Upregulation of DR5 by proteasome inhibitors potently sensitizes glioma cells to TRAIL-induced apoptosis.
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Marine Drugs Regulating Apoptosis Induced by Tumor Necrosis Factor-Related Apoptosis-Inducing Ligand (TRAIL)Fluid shear stress regulates the invasive potential of glioma cells via modulation of migratory activity and matrix metalloproteinase expressionLarge-scale analysis of UPR-mediated apoptosis in human cells.Utilization of the cellular stress response to sensitize cancer cells to TRAIL-mediated apoptosis.Fenretinide-dependent upregulation of death receptors through ASK1 and p38α enhances death receptor ligand-induced cell death in Ewing's sarcoma family of tumours.Diagnostic and clinical relevance of the autophago-lysosomal network in human gliomasUrsolic acid, a pentacyclin triterpene, potentiates TRAIL-induced apoptosis through p53-independent up-regulation of death receptors: evidence for the role of reactive oxygen species and JNKTherapeutic effect of neural stem cells expressing TRAIL and bortezomib in mice with glioma xenograftsRegulating TRAIL receptor-induced cell death at the membrane : a deadly discussionNoninvasive monitoring of pharmacodynamics and kinetics of a death receptor 5 antibody and its enhanced apoptosis induction in sequential application with doxorubicinModulation of Mcl-1 sensitizes glioblastoma to TRAIL-induced apoptosisNaturally occurring, tumor-specific, therapeutic proteins.New frontiers in promoting tumour cell death: targeting apoptosis, necroptosis and autophagy.Implications of endoplasmic reticulum stress, the unfolded protein response and apoptosis for molecular cancer therapy. Part I: targeting p53, Mdm2, GADD153/CHOP, GRP78/BiP and heat shock proteins.Mechanisms of proteasome inhibitor-induced cytotoxicity in malignant glioma.Modulation of CCAAT/enhancer binding protein homologous protein (CHOP)-dependent DR5 expression by nelfinavir sensitizes glioblastoma multiforme cells to tumor necrosis factor-related apoptosis-inducing ligand (TRAIL).Mitomycin C potentiates TRAIL-induced apoptosis through p53-independent upregulation of death receptors: evidence for the role of c-Jun N-terminal kinase activation.Novel insights into the synergistic interaction of Bortezomib and TRAIL: tBid provides the link.Death receptor 4 is preferentially recruited to lipid rafts in chronic lymphocytic leukemia cells contributing to tumor necrosis related apoptosis inducing ligand-induced synergistic apoptotic responses.Off-target lapatinib activity sensitizes colon cancer cells through TRAIL death receptor up-regulation.AT 101 induces early mitochondrial dysfunction and HMOX1 (heme oxygenase 1) to trigger mitophagic cell death in glioma cells.
P2860
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P2860
Upregulation of DR5 by proteasome inhibitors potently sensitizes glioma cells to TRAIL-induced apoptosis.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年学术文章
@wuu
2008年学术文章
@zh-cn
2008年学术文章
@zh-hans
2008年学术文章
@zh-my
2008年学术文章
@zh-sg
2008年學術文章
@yue
2008年學術文章
@zh
2008年學術文章
@zh-hant
name
Upregulation of DR5 by proteas ...... ls to TRAIL-induced apoptosis.
@en
type
label
Upregulation of DR5 by proteas ...... ls to TRAIL-induced apoptosis.
@en
prefLabel
Upregulation of DR5 by proteas ...... ls to TRAIL-induced apoptosis.
@en
P2093
P2860
P1433
P1476
Upregulation of DR5 by proteas ...... lls to TRAIL-induced apoptosis
@en
P2093
Donat Kögel
Holger Hetschko
Valerie Voss
Volker Seifert
P2860
P304
P356
10.1111/J.1742-4658.2008.06351.X
P407
P50
P577
2008-03-13T00:00:00Z