Src kinases mediate STAT growth pathways in squamous cell carcinoma of the head and neck.
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Preadministration of High-Dose Salicylates, Suppressors of NF- B Activation, May Increase the Chemosensitivity of Many Cancers: An Example of Proapoptotic Signal Modulation TherapySignal transducer and activator of transcription 5b: a new target of breast tumor kinase/protein tyrosine kinase 6Phosphotyrosine signaling networks in epidermal growth factor receptor overexpressing squamous carcinoma cellsThe role of Src in solid tumorsMast cell homeostasis and the JAK-STAT pathwayTumor and stromal-based contributions to head and neck squamous cell carcinoma invasionContribution of chaperones to STAT pathway signaling.Stimulation of hTAFII68 (NTD)-mediated transactivation by v-SrcSTAT3 as a therapeutic target in head and neck cancerS731 in the transactivation domain modulates STAT5b activity.Elevated Src family kinase activity stabilizes E-cadherin-based junctions and collective movement of head and neck squamous cell carcinomas.Chromium(VI) stimulates Fyn to initiate innate immune gene induction in human airway epithelial cells.The JAK2 inhibitor AZD1480 potently blocks Stat3 signaling and oncogenesis in solid tumors.Honokiol inhibits epidermal growth factor receptor signaling and enhances the antitumor effects of epidermal growth factor receptor inhibitors.Erlotinib, erlotinib-sulindac versus placebo: a randomized, double-blind, placebo-controlled window trial in operable head and neck cancer.Functional kinomics identifies candidate therapeutic targets in head and neck cancerElevated c-Src and c-Yes expression in malignant skin cancersAn endoplasmic reticulum stress-initiated sphingolipid metabolite, ceramide-1-phosphate, regulates epithelial innate immunity by stimulating β-defensin productionThe contribution of cetuximab in the treatment of recurrent and/or metastatic head and neck cancerSystematic prediction of human membrane receptor interactions.Multiple mechanisms are responsible for transactivation of the epidermal growth factor receptor in mammary epithelial cells.Constitutive activation of JAK3/STAT3 in colon carcinoma tumors and cell lines: inhibition of JAK3/STAT3 signaling induces apoptosis and cell cycle arrest of colon carcinoma cells.Dual blockade of EGFR and c-Met abrogates redundant signaling and proliferation in head and neck carcinoma cells.Differential Effects of Tyrosine Kinase Inhibitors on Normal and Oncogenic EGFR Signaling and Downstream EffectorsSRC-family kinases are activated in non-small cell lung cancer and promote the survival of epidermal growth factor receptor-dependent cell lines.Molecular parameters of head and neck cancer metastasis.STAT5A-mediated SOCS2 expression regulates Jak2 and STAT3 activity following c-Src inhibition in head and neck squamous carcinomaOpportunities and challenges facing biomarker development for personalized head and neck cancer treatment.Src Family Tyrosine Kinase Signaling Regulates FilGAP through Association with RBM10.STAT proteins as novel targets for cancer drug discovery.Signal transducers and activators of transcription: insights into the molecular basis of oral cancer.Combined inhibition of PLC{gamma}-1 and c-Src abrogates epidermal growth factor receptor-mediated head and neck squamous cell carcinoma invasion.Prognostic Significance of Serine-Phosphorylated STAT3 Expression in pT1-T2 Oral Tongue Carcinoma.Yes and Lyn play a role in nuclear translocation of the epidermal growth factor receptor.The influence of clinical and demographic risk factors on the establishment of head and neck squamous cell carcinoma cell lines.Constitutive activation of signal transducer and activator of transcription 5 contributes to tumor growth, epithelial-mesenchymal transition, and resistance to epidermal growth factor receptor targeting.Immunohistochemical analysis of phosphotyrosine signal transducer and activator of transcription 3 and epidermal growth factor receptor autocrine signaling pathways in head and neck cancers and metastatic lymph nodes.Multifocal angiostatic therapy: an update.Cross-regulation of JAK and Src kinases.Prognostic significance of cortactin levels in head and neck squamous cell carcinoma: comparison with epidermal growth factor receptor status
P2860
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P2860
Src kinases mediate STAT growth pathways in squamous cell carcinoma of the head and neck.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年学术文章
@wuu
2003年学术文章
@zh-cn
2003年学术文章
@zh-hans
2003年学术文章
@zh-my
2003年学术文章
@zh-sg
2003年學術文章
@yue
2003年學術文章
@zh
2003年學術文章
@zh-hant
name
Src kinases mediate STAT growth pathways in squamous cell carcinoma of the head and neck.
@en
type
label
Src kinases mediate STAT growth pathways in squamous cell carcinoma of the head and neck.
@en
prefLabel
Src kinases mediate STAT growth pathways in squamous cell carcinoma of the head and neck.
@en
P2093
P2860
P356
P1476
Src kinases mediate STAT growth pathways in squamous cell carcinoma of the head and neck.
@en
P2093
Edwina C Lerner
Jennifer Rubin Grandis
Joanne Kamens
Kevin F Dyer
Qing Zhang
Sichuan Xi
Thomas E Smithgall
William E Gooding
P2860
P304
31574-31583
P356
10.1074/JBC.M303499200
P407
P577
2003-05-27T00:00:00Z