More to life than death: molecular determinants of necroptotic and non-necroptotic RIP3 kinase signaling.
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Insights into the evolution of divergent nucleotide-binding mechanisms among pseudokinases revealed by crystal structures of human and mouse MLKLMolecular crosstalk between apoptosis, necroptosis, and survival signalingAn outline of necrosome triggersThe intersection of cell death and inflammasome activationRIPK3 promotes cell death and NLRP3 inflammasome activation in the absence of MLKLRIPK1 blocks early postnatal lethality mediated by caspase-8 and RIPK3Mitochondrial apoptosis is dispensable for NLRP3 inflammasome activation but non-apoptotic caspase-8 is required for inflammasome primingActivation of the pseudokinase MLKL unleashes the four-helix bundle domain to induce membrane localization and necroptotic cell death.RIP kinases: key decision makers in cell death and innate immunity.Upregulation of the receptor-interacting protein 3 expression and involvement in neural tissue damage after spinal cord injury in mice.Immune-Mediated Nephropathy and Systemic Autoimmunity in Mice Does Not Require Receptor Interacting Protein Kinase 3 (RIPK3).Caspase-8: not so silently deadly.Caspase-8 functions as a key mediator of inflammation and pro-IL-1β processing via both canonical and non-canonical pathways.Regulation of antiapoptotic and cytoprotective pathways in colonic epithelial cells in ulcerative colitis.Inhibition of Receptor Interacting Protein Kinases Attenuates Cardiomyocyte Hypertrophy Induced by Palmitic Acid.RIP1K and RIP3K provoked by shikonin induce cell cycle arrest in the triple negative breast cancer cell line, MDA-MB-468: necroptosis as a desperate programmed suicide pathway.Post-translational control of RIPK3 and MLKL mediated necroptotic cell death.Structural and molecular bases of rod photoreceptor morphogenesis and disease.Upregulation of PSMB4 is Associated with the Necroptosis after Spinal Cord Injury.RIP3 deficiency ameliorates inflammatory response in mice infected with influenza H7N9 virus infection.Active MLKL triggers the NLRP3 inflammasome in a cell-intrinsic manner.Targeting disease by immunomodulation.Receptor interacting protein 3 protects mice from high-fat diet-induced liver injuryRIP1 Inhibition Rescues from LPS-Induced RIP3-Mediated Programmed Cell Death, Distributed Energy Metabolism and Spatial Memory Impairment.The Canonical Inflammasome: A Macromolecular Complex Driving Inflammation.Ischemia Reperfusion Injury Triggers TNFα Induced-Necroptosis in Rat Retina.A FRET biosensor for necroptosis uncovers two different modes of the release of DAMPsRIPK1 is a critical modulator of both tonic and TLR-responsive inflammatory and cell death pathways in human macrophage differentiation
P2860
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P2860
More to life than death: molecular determinants of necroptotic and non-necroptotic RIP3 kinase signaling.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年学术文章
@wuu
2013年学术文章
@zh-cn
2013年学术文章
@zh-hans
2013年学术文章
@zh-my
2013年学术文章
@zh-sg
2013年學術文章
@yue
2013年學術文章
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2013年學術文章
@zh-hant
name
More to life than death: molec ...... optotic RIP3 kinase signaling.
@en
type
label
More to life than death: molec ...... optotic RIP3 kinase signaling.
@en
prefLabel
More to life than death: molec ...... optotic RIP3 kinase signaling.
@en
P1476
More to life than death: molec ...... optotic RIP3 kinase signaling.
@en
P2093
James E Vince
Nufail Khan
P356
10.1016/J.COI.2013.10.017
P577
2013-11-30T00:00:00Z