Highly divergent lentiviral Tat proteins activate viral gene expression by a common mechanism
about
Mice transgenic for equine cyclin T1 and ELR1 are susceptible to equine infectious anemia virus infectionCDK9 autophosphorylation regulates high-affinity binding of the human immunodeficiency virus type 1 tat-P-TEFb complex to TAR RNAStructural insights into the cyclin T1-Tat-TAR RNA transcription activation complex from EIAVTFIIH inhibits CDK9 phosphorylation during human immunodeficiency virus type 1 transcriptionP-TEFb, a cyclin-dependent kinase controlling elongation by RNA polymerase IIInhibition of Tat-mediated transactivation and HIV-1 replication by human anti-hCyclinT1 intrabodies.The human endogenous retrovirus K Rev response element coincides with a predicted RNA folding region.Flavopiridol inhibits P-TEFb and blocks HIV-1 replication.CDK9 inhibitor FIT-039 prevents replication of multiple DNA viruses.Flavopiridol inactivates P-TEFb and blocks most RNA polymerase II transcription in vivo.High natural permissivity of primary rabbit cells for HIV-1, with a virion infectivity defect in macrophages as the final replication barrier.HIV Tat, its TARgets and the control of viral gene expression.Replication of equine infectious anemia virus in engineered mouse NIH 3T3 cells.Making a Short Story Long: Regulation of P-TEFb and HIV-1 Transcriptional Elongation in CD4+ T Lymphocytes and Macrophages.Development of an equine-tropic replication-competent lentivirus assay for equine infectious anemia virus-based lentiviral vectors.Interactions between equine cyclin T1, Tat, and TAR are disrupted by a leucine-to-valine substitution found in human cyclin T1.Functional differences between human and bovine immunodeficiency virus Tat transcription factors.Binding of Tat to TAR and recruitment of positive transcription elongation factor b occur independently in bovine immunodeficiency virus.Cyclin T1 expression is mediated by a complex and constitutively active promoter and does not limit human immunodeficiency virus type 1 Tat function in unstimulated primary lymphocytesTransient induction of cyclin T1 during human macrophage differentiation regulates human immunodeficiency virus type 1 Tat transactivation function.A replication competent lentivirus (RCL) assay for equine infectious anaemia virus (EIAV)-based lentiviral vectors.Human immunodeficiency virus type 1 subtype C exhibits higher transactivation activity of Tat than subtypes B and E.Mechanism of H-8 inhibition of cyclin-dependent kinase 9: study using inhibitor-immobilized matrices.
P2860
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P2860
Highly divergent lentiviral Tat proteins activate viral gene expression by a common mechanism
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年学术文章
@wuu
1999年学术文章
@zh-cn
1999年学术文章
@zh-hans
1999年学术文章
@zh-my
1999年学术文章
@zh-sg
1999年學術文章
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1999年學術文章
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1999年學術文章
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name
Highly divergent lentiviral Ta ...... pression by a common mechanism
@en
Highly divergent lentiviral Ta ...... ression by a common mechanism.
@nl
type
label
Highly divergent lentiviral Ta ...... pression by a common mechanism
@en
Highly divergent lentiviral Ta ...... ression by a common mechanism.
@nl
prefLabel
Highly divergent lentiviral Ta ...... pression by a common mechanism
@en
Highly divergent lentiviral Ta ...... ression by a common mechanism.
@nl
P2093
P2860
P356
P1476
Highly divergent lentiviral Ta ...... pression by a common mechanism
@en
P2093
P2860
P304
P356
10.1128/MCB.19.7.4592
P407
P577
1999-07-01T00:00:00Z