The ability of positive transcription elongation factor B to transactivate human immunodeficiency virus transcription depends on a functional kinase domain, cyclin T1, and Tat.
about
The histone chaperone protein Nucleosome Assembly Protein-1 (hNAP-1) binds HIV-1 Tat and promotes viral transcriptionAcetylation of conserved lysines in the catalytic core of cyclin-dependent kinase 9 inhibits kinase activity and regulates transcriptionCDK9 autophosphorylation regulates high-affinity binding of the human immunodeficiency virus type 1 tat-P-TEFb complex to TAR RNADomains in the SPT5 protein that modulate its transcriptional regulatory propertiesThe growth factor granulin interacts with cyclin T1 and modulates P-TEFb-dependent transcriptionCrystal structure of HIV-1 Tat complexed with human P-TEFbRecruitment of human cyclin T1 to nuclear bodies through direct interaction with the PML proteinRegulation of P-TEFb elongation complex activity by CDK9 acetylationTat gets the "green" light on transcription initiationRoles and functions of HIV-1 Tat protein in the CNS: an overviewTranscriptional control of HIV latency: cellular signaling pathways, epigenetics, happenstance and the hope for a cureMolecular dynamics simulation and experimental verification of the interaction between cyclin T1 and HIV-1 Tat proteinsDynamics of human immunodeficiency virus transcription: P-TEFb phosphorylates RD and dissociates negative effectors from the transactivation response elementRNA polymerase is poised for activation across the genomeP-TEFb, a cyclin-dependent kinase controlling elongation by RNA polymerase IISelection of TAR RNA-binding chameleon peptides by using a retroviral replication system.Control of stochastic gene expression by host factors at the HIV promoter.Cell cycle-regulated transcription by the human immunodeficiency virus type 1 Tat transactivator.ICP22 and the UL13 protein kinase are both required for herpes simplex virus-induced modification of the large subunit of RNA polymerase II.Antiapoptotic function of Cdk9 (TAK/P-TEFb) in U937 promonocytic cells.Induction of TAK (cyclin T1/P-TEFb) in purified resting CD4(+) T lymphocytes by combination of cytokines.Ubiquitylation of Cdk9 by Skp2 facilitates optimal Tat transactivation.Genetic analysis of the structure and function of 7SK small nuclear ribonucleoprotein (snRNP) in cells.TAR RNA loop: a scaffold for the assembly of a regulatory switch in HIV replication.Transcriptional and posttranscriptional regulation of HIV-1 gene expressionAn analog of the natural steroidal alkaloid cortistatin A potently suppresses Tat-dependent HIV transcription.High natural permissivity of primary rabbit cells for HIV-1, with a virion infectivity defect in macrophages as the final replication barrier.The RNA polymerase II CTD coordinates transcription and RNA processing.Cyclin T1/CDK9 interacts with influenza A virus polymerase and facilitates its association with cellular RNA polymerase II.Replication of human immunodeficiency viruses engineered with heterologous Tat-transactivation response element interactions.Polo-like kinase 1 inhibits the activity of positive transcription elongation factor of RNA Pol II b (P-TEFb).Interactions between human cyclin T, Tat, and the transactivation response element (TAR) are disrupted by a cysteine to tyrosine substitution found in mouse cyclin TEffect of SWI/SNF chromatin remodeling complex on HIV-1 Tat activated transcription.Transcriptional profiles of latent human immunodeficiency virus in infected individuals: effects of Tat on the host and reservoir.Human topoisomerase I promotes HIV-1 proviral DNA synthesis: implications for the species specificity and cellular tropism of HIV-1 infection.BRCA1 functions as a novel transcriptional cofactor in HIV-1 infectionThe interaction between HIV-1 Tat and human cyclin T1 requires zinc and a critical cysteine residue that is not conserved in the murine CycT1 proteinStable Phenotypic Changes of the Host T Cells Are Essential to the Long-Term Stability of Latent HIV-1 Infection.Activation of P-TEFb at sites of dual HIV/TB infection, and inhibition of MTB-induced HIV transcriptional activation by the inhibitor of CDK9, Indirubin-3'-monoxime.Tat competes with HEXIM1 to increase the active pool of P-TEFb for HIV-1 transcription.
P2860
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P2860
The ability of positive transcription elongation factor B to transactivate human immunodeficiency virus transcription depends on a functional kinase domain, cyclin T1, and Tat.
description
1998 nî lūn-bûn
@nan
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
1998年论文
@zh
1998年论文
@zh-cn
name
The ability of positive transc ...... se domain, cyclin T1, and Tat.
@en
The ability of positive transc ...... se domain, cyclin T1, and Tat.
@nl
type
label
The ability of positive transc ...... se domain, cyclin T1, and Tat.
@en
The ability of positive transc ...... se domain, cyclin T1, and Tat.
@nl
prefLabel
The ability of positive transc ...... se domain, cyclin T1, and Tat.
@en
The ability of positive transc ...... se domain, cyclin T1, and Tat.
@nl
P2093
P2860
P1433
P1476
The ability of positive transc ...... se domain, cyclin T1, and Tat.
@en
P2093
B M Peterlin
K Fujinaga
P2860
P304
P407
P577
1998-09-01T00:00:00Z