As(2)O(3) enhances retroviral reverse transcription and counteracts Ref1 antiviral activity.
about
TRIM5 is an innate immune sensor for the retrovirus capsid latticeDisruption of human TRIM5alpha antiviral activity by nonhuman primate orthologuesTRIM family proteins: retroviral restriction and antiviral defenceCyclophilin A is required for TRIM5{alpha}-mediated resistance to HIV-1 in Old World monkey cellsThe human and African green monkey TRIM5alpha genes encode Ref1 and Lv1 retroviral restriction factor activitiesIntracellular immunity to HIV-1: newly defined retroviral battles inside infected cells.Patterns of evolution of host proteins involved in retroviral pathogenesis.Arsenic Trioxide Inhibits Hepatitis C Virus RNA Replication through Modulation of the Glutathione Redox System and Oxidative StressLv4 Is a Capsid-Specific Antiviral Activity in Human Blood Cells That Restricts Viruses of the SIVMAC/SIVSM/HIV-2 Lineage Prior to IntegrationCovalent modification of human immunodeficiency virus type 1 p6 by SUMO-1.Going wild: lessons from naturally occurring T-lymphotropic lentivirusesInhibition of early steps of HIV-1 replication by SNF5/Ini1.Functional evidence for the involvement of microtubules and dynein motor complexes in TRIM5α-mediated restriction of retroviruses.The cellular protein daxx interacts with avian sarcoma virus integrase and viral DNA to repress viral transcription.Arsenic counteracts human immunodeficiency virus type 1 restriction by various TRIM5 orthologues in a cell type-dependent mannerCyclophilin A and TRIM5alpha independently regulate human immunodeficiency virus type 1 infectivity in human cellsA novel aminoacid determinant of HIV-1 restriction in the TRIM5α variable 1 region isolated in a random mutagenic screenInhibition of HIV-1 infection by TNPO3 depletion is determined by capsid and detectable after viral cDNA enters the nucleus.Heterologous human immunodeficiency virus type 1 lentiviral vectors packaging a simian immunodeficiency virus-derived genome display a specific postentry transduction defect in dendritic cells.PML/TRIM19-Dependent Inhibition of Retroviral Reverse-Transcription by DaxxTRIM5 structure, HIV-1 capsid recognition, and innate immune signalingThe role of lysine 186 in HIV-1 integrase multimerizationIdentification of an arsenic-sensitive block to primate lentiviral infection of human dendritic cells.Evidence for Vpr-dependent HIV-1 replication in human CD4+ CEM.NKR T-cells.The interferon-induced antiviral protein PML (TRIM19) promotes the restriction and transcriptional silencing of lentiviruses in a context-specific, isoform-specific fashion.Interactions among infections, nutrients and xenobiotics.Inhibition of apoptosis in acute promyelocytic leukemia cells leads to increases in levels of oxidized protein and LMP2 immunoproteasome.Characterization of two distinct early post-entry blocks to HIV-1 in common marmoset lymphocytes.Cyclophilin A promotes HIV-1 reverse transcription but its effect on transduction correlates best with its effect on nuclear entry of viral cDNA.Lv1 inhibition of human immunodeficiency virus type 1 is counteracted by factors that stimulate synthesis or nuclear translocation of viral cDNASelection for loss of Ref1 activity in human cells releases human immunodeficiency virus type 1 from cyclophilin A dependence during infection.Target cell cyclophilin A modulates human immunodeficiency virus type 1 infectivityCell context-dependent involvement of ATR in early stages of retroviral replicationGene Knockout Shows That PML (TRIM19) Does Not Restrict the Early Stages of HIV-1 Infection in Human Cell LinesBoth TRIM5alpha and TRIMCyp have only weak antiviral activity in canine D17 cells.The V86M mutation in HIV-1 capsid confers resistance to TRIM5α by abrogation of cyclophilin A-dependent restriction and enhancement of viral nuclear import.Retroviral restriction factors Fv1 and TRIM5alpha act independently and can compete for incoming virus before reverse transcription.Restriction of feline immunodeficiency virus by Ref1, Lv1, and primate TRIM5alpha proteins.Isolation of an active Lv1 gene from cattle indicates that tripartite motif protein-mediated innate immunity to retroviral infection is widespread among mammals.TRIM5alpha and TRIMCyp form apparent hexamers and their multimeric state is not affected by exposure to restriction-sensitive viruses or by treatment with pharmacological inhibitors.
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As(2)O(3) enhances retroviral reverse transcription and counteracts Ref1 antiviral activity.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年学术文章
@wuu
2003年学术文章
@zh-cn
2003年学术文章
@zh-hans
2003年学术文章
@zh-my
2003年学术文章
@zh-sg
2003年學術文章
@yue
2003年學術文章
@zh
2003年學術文章
@zh-hant
name
As(2)O(3) enhances retroviral ...... racts Ref1 antiviral activity.
@en
type
label
As(2)O(3) enhances retroviral ...... racts Ref1 antiviral activity.
@en
prefLabel
As(2)O(3) enhances retroviral ...... racts Ref1 antiviral activity.
@en
P2093
P2860
P1433
P1476
As(2)O(3) enhances retroviral ...... racts Ref1 antiviral activity.
@en
P2093
Cagan Gurer
Greg J Towers
Lionel Berthoux
Paolo Salomoni
P2860
P304
P356
10.1128/JVI.77.5.3167-3180.2003
P407
P577
2003-03-01T00:00:00Z