Receptor for advanced glycation end-products signals through Ras during tobacco smoke-induced pulmonary inflammation.
about
RAGE: a new frontier in chronic airways diseaseImpacts of cigarette smoking on immune responsiveness: Up and down or upside down?Plausible Roles for RAGE in Conditions Exacerbated by Direct and Indirect (Secondhand) Smoke Exposure.The role of 8-oxoguanine DNA glycosylase-1 in inflammationAntenatal exposure of maternal secondhand smoke (SHS) increases fetal lung expression of RAGE and induces RAGE-mediated pulmonary inflammation.The receptor for advanced glycation end products (RAGE) contributes to the progression of emphysema in miceCigarette smoking and inflammation: cellular and molecular mechanisms.RAGE and tobacco smoke: insights into modeling chronic obstructive pulmonary disease.Antiglycation Activity of Iridoids and Their Food SourcesEffect of chronic hypoxia on RAGE and its soluble forms in lungs and plasma of mice.Association of polymorphisms of the receptor for advanced glycation end products gene with COPD in the Chinese population.Circulating soluble receptor for advanced glycation end products (sRAGE) as a biomarker of emphysema and the RAGE axis in the lung.Blockade of RAGE ameliorates elastase-induced emphysema development and progression via RAGE-DAMP signaling.Primary alveolar macrophages exposed to diesel particulate matter increase RAGE expression and activate RAGE signaling.Targeting Inflammation in Cancer Prevention and Therapy.All the "RAGE" in lung disease: The receptor for advanced glycation endproducts (RAGE) is a major mediator of pulmonary inflammatory responses.RAGE/NF-κB signaling mediates lipopolysaccharide induced acute lung injury in neonate rat model.Receptor for advanced glycation end-products and World Trade Center particulate induced lung function loss: a case-cohort study and murine model of acute particulate exposureConditional over-expression of RAGE by embryonic alveolar epithelium compromises the respiratory membrane and impairs endothelial cell differentiation.Conditionally induced RAGE expression by proximal airway epithelial cells in transgenic mice causes lung inflammation.A modular cell-type focused inflammatory process network model for non-diseased pulmonary tissue.RAGE/NF-κB pathway mediates lipopolysaccharide-induced inflammation in alveolar type I epithelial cells isolated from neonate rats.Inhibition of the receptor for advanced glycation end-products (RAGE) protects from secondhand smoke (SHS)-induced intrauterine growth restriction IUGR in mice.Emphysema requires the receptor for advanced glycation end-products triggering on structural cells.Effect of long-term maternal smoking on the offspring's lung health.Cardiomyocyte mitochondrial respiration is reduced by receptor for advanced glycation end-product signaling in a ceramide-dependent manner.Acute secondhand smoke-induced pulmonary inflammation is diminished in RAGE knockout mice.Conditional overexpression of receptors for advanced glycation end-products in the adult murine lung causes airspace enlargement and induces inflammation.Mesenchymal stem cells protects hyperoxia-induced lung injury in newborn rats via inhibiting receptor for advanced glycation end-products/nuclear factor κB signaling.RAGE-ligands axis: A new 'driving force' for cigarette smoke-induced airway inflammation in COPD?Iridoids are natural glycation inhibitors.RAGE signaling by alveolar macrophages influences tobacco smoke-induced inflammation.Embryonic overexpression of receptors for advanced glycation end-products by alveolar epithelium induces an imbalance between proliferation and apoptosis.COPD as an endothelial disorder: endothelial injury linking lesions in the lungs and other organs? (2017 Grover Conference Series).Overexpression of RAGE contributes to cigarette smoke-induced nitric oxide generation in COPD.Tobacco Smoke Induces and Alters Immune Responses in the Lung Triggering Inflammation, Allergy, Asthma and Other Lung Diseases: A Mechanistic Review.The Interplay Between Immune Response and Bacterial Infection in COPD: Focus Upon Non-typeable
P2860
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P2860
Receptor for advanced glycation end-products signals through Ras during tobacco smoke-induced pulmonary inflammation.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
2010年论文
@zh
2010年论文
@zh-cn
name
Receptor for advanced glycatio ...... nduced pulmonary inflammation.
@en
Receptor for advanced glycatio ...... nduced pulmonary inflammation.
@nl
type
label
Receptor for advanced glycatio ...... nduced pulmonary inflammation.
@en
Receptor for advanced glycatio ...... nduced pulmonary inflammation.
@nl
prefLabel
Receptor for advanced glycatio ...... nduced pulmonary inflammation.
@en
Receptor for advanced glycatio ...... nduced pulmonary inflammation.
@nl
P2093
P2860
P356
P1476
Receptor for advanced glycatio ...... induced pulmonary inflammation
@en
P2093
John R Hoidal
Robert E Schmitt
Stephen D Kasteler
P2860
P304
P356
10.1165/RCMB.2010-0231OC
P577
2010-12-03T00:00:00Z