Requirement of nuclear factor of activated T-cells in calcineurin-mediated cardiomyocyte hypertrophy. - Wikidata - wikimedia - TriplyDB

Requirement of nuclear factor of activated T-cells in calcineurin-mediated cardiomyocyte hypertrophy.

Requirement of nuclear factor of activated T-cells in calcineurin-mediated cardiomyocyte hypertrophy.

http://www.wikidata.org/entity/Q38363027

about

Regulation of the stability and transcriptional activity of NFATc4 by ubiquitination Rationale and design of the SMART Heart study: A prediction model for left ventricular hypertrophy in hypertension Mechanisms of altered Ca²⁺ handling in heart failure Syndecan-4 is essential for development of concentric myocardial hypertrophy via stretch-induced activation of the calcineurin-NFAT pathway PPARα activation inhibits endothelin-1-induced cardiomyocyte hypertrophy by prevention of NFATc4 binding to GATA-4 Doxorubicin activates nuclear factor of activated T-lymphocytes and Fas ligand transcription: role of mitochondrial reactive oxygen species and calcium A ternary complex of transcription factors, Nishéd and NFATc4, and co-activator p300 bound to an intronic sequence, intronic regulatory element, is pivotal for the up-regulation of myosin light chain-2v gene in cardiac hypertrophy.Isoform- and tissue-specific regulation of the Ca(2+)-sensitive transcription factor NFAT in cardiac myocytes and heart failure.MEK1 inhibits cardiac PPARα activity by direct interaction and prevents its nuclear localization.Selective inhibition of calcineurin-NFAT signaling by blocking protein-protein interaction with small organic molecules.Small engine, big power: microRNAs as regulators of cardiac diseases and regeneration.The scaffold protein muscle A-kinase anchoring protein β orchestrates cardiac myocyte hypertrophic signaling required for the development of heart failure.Peroxisome proliferator-activated receptor (PPAR) gene profiling uncovers insulin-like growth factor-1 as a PPARalpha target gene in cardioprotection.Succinate causes pathological cardiomyocyte hypertrophy through GPR91 activation.FHL1 reduces dystrophy in transgenic mice overexpressing FSHD muscular dystrophy region gene 1 (FRG1).Calcineurin/NFAT signaling represses genes Vamp1 and Vamp2 via PMCA-dependent mechanism during dopamine secretion by Pheochromocytoma cells.Targeted inhibition of calpain reduces myocardial hypertrophy and fibrosis in mouse models of type 1 diabetes Re-employment of developmental transcription factors in adult heart disease O-GlcNAc signaling is essential for NFAT-mediated transcriptional reprogramming during cardiomyocyte hypertrophy.Identification of "Multiple Components-Multiple Targets-Multiple Pathways" Associated with Naoxintong Capsule in the Treatment of Heart Diseases Using UPLC/Q-TOF-MS and Network Pharmacology Calcineurin activates cytoglobin transcription in hypoxic myocytes.Targeting the p53 pathway.Fibronectin contributes to pathological cardiac hypertrophy but not physiological growth Designing heart performance by gene transfer.Cross-talk between calcineurin/NFAT and Jak/STAT signalling induces cardioprotective alphaB-crystallin gene expression in response to hypertrophic stimuli.ANGPTL2 activity in cardiac pathologies accelerates heart failure by perturbing cardiac function and energy metabolism.Acute molecular response of mouse hindlimb muscles to chronic stimulation.Increased dosage of DYRK1A and DSCR1 delays neuronal differentiation in neocortical progenitor cells.NFAT transcription factor regulation by urocortin II in cardiac myocytes and heart failure.Mechanisms of Chinese Medicine Xinmailong's protection against heart failure in pressure-overloaded mice and cultured cardiomyocytes.Focal adhesion kinase and p53 signal transduction pathways in cancer.Calcineurin regulation of cytoskeleton organization: a new paradigm to analyse the effects of calcineurin inhibitors on the kidney.MicroRNAs in control of cardiac hypertrophy.Mechanisms of muscle growth and atrophy in mammals and Drosophila.Calcineurin signaling in the heart: The importance of time and place.Capillary electrophoretic mobility shift assay for binding of DNA with NFAT3, a transcription factor from H9c2 cardiac myoblast cells.Segregation of myoblast fusion and muscle-specific gene expression by distinct ligand-dependent inactivation of GSK-3β.Glycogen synthase kinase 3 suppresses myogenic differentiation through negative regulation of NFATc3.Atrogin-1/muscle atrophy F-box inhibits calcineurin-dependent cardiac hypertrophy by participating in an SCF ubiquitin ligase complex.MEF2 transcriptional activity maintains mitochondrial adaptation in cardiac pressure overload.

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P2860

Requirement of nuclear factor of activated T-cells in calcineurin-mediated cardiomyocyte hypertrophy.

http://www.wikidata.org/entity/Q38363027

description

2002 nî lūn-bûn

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2002年の論文

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2002年学术文章

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2002年学术文章

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2002年学术文章

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2002年学术文章

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2002年学术文章

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2002年學術文章

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2002年學術文章

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2002年學術文章

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Requirement of nuclear factor ...... ted cardiomyocyte hypertrophy.

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type

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Requirement of nuclear factor ...... ted cardiomyocyte hypertrophy.

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Requirement of nuclear factor ...... ted cardiomyocyte hypertrophy.

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Requirement of nuclear factor ...... ted cardiomyocyte hypertrophy.

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Chiel C de Theije

http://www.w3.org/2001/XMLSchema#string

Eva van Rooij

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Fawzi A Babiker

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Leon J de Windt

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Pieter A Doevendans

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P2860

Calcineurin-GATA-6 pathway is involved in smooth muscle-specific transcription.

Targeted disruption of NFATc3, but not NFATc4, reveals an intrinsic defect in calcineurin-mediated cardiac hypertrophic growth

Cabin 1, a negative regulator for calcineurin signaling in T lymphocytes

Isolation of two new members of the NF-AT gene family and functional characterization of the NF-AT proteins

A protein encoded within the Down syndrome critical region is enriched in striated muscles and inhibits calcineurin signaling

Signals transduced by Ca(2+)/calcineurin and NFATc3/c4 pattern the developing vasculature

NFAT signaling: choreographing the social lives of cells

Transcription factors of the NFAT family: regulation and function

Cardiac hypertrophy induced by mitogen-activated protein kinase kinase 7, a specific activator for c-Jun NH2-terminal kinase in ventricular muscle cells

A calcineurin-dependent transcriptional pathway for cardiac hypertrophy

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48617-48626

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scholarly article

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10.1074/JBC.M206532200

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50

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277

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Jeffery D. Molkentin

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2002-09-10T00:00:00Z

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12226086

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