Raf-MEK-Erk cascade in anoikis is controlled by Rac1 and Cdc42 via Akt.
about
Extracellular signal-regulated kinase 2 is necessary for mesoderm differentiationLaminin-10/11 and fibronectin differentially prevent apoptosis induced by serum removal via phosphatidylinositol 3-kinase/Akt- and MEK1/ERK-dependent pathwaysCdc42 is not essential for filopodium formation, directed migration, cell polarization, and mitosis in fibroblastoid cellsLysophosphatidic acid induces MDA-MB-231 breast cancer cells migration through activation of PI3K/PAK1/ERK signaling.miR-137 targets Cdc42 expression, induces cell cycle G1 arrest and inhibits invasion in colorectal cancer cells.PTEN inhibition improves wound healing in lung epithelia through changes in cellular mechanics that enhance migrationColon cancer cell-derived high mobility group 1/amphoterin induces growth inhibition and apoptosis in macrophagesp70 S6-kinase mediates the cooperation between Akt1 and Mek1 pathways in fibroblast-mediated extracellular matrix remodelingFamilies of microRNAs Expressed in Clusters Regulate Cell Signaling in Cervical Cancer.Phosphorylation of EBP50 negatively regulates β-PIX-dependent Rac1 activity in anoikis.Higher polymerase activity of a human influenza virus enhances activation of the hemagglutinin-induced Raf/MEK/ERK signal cascade.Anoikis effector Bit1 negatively regulates Erk activity.Cdc42 deficiency induces podocyte apoptosis by inhibiting the Nwasp/stress fibers/YAP pathway.Rac GTPase isoforms Rac1 and Rac2 play a redundant and crucial role in T-cell development.Plasma Fibronectin Promotes Tumor Cell Survival and Invasion through Regulation of Tie2.Role of PKCα activation of Src, PI-3K/AKT, and ERK in EGF-stimulated proliferation of rat and human conjunctival goblet cells.Silencing of D4-GDI inhibits growth and invasive behavior in MDA-MB-231 cells by activation of Rac-dependent p38 and JNK signalingRho GTPase Cdc42 is essential for B-lymphocyte development and activation.Downregulation of ARHGDIA contributes to human glioma progression through activation of Rho GTPase signaling pathway.ERK and cell death: mechanisms of ERK-induced cell death--apoptosis, autophagy and senescence.P-cadherin promotes ovarian cancer dissemination through tumor cell aggregation and tumor-peritoneum interactions.MiR-384 inhibits human colorectal cancer metastasis by targeting KRAS and CDC42.Hepatitis B Virus X Protein Stimulates Proliferation, Wound Closure and Inhibits Apoptosis of HuH-7 Cells via CDC42.Integration of autophagy and anoikis resistance in solid tumors.Constitutive ERK MAPK activity regulates macrophage ATP production and mitochondrial integrityDivergent behaviors and underlying mechanisms of cell migration and invasion in non-metastatic T24 and its metastatic derivative T24T bladder cancer cell linesDifferential expression of miR-195 in esophageal squamous cell carcinoma and miR-195 expression inhibits tumor cell proliferation and invasion by targeting of Cdc42.WNT-5A triggers Cdc42 activation leading to an ERK1/2 dependent decrease in MMP9 activity and invasive migration of breast cancer cells.miR-137 inhibits the proliferation of lung cancer cells by targeting Cdc42 and Cdk6.Cdc42 negatively regulates intrinsic migration of highly aggressive breast cancer cells.miR-137 is frequently down-regulated in gastric cancer and is a negative regulator of Cdc42.E7 abolishes raf-induced arrest via mislocalization of p21(Cip1).The Rho GTPase effector ROCK regulates cyclin A, cyclin D1, and p27Kip1 levels by distinct mechanisms.c-Fos is a mediator of the c-myc-induced apoptotic signaling in serum-deprived hepatoma cells via the p38 mitogen-activated protein kinase pathway.Targeted nanoconjugate co-delivering siRNA and tyrosine kinase inhibitor to KRAS mutant NSCLC dissociates GAB1-SHP2 post oncogene knockdownSuppression of extracellular signal-regulated kinase activity in herpes simplex virus 1-infected cells by the Us3 protein kinaseMechanical compression of articular cartilage induces chondrocyte proliferation and inhibits proteoglycan synthesis by activation of the ERK pathway: implications for tissue engineering and regenerative medicineRac1 pathway mediates stretch response in pulmonary alveolar epithelial cells.MAP kinases regulate unfertilized egg apoptosis and fertilization suppresses death via Ca2+ signaling.Identification of Rho GTPases implicated in terminal differentiation of muscle cells in ascidia.
P2860
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P2860
Raf-MEK-Erk cascade in anoikis is controlled by Rac1 and Cdc42 via Akt.
description
2001 nî lūn-bûn
@nan
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
2001年论文
@zh
2001年论文
@zh-cn
name
Raf-MEK-Erk cascade in anoikis is controlled by Rac1 and Cdc42 via Akt.
@en
Raf-MEK-Erk cascade in anoikis is controlled by Rac1 and Cdc42 via Akt.
@nl
type
label
Raf-MEK-Erk cascade in anoikis is controlled by Rac1 and Cdc42 via Akt.
@en
Raf-MEK-Erk cascade in anoikis is controlled by Rac1 and Cdc42 via Akt.
@nl
prefLabel
Raf-MEK-Erk cascade in anoikis is controlled by Rac1 and Cdc42 via Akt.
@en
Raf-MEK-Erk cascade in anoikis is controlled by Rac1 and Cdc42 via Akt.
@nl
P2093
P2860
P1476
Raf-MEK-Erk cascade in anoikis is controlled by Rac1 and Cdc42 via Akt.
@en
P2093
C Peyssonnaux
T F Franke
P2860
P304
P356
10.1128/MCB.21.19.6706-6717.2001
P407
P577
2001-10-01T00:00:00Z