Cross-Disease Transcriptomics: Unique IL-17A Signaling in Psoriasis Lesions and an Autoimmune PBMC Signature
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AP1S3 Mutations Cause Skin Autoinflammation by Disrupting Keratinocyte Autophagy and Up-Regulating IL-36 ProductionCytokine Imbalance as a Common Mechanism in Both Psoriasis and Rheumatoid Arthritis.Imiquimod has strain-dependent effects in mice and does not uniquely model human psoriasis.Endogenous Glucocorticoid Deficiency in Psoriasis Promotes Inflammation and Abnormal Differentiation.Signs of innate immune activation and premature immunosenescence in psoriasis patientsModels in the Research Process of Psoriasis.Molecular action of isoflavone genistein in the human epithelial cell line HaCaT.A novel joint analysis framework improves identification of differentially expressed genes in cross disease transcriptomic analysis.Induction of Alternative Proinflammatory Cytokines Accounts for Sustained Psoriasiform Skin Inflammation in IL-17C+IL-6KO Mice.Mycosis fungoides progression could be regulated by microRNAs.
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P2860
Cross-Disease Transcriptomics: Unique IL-17A Signaling in Psoriasis Lesions and an Autoimmune PBMC Signature
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
2016年论文
@zh
2016年论文
@zh-cn
name
Cross-Disease Transcriptomics: ...... d an Autoimmune PBMC Signature
@en
type
label
Cross-Disease Transcriptomics: ...... d an Autoimmune PBMC Signature
@en
prefLabel
Cross-Disease Transcriptomics: ...... d an Autoimmune PBMC Signature
@en
P2093
P2860
P31
P1476
Cross-Disease Transcriptomics: ...... d an Autoimmune PBMC Signature
@en
P2093
Johann E Gudjonsson
William R Swindell
Xianying Xing
P2860
P304
P356
10.1016/J.JID.2016.04.035
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P577
2016-05-17T00:00:00Z