Two functionally distinct forms of a retroviral receptor explain the nonreciprocal receptor interference among subgroups B, D, and E avian leukosis viruses.
about
Cellular transcription factor ZASC1 regulates murine leukemia virus transcriptionAntibody blockade of the Cripto CFC domain suppresses tumor cell growth in vivoUse of different but overlapping determinants in a retrovirus receptor accounts for non-reciprocal interference between xenotropic and polytropic murine leukemia viruses.The host cell sulfonation pathway contributes to retroviral infection at a step coincident with provirus establishmentThe receptor for the subgroup C avian sarcoma and leukosis viruses, Tvc, is related to mammalian butyrophilins, members of the immunoglobulin superfamily.Receptors and entry cofactors for retroviruses include single and multiple transmembrane-spanning proteins as well as newly described glycophosphatidylinositol-anchored and secreted proteins.Resistance to infection by subgroups B, D, and E avian sarcoma and leukosis viruses is explained by a premature stop codon within a resistance allele of the tvb receptor gene.Two different molecular defects in the Tva receptor gene explain the resistance of two tvar lines of chickens to infection by subgroup A avian sarcoma and leukosis viruses.Characterization of a cytolytic strain of equine infectious anemia virus.Mechanisms of avian retroviral host range extension.Intronic deletions of tva receptor gene decrease the susceptibility to infection by avian sarcoma and leukosis virus subgroup A.Intronic deletions that disrupt mRNA splicing of the tva receptor gene result in decreased susceptibility to infection by avian sarcoma and leukosis virus subgroup A.A single-amino-acid substitution in the TvbS1 receptor results in decreased susceptibility to infection by avian sarcoma and leukosis virus subgroups B and D and resistance to infection by subgroup E in vitro and in vivo.Efficient subgroup C avian sarcoma and leukosis virus receptor activity requires the IgV domain of the Tvc receptor and proper display on the cell membraneDevelopment and application of a multiplex PCR method for rapid differential detection of subgroup A, B, and J avian leukosis viruses.Molecular events accompanying rous sarcoma virus rescue from rodent cells and the role of viral gene complementation.Sequencing the Biology of Entry: The Retroviral env Gene.A fifteen-amino-acid TVB peptide serves as a minimal soluble receptor for subgroup B avian leukosis and sarcoma viruses.An equine infectious anemia virus variant superinfects cells through novel receptor interactions.Acquisition of resistance to avian leukosis virus subgroup B through mutations on tvb cysteine-rich domains in DF-1 chicken fibroblasts.The hr1 and fusion peptide regions of the subgroup B avian sarcoma and leukosis virus envelope glycoprotein influence low pH-dependent membrane fusion.Isolation of cell lines that show novel, murine leukemia virus-specific blocks to early steps of retroviral replicationLow pH is required for avian sarcoma and leukosis virus Env-induced hemifusion and fusion pore formation but not for pore growth.Identification of a linear B-cell epitope on the avian leukosis virus P27 protein using monoclonal antibodies.Identification of a novel B-cell epitope specific for avian leukosis virus subgroup J gp85 protein.Model of the TVA receptor determinants required for efficient infection by subgroup A avian sarcoma and leukosis viruses.Inhibition of superinfection and the evolution of viral latencyNonconserved tryptophan 38 of the cell surface receptor for subgroup J avian leukosis virus discriminates sensitive from resistant avian species.Development and validation of a PCR-RFLP assay to evaluate TVB haplotypes coding receptors for subgroup B and subgroup E avian leukosis viruses in White Leghorns.Genetic variation at the tumour virus B locus in commercial and laboratory chicken populations assessed by a medium-throughput or a high-throughput assay.Transmission, Evolution, and Endogenization: Lessons Learned from Recent Retroviral Invasions.Naturally Occurring Frame-Shift Mutations in the tvb Receptor Gene Are Responsible for Decreased Susceptibility of Chicken to Infection with Avian Leukosis Virus Subgroups B, D, and E.
P2860
Q24314142-A1642DC0-B749-432B-9828-CA9732020D3FQ24316195-B2CAFAC0-ECC6-47D0-98FF-BD466056AA15Q25255014-5A7BF1F9-6E2F-4A40-A497-6E5560393A44Q28473991-AA723828-271A-42CF-825A-75C1CA91B751Q33911871-EC0BF609-74D7-4085-9A25-70605FE93B7AQ34010373-81606F17-9225-4465-9F05-DFB240382EF1Q34343436-4FF70BE6-65E2-402E-ACC3-79786433C485Q34371305-99218307-2654-47E0-A8CD-A8A7C3D1A6BAQ34470748-0A6A074A-BEBB-4D95-B38B-5D1BE647B200Q35020496-2727BE3C-9771-4D51-99DD-518FAE951691Q35422927-C86755B2-1540-4FCB-8FA5-9071954A174FQ35826549-554D50C8-58C4-43C9-8E77-100B8618B131Q36483815-5A50C234-64FF-4ADC-94C7-463DE054DC04Q36949793-E88B6B93-7B59-4F79-92EF-6F45C3DE2848Q37546730-96B8D8A0-307C-413F-A19E-B60E430D1724Q38326833-4035853A-2776-44C4-A17C-D49087A94820Q39422249-89FFAA6D-BEDB-49FF-A7F2-3AE4EF228665Q39686823-47CC96AD-D961-4E55-9BF5-D34F45C883A1Q39955743-B7745EBC-1BFB-449A-9FAC-D4356B64752EQ40042857-94CD5802-7DD5-4EDC-9751-D56607E45693Q40179783-B2D3FD33-EE27-495D-A729-67DFD34DEE08Q40368031-47082BD1-0235-4E8F-A974-BEB26001CF1AQ40579178-95C9CE63-01E4-40D6-B5E1-16D38E116C29Q40606565-2A49C3E4-3EDA-4514-8B2B-2CA11C0F97BFQ41473947-6FA97F2A-AD1F-4FFE-ACF1-F0BC487D9FA9Q41619347-3CD57CA3-E148-4F72-92EF-FB3E28E0EF20Q42083954-1FD663E6-1CF4-4951-8568-7D555632E395Q43079602-7FF7DF94-D74F-46B4-A1EB-62B4BB1A2375Q45118666-4E155A21-60F6-49C2-B4F1-B65748CE22C9Q45402135-66C616D9-38E2-4E99-9BFD-A91293D6EABCQ46241929-BD4054B2-DA0F-4A9A-B85C-3E681EC26ABEQ47230811-1843D4C2-0DFE-4A65-A72C-8D8BC5D42FD5
P2860
Two functionally distinct forms of a retroviral receptor explain the nonreciprocal receptor interference among subgroups B, D, and E avian leukosis viruses.
description
2001 nî lūn-bûn
@nan
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
2001年论文
@zh
2001年论文
@zh-cn
name
Two functionally distinct form ...... and E avian leukosis viruses.
@en
Two functionally distinct form ...... and E avian leukosis viruses.
@nl
type
label
Two functionally distinct form ...... and E avian leukosis viruses.
@en
Two functionally distinct form ...... and E avian leukosis viruses.
@nl
prefLabel
Two functionally distinct form ...... and E avian leukosis viruses.
@en
Two functionally distinct form ...... and E avian leukosis viruses.
@nl
P2093
P2860
P1433
P1476
Two functionally distinct form ...... and E avian leukosis viruses.
@en
P2093
H B Adkins
S C Blacklow
P2860
P304
P356
10.1128/JVI.75.8.3520-3526.2001
P407
P577
2001-04-01T00:00:00Z