1α regulates the expression of L-type voltage-dependent Ca(2+) channels in PC12 cells under hypoxia.Multi-Target Analysis and Design of Mitochondrial Metabolism.Identification of ER-000444793, a Cyclophilin D-independent inhibitor of mitochondrial permeability transition, using a high-throughput screen in cryopreserved mitochondria.Na+ and K+ ion imbalances in Alzheimer's disease.Histamine Induces Alzheimer's Disease-Like Blood Brain Barrier Breach and Local Cellular Responses in Mouse Brain Organotypic Cultures.Glutamate system, amyloid ß peptides and tau protein: functional interrelationships and relevance to Alzheimer disease pathologyIncreasing the Receptor Tyrosine Kinase EphB2 Prevents Amyloid-β-induced Depletion of Cell Surface Glutamate Receptors by a Mechanism That Requires the PDZ-binding Motif of EphB2 and Neuronal Activity.Upregulation of calpain activity precedes tau phosphorylation and loss of synaptic proteins in Alzheimer's disease brain.Recent advances in RNA interference therapeutics for CNS diseases.Neuroprotective effects of apigenin against inflammation, neuronal excitability and apoptosis in an induced pluripotent stem cell model of Alzheimer's disease.Role of endoplasmic reticulum Ca2+ signaling in the pathogenesis of Alzheimer diseaseInvolvement of presenilin holoprotein upregulation in calcium dyshomeostasis of Alzheimer's disease.S100A9 knockout decreases the memory impairment and neuropathology in crossbreed mice of Tg2576 and S100A9 knockout mice modelNeuronal calcium signaling, mitochondrial dysfunction, and Alzheimer's disease.Endoplasmic-reticulum calcium depletion and disease.The closing and opening of TRPC channels by Homer1 and STIM1.Presenilins function in ER calcium leak and Alzheimer's disease pathogenesis.Presenilins as endoplasmic reticulum calcium leak channels and Alzheimer's disease pathogenesis.Road to Alzheimer's disease: the pathomechanism underlying.Ca(2+) signaling: an outlook on the characterization of Ca(2+) channels and their importance in cellular functions
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The dysregulation of intracellular calcium in Alzheimer disease.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 18 January 2010
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
The dysregulation of intracellular calcium in Alzheimer disease.
@en
The dysregulation of intracellular calcium in Alzheimer disease.
@nl
type
label
The dysregulation of intracellular calcium in Alzheimer disease.
@en
The dysregulation of intracellular calcium in Alzheimer disease.
@nl
prefLabel
The dysregulation of intracellular calcium in Alzheimer disease.
@en
The dysregulation of intracellular calcium in Alzheimer disease.
@nl
P2860
P1433
P1476
The dysregulation of intracellular calcium in Alzheimer disease.
@en
P2093
Charlene Supnet
Ilya Bezprozvanny
P2860
P304
P356
10.1016/J.CECA.2009.12.014
P577
2010-01-18T00:00:00Z