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Transcription initiation sites and promoter structure of the human TRAIL-R3 geneStructural basis for inhibition of the histone chaperone activity of SET/TAF-Iβ by cytochrome c.Control of FLIP(L) expression and TRAIL resistance by the extracellular signal-regulated kinase1/2 pathway in breast epithelial cells.TAK1 activates AMPK-dependent cytoprotective autophagy in TRAIL-treated epithelial cells.Autophagy requires poly(adp-ribosyl)ation-dependent AMPK nuclear export.The therapeutic potential of TRAIL receptor signalling in cancer cells.The long and winding road to cancer treatment: the TRAIL system.Deciphering the insights of poly(ADP-ribosylation) in tumor progression.Delaying mitotic exit downregulates FLIP expression and strongly sensitizes tumor cells to TRAIL.Activated ERBB2/HER2 licenses sensitivity to apoptosis upon endoplasmic reticulum stress through a PERK-dependent pathway.ER stress sensitizes cells to TRAIL through down-regulation of FLIP and Mcl-1 and PERK-dependent up-regulation of TRAIL-R2.Itch/AIP4-independent proteasomal degradation of cFLIP induced by the histone deacetylase inhibitor SAHA sensitizes breast tumour cells to TRAIL.Cellular FLIP(L) plays a survival role and regulates morphogenesis in breast epithelial cells.Opposing roles of TGF-β and EGF in the regulation of TRAIL-induced apoptosis in human breast epithelial cells.Mitotic arrest and JNK-induced proteasomal degradation of FLIP and Mcl-1 are key events in the sensitization of breast tumor cells to TRAIL by antimicrotubule agents.AMPK-independent down-regulation of cFLIP and sensitization to TRAIL-induced apoptosis by AMPK activators.Down-regulation of RIP expression by 17-dimethylaminoethylamino-17-demethoxygeldanamycin promotes TRAIL-induced apoptosis in breast tumor cells.Statins activate a mitochondria-operated pathway of apoptosis in breast tumor cells by a mechanism regulated by ErbB2 and dependent on the prenylation of proteins.Roscovitine sensitizes breast cancer cells to TRAIL-induced apoptosis through a pleiotropic mechanism.Flavopiridol induces cellular FLICE-inhibitory protein degradation by the proteasome and promotes TRAIL-induced early signaling and apoptosis in breast tumor cells.Thymidylate synthase inhibition triggers glucose-dependent apoptosis in p53-negative leukemic cells.Tumor necrosis factor-related apoptosis-inducing ligand (TRAIL) decoy receptor TRAIL-R3 is up-regulated by p53 in breast tumor cells through a mechanism involving an intronic p53-binding site.Inhibition of glucose metabolism sensitizes tumor cells to death receptor-triggered apoptosis through enhancement of death-inducing signaling complex formation and apical procaspase-8 processing.Growth factors as survival factors: regulation of apoptosis.The control of apoptosis in mammalian cells.Overexpression of a heterologous thymidine kinase delays apoptosis induced by factor deprivation and inhibitors of deoxynucleotide metabolism.dNTP pools imbalance as a signal to initiate apoptosis.Reactive oxygen intermediate(s) (ROI): common mediator(s) of poly(ADP-ribose)polymerase (PARP) cleavage and apoptosis.Activation-induced apoptosis in Jurkat cells through a myc-independent mechanism.Interleukin-3 and Bcl-2 cooperatively inhibit etoposide-induced apoptosis in a murine pre-B cell line.Bcl-2 oncogene protects a bone marrow-derived pre-B-cell line from 5'-fluor,2'-deoxyuridine-induced apoptosis.Apoptosis in human thymocytes after treatment with glucocorticoids.A monoclonal antibody to CD11c antigen inhibits the production of superoxide anion induced by concanavalin A in PMA-differentiated U-937 cells.Mitochondria-dependent and -independent mechanisms in tumour necrosis factor-related apoptosis-inducing ligand (TRAIL)-induced apoptosis are both regulated by interferon-gamma in human breast tumour cells.Phorbol esters and diacylglycerol inhibit vasopressin-induced increases in cytoplasmic-free Ca2+ and 45Ca2+ efflux in Swiss 3T3 cells.Serum rapidly mobilizes calcium from an intracellular pool in quiescent fibroblastic cells.Autophagy inhibition sensitizes multiple myeloma cells to 17-dimethylaminoethylamino-17-demethoxygeldanamycin-induced apoptosis.Doxorubicin induces apoptosis and CD95 gene expression in human primary endothelial cells through a p53-dependent mechanism.Glucocorticoids activate a suicide program in mature T lymphocytes: protective action of interleukin-2.The mitogen-activated protein kinase pathway can inhibit TRAIL-induced apoptosis by prohibiting association of truncated Bid with mitochondria.
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P50
description
hulumtues
@sq
onderzoeker
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researcher
@en
հետազոտող
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name
Abelardo López-Rivas
@ast
Abelardo López-Rivas
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Abelardo López-Rivas
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Abelardo López-Rivas
@nl
Abelardo López-Rivas
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type
label
Abelardo López-Rivas
@ast
Abelardo López-Rivas
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Abelardo López-Rivas
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Abelardo López-Rivas
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Abelardo López-Rivas
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prefLabel
Abelardo López-Rivas
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Abelardo López-Rivas
@en
Abelardo López-Rivas
@es
Abelardo López-Rivas
@nl
Abelardo López-Rivas
@sl
P1053
E-5115-2015
P106
P1153
7006074182
P21
P31
P3829
P496
0000-0002-9351-9690