Defective lysosomal exocytosis and plasma membrane repair in Chediak-Higashi/beige cells. - Wikidata - wikimedia - TriplyDB

Defective lysosomal exocytosis and plasma membrane repair in Chediak-Higashi/beige cells.

Defective lysosomal exocytosis and plasma membrane repair in Chediak-Higashi/beige cells.

http://www.wikidata.org/entity/Q37695560

about

The FTLD risk factor TMEM106B and MAP6 control dendritic trafficking of lysosomes Towards the targeted management of Chediak-Higashi syndrome Lysosomal storage diseases and the heat shock response: convergences and therapeutic opportunities Deleterious mutations in LRBA are associated with a syndrome of immune deficiency and autoimmunity Elevated oxidative membrane damage associated with genetic modifiers of Lyst-mutant phenotypes Depletion of kinesin 5B affects lysosomal distribution and stability and induces peri-nuclear accumulation of autophagosomes in cancer cells X-linked myopathy with excessive autophagy: a failure of self-eating Autophagy and lysosomal dysfunction as emerging mechanisms of nanomaterial toxicity Control of parasitophorous vacuole expansion by LYST/Beige restricts the intracellular growth of Leishmania amazonensis Induction of transient macroapertures in endothelial cells through RhoA inhibition by Staphylococcus aureus factors.Neuraminidase 1 is a negative regulator of lysosomal exocytosis S100A11 is required for efficient plasma membrane repair and survival of invasive cancer cells.Qualitative disorders of platelets and megakaryocytes.An inherited macrothrombocytopenic disorder with abnormal large granules.Transcription factor foxq1 controls mucin gene expression and granule content in mouse stomach surface mucous cells.The small chemical vacuolin-1 alters the morphology of lysosomes without inhibiting Ca2+-regulated exocytosis.Lipid raft redox signaling: molecular mechanisms in health and disease.S100 and annexin proteins identify cell membrane damage as the Achilles heel of metastatic cancer cells.Induction of natural killer cell responses by ectromelia virus controls infection.Clinical Outcome 3 Years After Autologous Chondrocyte Implantation Does Not Correlate With the Expression of a Predefined Gene Marker Set in Chondrocytes Prior to Implantation but Is Associated With Critical Signaling Pathways Cell Electrosensitization Exists Only in Certain Electroporation Buffers Membrane Repair: Mechanisms and Pathophysiology.Helicobacter pylori disrupts host cell membranes, initiating a repair response and cell proliferation.Expression and function of synaptotagmin VII in CTLs Evidence for lysosomal exocytosis and release of aggrecan-degrading hydrolases from hypertrophic chondrocytes, in vitro and in vivo.Approaches for plasma membrane wounding and assessment of lysosome-mediated repair responses.Atypical Chédiak-Higashi syndrome with attenuated phenotype: three adult siblings homozygous for a novel LYST deletion and with neurodegenerative disease.Neurologic involvement in patients with atypical Chediak-Higashi disease.Endothelial peroxisomal dysfunction and impaired pexophagy promotes oxidative damage in lipopolysaccharide-induced acute kidney injury.Membrane raft redox signalosomes in endothelial cells Implications for the mammalian sialidases in the physiopathology of skeletal muscle Electrochemotherapy of tumors as in situ vaccination boosted by immunogene electrotransfer Inhibition of Transient Receptor Potential Channel Mucolipin-1 (TRPML1) by Lysosomal Adenosine Involved in Severe Combined Immunodeficiency Diseases.Gene Electrotransfer: A Mechanistic Perspective.A double-pulse approach for electrotransfection.Plasma membrane wounding and repair in pulmonary diseases.Release of calcium from endolysosomes increases calcium influx through N-type calcium channels: Evidence for acidic store-operated calcium entry in neurons Peripheral nervous system manifestations of Chediak-Higashi disease.Oxygen-glucose deprivation (OGD) and interleukin-1 (IL-1) differentially modulate cathepsin B/L mediated generation of neuroprotective perlecan LG3 by neurons.Calcium-dependent freezing tolerance in Arabidopsis involves membrane resealing via synaptotagmin SYT1.

P2860

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P2860

Defective lysosomal exocytosis and plasma membrane repair in Chediak-Higashi/beige cells.

http://www.wikidata.org/entity/Q37695560

description

2004 nî lūn-bûn

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2004年の論文

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年學術文章

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2004年學術文章

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2004年學術文章

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name

Defective lysosomal exocytosis and plasma membrane repair in Chediak-Higashi/beige cells.

@en

Defective lysosomal exocytosis and plasma membrane repair in Chediak-Higashi/beige cells.

@nl

type

label

Defective lysosomal exocytosis and plasma membrane repair in Chediak-Higashi/beige cells.

@en

Defective lysosomal exocytosis and plasma membrane repair in Chediak-Higashi/beige cells.

@nl

prefLabel

Defective lysosomal exocytosis and plasma membrane repair in Chediak-Higashi/beige cells.

@en

Defective lysosomal exocytosis and plasma membrane repair in Chediak-Higashi/beige cells.

@nl

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Defective lysosomal exocytosis and plasma membrane repair in Chediak-Higashi/beige cells

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Chau Huynh

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Diane M Ward

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Doris Roth

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Jerry Kaplan

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P2860

Chédiak-Higashi gene in humans I. Impairment of natural-killer function

Identification of SNAREs involved in synaptotagmin VII-regulated lysosomal exocytosis

Deficient peptide loading and MHC class II endosomal sorting in a human genetic immunodeficiency disease: the Chediak-Higashi syndrome

Rapid colorimetric assay for cellular growth and survival: Application to proliferation and cytotoxicity assays

Loss of cytotoxic T lymphocyte function in Chediak-Higashi syndrome arises from a secretory defect that prevents lytic granule exocytosis

Impaired membrane resealing and autoimmune myositis in synaptotagmin VII-deficient mice

Plasma membrane repair is mediated by Ca(2+)-regulated exocytosis of lysosomes

Synaptotagmin VII regulates Ca(2+)-dependent exocytosis of lysosomes in fibroblasts

The Beige/Chediak-Higashi syndrome gene encodes a widely expressed cytosolic protein

Defective lysosomal enzyme secretion in kidneys of Chediak-Higashi (beige) mice

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