Targeting STAT1 by myricetin and delphinidin provides efficient protection of the heart from ischemia/reperfusion-induced injury. - Wikidata - wikimedia - TriplyDB

Targeting STAT1 by myricetin and delphinidin provides efficient protection of the heart from ischemia/reperfusion-induced injury.

Targeting STAT1 by myricetin and delphinidin provides efficient protection of the heart from ischemia/reperfusion-induced injury.

http://www.wikidata.org/entity/Q39900090

about

Citrus medica "Otroj": attenuates oxidative stress and cardiac dysrhythmia in isoproterenol-induced cardiomyopathy in rats Myricetin: A Dietary Molecule with Diverse Biological Activities Phytochemical Compounds and Protection from Cardiovascular Diseases: A State of the Art Anthocyanins in cardiovascular disease The exposome concept in a human nutrigenomics study: evaluating the impact of exposure to a complex mixture of phytochemicals using transcriptomics signatures Flavanols and anthocyanins in cardiovascular health: a review of current evidence.Cytoprotective and antioxidant effects of Echium amoenum anthocyanin-rich extract in human endothelial cells (HUVECs).STAT transcription in the ischemic heart.Anthocyanin Attenuates Doxorubicin-Induced Cardiomyotoxicity via Estrogen Receptor-α/β and Stabilizes HSF1 to Inhibit the IGF-IIR Apoptotic Pathway.STAT1 deficiency in the heart protects against myocardial infarction by enhancing autophagy.Dietary anthocyanin-rich plants: biochemical basis and recent progress in health benefits studies.Molecular mechanisms of the cardiovascular protective effects of polyphenols.Chemistry, Pharmacology and Health Benefits of Anthocyanins.Direct interaction of natural and synthetic catechins with signal transducer activator of transcription 1 affects both its phosphorylation and activity.Protective effects of Echium amoenum Fisch. and C.A. Mey. against cerebral ischemia in the rats.Cellular apoptosis and cardiac dysfunction in STZ-induced diabetic rats attenuated by anthocyanins via activation of IGFI-R/PI3K/Akt survival signaling.Myricetin attenuated LPS induced cardiac injury in vivo and in vitro.Persistence of STAT-1 inhibition and induction of cytokine resistance in pancreatic β cells treated with St John's wort and its component hyperforin.Luteolinidin Protects the Postischemic Heart through CD38 Inhibition with Preservation of NAD(P)(H).Anthocyanins as substrates for mitochondrial complex I - protective effect against heart ischemic injury.Synthetic CpG oligonucleotides induce a genetic profile ameliorating murine myocardial I/R injury.Food Bioactive HDAC Inhibitors in the Epigenetic Regulation of Heart Failure

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P2860

Targeting STAT1 by myricetin and delphinidin provides efficient protection of the heart from ischemia/reperfusion-induced injury.

http://www.wikidata.org/entity/Q39900090

description

2008 nî lūn-bûn

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2008年の論文

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2008年学术文章

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2008年学术文章

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2008年学术文章

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2008年學術文章

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Targeting STAT1 by myricetin a ...... ia/reperfusion-induced injury.

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J.FEBSLET.2008.12.037

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Targeting STAT1 by myricetin a ...... mia/reperfusion-induced injury

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Alessandra Carcereri de Prati

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Anastasis Stephanou

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Anna Ciampa

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Carol Chen-Scarabelli

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Cosimo Pizza

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David Latchman

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David Perahia

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Elisabetta Cavalieri

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Hisanori Suzuki

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Kazuo Shoji

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P2860

Structural bases of unphosphorylated STAT1 association and receptor binding

The JAK-binding protein JAB inhibits Janus tyrosine kinase activity through binding in the activation loop

Improved prediction of HIV-1 protease-inhibitor binding energies by molecular dynamics simulations

VEGA – An open platform to develop chemo-bio-informatics applications, using plug-in architecture and script programming

UCSF Chimera--a visualization system for exploratory research and analysis

Development and validation of a genetic algorithm for flexible docking

Stats: transcriptional control and biological impact

Tumor necrosis factor alpha and interleukin 1 stimulate the human immunodeficiency virus enhancer by activation of the nuclear factor kappa B

Comparative study of several algorithms for flexible ligand docking.

Redox control of cell death.

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10.1016/J.FEBSLET.2008.12.037

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Jean-Didier Maréchal

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