Translational inhibition and increased interferon induction in cells infected with C protein-deficient measles virus.
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Measles virus V protein is a decoy substrate for IkappaB kinase alpha and prevents Toll-like receptor 7/9-mediated interferon inductionBoth RIG-I and MDA5 RNA Helicases Contribute to the Induction of Alpha/Beta Interferon in Measles Virus-Infected Human CellsIntracellular transport of the measles virus ribonucleoprotein complex is mediated by Rab11A-positive recycling endosomes and drives virus release from the apical membrane of polarized epithelial cells.Mechanisms of protein kinase PKR-mediated amplification of beta interferon induction by C protein-deficient measles virusInterplay between innate immunity and negative-strand RNA viruses: towards a rational modelDistinct and overlapping roles of Nipah virus P gene products in modulating the human endothelial cell antiviral response.A recombinant measles virus unable to antagonize STAT1 function cannot control inflammation and is attenuated in rhesus monkeys.The V protein of canine distemper virus is required for virus replication in human epithelial cells.Evolution and structural organization of the C proteins of paramyxovirinae.Expression of the Sendai (murine parainfluenza) virus C protein alleviates restriction of measles virus growth in mouse cells.Antagonism of innate immunity by paramyxovirus accessory proteins.Protein kinase PKR catalytic activity is required for the PKR-dependent activation of mitogen-activated protein kinases and amplification of interferon beta induction following virus infection.A correlation of measles specific antibodies and the number of plasmacytoid dendritic cells is observed after measles vaccination in 9 month old infants.Actin-Modulating Protein Cofilin Is Involved in the Formation of Measles Virus Ribonucleoprotein Complex at the Perinuclear Region.Nonstructural Nipah virus C protein regulates both the early host proinflammatory response and viral virulence.A human lung carcinoma cell line supports efficient measles virus growth and syncytium formation via a SLAM- and CD46-independent mechanism.Control of the induction of type I interferon by Peste des petits ruminants virus.Wild type measles virus attenuation independent of type I IFN.Attenuation of V- or C-defective measles viruses: infection control by the inflammatory and interferon responses of rhesus monkeys.Sendai virus C protein plays a role in restricting PKR activation by limiting the generation of intracellular double-stranded RNA.Protein kinase PKR mediates the apoptosis induction and growth restriction phenotypes of C protein-deficient measles virus.Measles virus nonstructural C protein modulates viral RNA polymerase activity by interacting with host protein SHCBP1N-terminally truncated C protein, CNDelta25, of human parainfluenza virus type 3 is a potent inhibitor of viral replication.Measles virus C protein impairs production of defective copyback double-stranded viral RNA and activation of protein kinase R.Differential cellular immune responses to wild-type and attenuated edmonston tag measles virus strains are primarily defined by the viral phosphoprotein gene.The regulation of type I interferon production by paramyxoviruses.Paramyxovirus activation and inhibition of innate immune responses.The measles virus phosphoprotein interacts with the linker domain of STAT1.Measles virus C protein interferes with Beta interferon transcription in the nucleus.A highly attenuated measles virus vaccine strain encodes a fully functional C proteinThe matrix protein of measles virus regulates viral RNA synthesis and assembly by interacting with the nucleocapsid protein.Paramyxovirus-induced shutoff of host and viral protein synthesis: role of the P and V proteins in limiting PKR activation.Non-encapsidated 5' copy-back defective-interfering genomes produced by recombinant measles viruses are recognized by RIG-I and LGP2 but not MDA5.Altered interaction of the matrix protein with the cytoplasmic tail of hemagglutinin modulates measles virus growth by affecting virus assembly and cell-cell fusion.Measles virus circumvents the host interferon response by different actions of the C and V proteins.Modified vaccinia virus Ankara induces Toll-like receptor-independent type I interferon responses.Remarkable similarity in genome nucleotide sequences between the Schwarz FF-8 and AIK-C measles virus vaccine strains and apparent nucleotide differences in the phosphoprotein gene.Annexin A2 Mediates the Localization of Measles Virus Matrix Protein at the Plasma Membrane.Measles
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P2860
Translational inhibition and increased interferon induction in cells infected with C protein-deficient measles virus.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年学术文章
@wuu
2006年学术文章
@zh-cn
2006年学术文章
@zh-hans
2006年学术文章
@zh-my
2006年学术文章
@zh-sg
2006年學術文章
@yue
2006年學術文章
@zh
2006年學術文章
@zh-hant
name
Translational inhibition and i ...... otein-deficient measles virus.
@en
Translational inhibition and i ...... otein-deficient measles virus.
@nl
type
label
Translational inhibition and i ...... otein-deficient measles virus.
@en
Translational inhibition and i ...... otein-deficient measles virus.
@nl
prefLabel
Translational inhibition and i ...... otein-deficient measles virus.
@en
Translational inhibition and i ...... otein-deficient measles virus.
@nl
P2093
P2860
P356
P1433
P1476
Translational inhibition and i ...... otein-deficient measles virus.
@en
P2093
Ritsuko Koga
Shinji Ohno
Yuichiro Nakatsu
P2860
P304
11861-11867
P356
10.1128/JVI.00751-06
P407
P577
2006-09-20T00:00:00Z