c-Jun is a JNK-independent coactivator of the PU.1 transcription factor.
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The transcriptional repressor GFI-1 antagonizes PU.1 activity through protein-protein interactionRole of activator protein-1 in the down-regulation of the human CYP2J2 gene in hypoxiaAn alternative model of H ferritin promoter transactivation by c-JunNegative cross-talk between hematopoietic regulators: GATA proteins repress PU.1Schnurri-3 (KRC) interacts with c-Jun to regulate the IL-2 gene in T cellsIdentification of amino acid residues in the ETS transcription factor Erg that mediate Erg-Jun/Fos-DNA ternary complex formationDisabled-2 is transcriptionally regulated by ICSBP and augments macrophage spreading and adhesion.Analyzing M-CSF dependent monocyte/macrophage differentiation: expression modes and meta-modes derived from an independent component analysisPotential autoregulation of transcription factor PU.1 by an upstream regulatory elementSki can negatively regulates macrophage differentiation through its interaction with PU.1.BSAP can repress enhancer activity by targeting PU.1 function.The AP-1 binding sites located in the pol gene intragenic regulatory region of HIV-1 are important for viral replicationRegulation of Ets function by protein - protein interactions.When Ets transcription factors meet their partners.Downregulation of c-Jun expression by transcription factor C/EBPalpha is critical for granulocytic lineage commitmentc-Myb and members of the c-Ets family of transcription factors act as molecular switches to mediate opposite steroid regulation of the human glucocorticoid receptor 1A promoter.Evolution of hematopoiesis: Three members of the PU.1 transcription factor family in a cartilaginous fish, Raja eglanteria.Disruption of differentiation in human cancer: AML shows the way.SHP2 tyrosine phosphatase stimulates CEBPA gene expression to mediate cytokine-dependent granulopoiesisSelection and characterization of a DNA aptamer that can discriminate between cJun/cJun and cJun/cFos.The PU.1-Modulated MicroRNA-22 Is a Regulator of Monocyte/Macrophage Differentiation and Acute Myeloid Leukemia.PU.1 and pRB interact and cooperate to repress GATA-1 and block erythroid differentiationThe Janus-faced Nature of miR-22 in Hematopoiesis: Is It an Oncogenic Tumor Suppressor or Rather a Tumor-Suppressive Oncogene?Reciprocal roles for CCAAT/enhancer binding protein (C/EBP) and PU.1 transcription factors in Langerhans cell commitmentNFATc2 recruits cJun homodimers to an NFAT site to synergistically activate interleukin-2 transcriptionActivating PTPN11 mutants promote hematopoietic progenitor cell-cycle progression and survival.Gene deregulation and spatial genome reorganization near breakpoints prior to formation of translocations in anaplastic large cell lymphoma.Lipopolysaccharide-dependent interaction between PU.1 and c-Jun determines production of lipocalin-type prostaglandin D synthase and prostaglandin D2 in macrophages.Increased c-Jun expression and reduced GATA2 expression promote aberrant monocytic differentiation induced by activating PTPN11 mutantsPU.1 and partners: regulation of haematopoietic stem cell fate in normal and malignant haematopoiesis.Zbtb1 prevents default myeloid differentiation of lymphoid-primed multipotent progenitorsAcupuncture as anticancer treatment?The role of PU.1 and GATA-1 transcription factors during normal and leukemogenic hematopoiesis.c-Jun, at the crossroad of the signaling network.Concise review: erythroid versus myeloid lineage commitment: regulating the master regulators.Recent advances in the structural molecular biology of Ets transcription factors: interactions, interfaces and inhibition.Monocytic cell differentiation from band-stage neutrophils under inflammatory conditions via MKK6 activation.Recruitment of nuclear factor Y to the inverted CCAAT element (ICE) by c-Jun and E1A stimulates basal transcription of the bone sialoprotein gene in osteosarcoma cells.Eicosapentaenoic acid demethylates a single CpG that mediates expression of tumor suppressor CCAAT/enhancer-binding protein delta in U937 leukemia cellsIdentification of a novel tumor necrosis factor alpha-responsive region in the NCF2 promoter.
P2860
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P2860
c-Jun is a JNK-independent coactivator of the PU.1 transcription factor.
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年学术文章
@wuu
1999年学术文章
@zh-cn
1999年学术文章
@zh-hans
1999年学术文章
@zh-my
1999年学术文章
@zh-sg
1999年學術文章
@yue
1999年學術文章
@zh
1999年學術文章
@zh-hant
name
c-Jun is a JNK-independent coactivator of the PU.1 transcription factor.
@en
type
label
c-Jun is a JNK-independent coactivator of the PU.1 transcription factor.
@en
prefLabel
c-Jun is a JNK-independent coactivator of the PU.1 transcription factor.
@en
P2093
P2860
P356
P1476
c-Jun is a JNK-independent coactivator of the PU.1 transcription factor
@en
P2093
P2860
P304
P356
10.1074/JBC.274.8.4939
P407
P577
1999-02-01T00:00:00Z