Cell type-specific transcriptional regulation of the human adenosine deaminase gene.
about
The genetic defect in Cockayne syndrome is associated with a defect in repair of UV-induced DNA damage in transcriptionally active DNA.Characterization of the deoxycytidine kinase promoter in human lymphoblast cell linesDeficient repair of the transcribed strand of active genes in Cockayne's syndrome cells.Sp1 is essential for both enhancer-mediated and basal activation of the TATA-less human adenosine deaminase promoter.Hereditary overexpression of adenosine deaminase in erythrocytes: evidence for a cis-acting mutation.The residual repair capacity of xeroderma pigmentosum complementation group C fibroblasts is highly specific for transcriptionally active DNAHIV-1 Tat protein promotes formation of more-processive elongation complexes.In vivo transcriptional pausing and cap formation on three Drosophila heat shock genes.Functional analysis of a stable transcription arrest site in the first intron of the murine adenosine deaminase gene.Sequence requirements for premature transcription arrest within the first intron of the mouse c-fos gene.Identification and characterization of transcriptional arrest sites in exon 1 of the human adenosine deaminase gene.Xeroderma pigmentosum complementation group C cells remove pyrimidine dimers selectively from the transcribed strand of active genes.In vitro analysis of a transcription termination site for RNA polymerase II.Sequence requirements for transcriptional arrest in exon 1 of the human adenosine deaminase geneSequence requirements for transcriptional arrest in exon 1 of the murine adenosine deaminase geneStability of Drosophila RNA polymerase II elongation complexes in vitro.Control of formation of two distinct classes of RNA polymerase II elongation complexes.The sensitivity of human fibroblasts to N-acetoxy-2-acetylaminofluorene is determined by the extent of transcription-coupled repair, and/or their capability to counteract RNA synthesis inhibitionThe sensitivity of Cockayne's syndrome cells to DNA-damaging agents is not due to defective transcription-coupled repair of active genes.Regulation of eukaryotic gene expression by transcriptional attenuation.Transcription-coupled repair removes both cyclobutane pyrimidine dimers and 6-4 photoproducts with equal efficiency and in a sequential way from transcribed DNA in xeroderma pigmentosum group C fibroblasts.Control of transcription arrest in intron 1 of the murine adenosine deaminase gene.Functional analysis of the human adenosine deaminase gene thymic regulatory region and its ability to generate position-independent transgene expression.A duodenum-specific enhancer regulates expression along three axes in the small intestine.
P2860
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P2860
Cell type-specific transcriptional regulation of the human adenosine deaminase gene.
description
1989 nî lūn-bûn
@nan
1989年の論文
@ja
1989年論文
@yue
1989年論文
@zh-hant
1989年論文
@zh-hk
1989年論文
@zh-mo
1989年論文
@zh-tw
1989年论文
@wuu
1989年论文
@zh
1989年论文
@zh-cn
name
Cell type-specific transcriptional regulation of the human adenosine deaminase gene.
@ast
Cell type-specific transcriptional regulation of the human adenosine deaminase gene.
@en
type
label
Cell type-specific transcriptional regulation of the human adenosine deaminase gene.
@ast
Cell type-specific transcriptional regulation of the human adenosine deaminase gene.
@en
prefLabel
Cell type-specific transcriptional regulation of the human adenosine deaminase gene.
@ast
Cell type-specific transcriptional regulation of the human adenosine deaminase gene.
@en
P2093
P2860
P356
P1476
Cell type-specific transcriptional regulation of the human adenosine deaminase gene.
@en
P2093
D A Wiginton
D L Lattier
J C States
J J Hutton
P2860
P304
P356
10.1093/NAR/17.3.1061
P407
P577
1989-02-01T00:00:00Z