Inhibition of glutamate decarboxylase (GAD) by ethyl ketopentenoate (EKP) induces treatment-resistant epileptic seizures in zebrafish.

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Inhibition of glutamate decarboxylase (GAD) by ethyl ketopentenoate (EKP) induces treatment-resistant epileptic seizures in zebrafish.

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2017 nî lūn-bûn

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2017年の論文

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2017年論文

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2017年論文

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2017年論文

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2017年論文

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2017年論文

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2017年论文

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Inhibition of glutamate decarb ...... ileptic seizures in zebrafish.

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Inhibition of glutamate decarb ...... ileptic seizures in zebrafish.

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Inhibition of glutamate decarb ...... ileptic seizures in zebrafish.

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P1476

Inhibition of glutamate decarb ...... pileptic seizures in zebrafish

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P2093

Aleksandra Siekierska

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Jubi John

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Wim Dehaen

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The Protein Data Bank

MolProbity: all-atom structure validation for macromolecular crystallography

Pharmacological characterization of an antisense knockdown zebrafish model of Dravet syndrome: inhibition of epileptic seizures by the serotonin agonist fenfluramine

UCSF Chimera--a visualization system for exploratory research and analysis

Mice lacking the 65 kDa isoform of glutamic acid decarboxylase (GAD65) maintain normal levels of GAD67 and GABA in their brains but are susceptible to seizures

Seizures induced by allylglycine, 3-mercaptopropionic acid and 4-deoxypyridoxine in mice and photosensitive baboons, and different modes of inhibition of cerebral glutamic acid decarboxylase

AutoDock4 and AutoDockTools4: Automated docking with selective receptor flexibility

LigPlot+: multiple ligand-protein interaction diagrams for drug discovery

The I-TASSER Suite: protein structure and function prediction.

Knockdown of zebrafish Lgi1a results in abnormal development, brain defects and a seizure-like behavioral phenotype

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scholarly article

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10.1038/S41598-017-06294-W

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English

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2017-08-03T00:00:00Z

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