The sensitivity of Cockayne's syndrome cells to DNA-damaging agents is not due to defective transcription-coupled repair of active genes.
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The ATPase domain but not the acidic region of Cockayne syndrome group B gene product is essential for DNA repairCockayne syndrome: defective repair of transcription?Molecular analysis of mutations in the CSB (ERCC6) gene in patients with Cockayne syndromeRecruitment of the putative transcription-repair coupling factor CSB/ERCC6 to RNA polymerase II elongation complexesCockayne syndrome group B protein stimulates repair of formamidopyrimidines by NEIL1 DNA glycosylaseHuman XPC-hHR23B interacts with XPA-RPA in the recognition of triplex-directed psoralen DNA interstrand crosslinks.Cockayne syndrome B protein stimulates apurinic endonuclease 1 activity and protects against agents that introduce base excision repair intermediatesCockayne syndrome: varied requirement of transcription-coupled nucleotide excision repair for the removal of three structurally different adducts from transcribed DNADecreased transcription-coupled nucleotide excision repair capacity is associated with increased p53- and MLH1-independent apoptosis in response to cisplatin.Molecular characterization of an acidic region deletion mutant of Cockayne syndrome group B protein.The Cockayne syndrome B protein, involved in transcription-coupled DNA repair, resides in an RNA polymerase II-containing complexRestoration of nucleotide excision repair in a helicase-deficient XPD mutant from intragenic suppression by a trichothiodystrophy mutationReduced RNA polymerase II transcription in extracts of cockayne syndrome and xeroderma pigmentosum/Cockayne syndrome cells.Histone methyltransferase DOT1L drives recovery of gene expression after a genotoxic attack.Nucleotide excision repair in rat male germ cells: low level of repair in intact cells contrasts with high dual incision activity in vitroIdentification of Novel Proteins Co-Purifying with Cockayne Syndrome Group B (CSB) Reveals Potential Roles for CSB in RNA Metabolism and Chromatin Dynamics.Reduced RNA polymerase II transcription in intact and permeabilized Cockayne syndrome group B cells.Transitions in the coupling of transcription and nucleotide excision repair within RNA polymerase II-transcribed genes of Saccharomyces cerevisiae.UV-induced inhibition of transcription involves repression of transcription initiation and phosphorylation of RNA polymerase II.The many faces of Cockayne syndrome.Coupling of human DNA excision repair and the DNA damage checkpoint in a defined in vitro system.Yeast RNA polymerase II transcription in vitro is inhibited in the presence of nucleotide excision repair: complementation of inhibition by Holo-TFIIH and requirement for RAD26.The sensitivity of human fibroblasts to N-acetoxy-2-acetylaminofluorene is determined by the extent of transcription-coupled repair, and/or their capability to counteract RNA synthesis inhibitionUV damage causes uncontrolled DNA breakage in cells from patients with combined features of XP-D and Cockayne syndrome.Replication protein A safeguards genome integrity by controlling NER incision events.Local UV-induced DNA damage in cell nuclei results in local transcription inhibition.
P2860
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P2860
The sensitivity of Cockayne's syndrome cells to DNA-damaging agents is not due to defective transcription-coupled repair of active genes.
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年論文
@yue
1996年論文
@zh-hant
1996年論文
@zh-hk
1996年論文
@zh-mo
1996年論文
@zh-tw
1996年论文
@wuu
1996年论文
@zh
1996年论文
@zh-cn
name
The sensitivity of Cockayne's ...... oupled repair of active genes.
@en
type
label
The sensitivity of Cockayne's ...... oupled repair of active genes.
@en
prefLabel
The sensitivity of Cockayne's ...... oupled repair of active genes.
@en
P2093
P2860
P1476
The sensitivity of Cockayne's ...... oupled repair of active genes.
@en
P2093
Mullenders LH
Versteeg A
van Oosterwijk MF
van Zeeland AA
P2860
P304
P407
P577
1996-08-01T00:00:00Z