Pain disorders and erythromelalgia caused by voltage-gated sodium channel mutations. - Wikidata - wikimedia - TriplyDB

Pain disorders and erythromelalgia caused by voltage-gated sodium channel mutations.

Pain disorders and erythromelalgia caused by voltage-gated sodium channel mutations.

http://www.wikidata.org/entity/Q37943756

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The perception and endogenous modulation of pain Varicella-zoster viruses associated with post-herpetic neuralgia induce sodium current density increases in the ND7-23 Nav-1.8 neuroblastoma cell line.Advanced Genetic Testing Comes to the Pain Clinic to Make a Diagnosis of Paroxysmal Extreme Pain Disorder.The fundamental unit of pain is the cell.Advances in targeting voltage-gated sodium channels with small molecules.Mutational consequences of aberrant ion channels in neurological disorders.Erythromelalgia: a cutaneous manifestation of neuropathy?Pain thresholds, supra-threshold pain and lidocaine sensitivity in patients with erythromelalgia, including the I848Tmutation in NaV 1.7.

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P2860

Pain disorders and erythromelalgia caused by voltage-gated sodium channel mutations.

http://www.wikidata.org/entity/Q37943756

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Pain disorders and erythromelalgia caused by voltage-gated sodium channel mutations.

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Pain disorders and erythromelalgia caused by voltage-gated sodium channel mutations.

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type

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Pain disorders and erythromelalgia caused by voltage-gated sodium channel mutations.

@en

Pain disorders and erythromelalgia caused by voltage-gated sodium channel mutations.

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prefLabel

Pain disorders and erythromelalgia caused by voltage-gated sodium channel mutations.

@en

Pain disorders and erythromelalgia caused by voltage-gated sodium channel mutations.

@nl

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Current Neurology and Neuroscience Reports

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Pain disorders and erythromelalgia caused by voltage-gated sodium channel mutations.

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Ron Dabby

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P2860

Overview of the voltage-gated sodium channel family

Nociceptor-specific gene deletion reveals a major role for Nav1.7 (PN1) in acute and inflammatory pain

Mutations in SCN9A, encoding a sodium channel alpha subunit, in patients with primary erythermalgia

SCN9A mutations in paroxysmal extreme pain disorder: allelic variants underlie distinct channel defects and phenotypes

From ionic currents to molecular mechanisms: the structure and function of voltage-gated sodium channels

Rectal, ocular, and submaxillary pain; a familial autonomic disorder related to proctalgia fugaz: report of a family

Comparative distribution of voltage-gated sodium channel proteins in human brain

Paroxysmal extreme pain disorder (previously familial rectal pain syndrome)

An SCN9A channelopathy causes congenital inability to experience pain

International Union of Pharmacology. XLVII. Nomenclature and structure-function relationships of voltage-gated sodium channels

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s11910-011-0233-8

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76-83

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10.1007/S11910-011-0233-8

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1

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