Evidence of Abeta- and transgene-dependent defects in ERK-CREB signaling in Alzheimer's models. - Wikidata - wikimedia - TriplyDB

Evidence of Abeta- and transgene-dependent defects in ERK-CREB signaling in Alzheimer's models.

Evidence of Abeta- and transgene-dependent defects in ERK-CREB signaling in Alzheimer's models.

http://www.wikidata.org/entity/Q40087274

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The endocannabinoid system in normal and pathological brain ageing Alzheimer's Disease and Hippocampal Adult Neurogenesis; Exploring Shared Mechanisms Evaluation of traditional medicines for neurodegenerative diseases using Drosophila models Diminished CRE-Induced Plasticity is Linked to Memory Deficits in Familial Alzheimer's Disease Mice The role of mTOR signaling in Alzheimer disease GSK3 inhibitors show benefits in an Alzheimer's disease (AD) model of neurodegeneration but adverse effects in control animals Molecular mechanisms of neurodegeneration in Alzheimer's disease.PERK mediates eIF2α phosphorylation responsible for BACE1 elevation, CREB dysfunction and neurodegeneration in a mouse model of Alzheimer's disease.Calpain activation promotes BACE1 expression, amyloid precursor protein processing, and amyloid plaque formation in a transgenic mouse model of Alzheimer disease Intranasal "painless" human Nerve Growth Factor [corrected] slows amyloid neurodegeneration and prevents memory deficits in App X PS1 mice CBP gene transfer increases BDNF levels and ameliorates learning and memory deficits in a mouse model of Alzheimer's disease.Amyloid precursor protein (APP) processing genes and cerebrospinal fluid APP cleavage product levels in Alzheimer's disease Molecular mechanisms of environmental enrichment: impairments in Akt/GSK3β, neurotrophin-3 and CREB signaling.Constitutive cAMP response element binding protein (CREB) activation by Alzheimer's disease presenilin-driven inositol trisphosphate receptor (InsP3R) Ca2+ signaling.Characterization of Aldh2 (-/-) mice as an age-related model of cognitive impairment and Alzheimer's disease Tong Luo Jiu Nao ameliorates Aβ1-40-induced cognitive impairment on adaptive behavior learning by modulating ERK/CaMKII/CREB signaling in the hippocampus.CREB-activity and nmnat2 transcription are down-regulated prior to neurodegeneration, while NMNAT2 over-expression is neuroprotective, in a mouse model of human tauopathy Calpain-mediated cleavage of DARPP-32 in Alzheimer's disease.Hippocampal Injections of Oligomeric Amyloid β-peptide (1-42) Induce Selective Working Memory Deficits and Long-lasting Alterations of ERK Signaling Pathway.Steamed and Fermented Ethanolic Extract from Codonopsis lanceolata Attenuates Amyloid-β-Induced Memory Impairment in Mice.Reduction of SorLA/LR11, a sorting protein limiting beta-amyloid production, in Alzheimer disease cerebrospinal fluid.Failures to reconsolidate memory in a mouse model of Alzheimer's disease.CREB: a multifaceted regulator of neuronal plasticity and protection.The role of CREB signaling in Alzheimer's disease and other cognitive disorders.Modulating nitric oxide signaling in the CNS for Alzheimer's disease therapy.Chemopreventative effects of tetrahydrocurcumin on human diseases.The role of cytosolic phospholipase A2 α in amyloid precursor protein induction by amyloid beta1-42 : implication for neurodegeneration.Chronic diabetic states worsen Alzheimer neuropathology and cognitive deficits accompanying disruption of calcium signaling in leptin-deficient APP/PS1 mice.Age-related toxicity of amyloid-beta associated with increased pERK and pCREB in primary hippocampal neurons: reversal by blueberry extract.Coriandrum sativum Suppresses Aβ42-Induced ROS Increases, Glial Cell Proliferation, and ERK Activation.Changes in insulin-signaling transduction pathway underlie learning/memory deficits in an Alzheimer's disease rat model.Chronic Stress Decreases Basal Levels of Memory-Related Signaling Molecules in Area CA1 of At-Risk (Subclinical) Model of Alzheimer's Disease.Liraglutide Improves Water Maze Learning and Memory Performance While Reduces Hyperphosphorylation of Tau and Neurofilaments in APP/PS1/Tau Triple Transgenic Mice.Chronic psychosocial stress accelerates impairment of long-term memory and late-phase long-term potentiation in an at-risk model of Alzheimer's disease.

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Evidence of Abeta- and transgene-dependent defects in ERK-CREB signaling in Alzheimer's models.

http://www.wikidata.org/entity/Q40087274

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Evidence of Abeta- and transge ...... gnaling in Alzheimer's models.

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Evidence of Abeta- and transge ...... gnaling in Alzheimer's models.

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Evidence of Abeta- and transge ...... gnaling in Alzheimer's models.

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J.1471-4159.2007.04869.X

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Journal of Neurochemistry

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Evidence of Abeta- and transge ...... gnaling in Alzheimer's models.

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P2093

Bruce Teter

http://www.w3.org/2001/XMLSchema#string

Giselle P Lim

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Greg M Cole

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Marni E Harris-White

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Mychica Simmons

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Oliver J Ubeda

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Qiu-Lan Ma

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Sally A Frautschy

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Walter Beech

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P2860

Genetic enhancement of learning and memory in mice

Soluble amyloid beta peptide concentration as a predictor of synaptic change in Alzheimer's disease

The NMDA receptor is coupled to the ERK pathway by a direct interaction between NR2B and RasGRF1.

Regulation of NMDA receptor trafficking by amyloid-beta

alpha1 Integrin activation: a link between beta-amyloid deposition and neuronal death in aging hippocampal neurons

Mechanism for the learning deficits in a mouse model of neurofibromatosis type 1

Beta-amyloid activates the mitogen-activated protein kinase cascade via hippocampal alpha7 nicotinic acetylcholine receptors: In vitro and in vivo mechanisms related to Alzheimer's disease

A specific amyloid-beta protein assembly in the brain impairs memory

MAPK recruitment by beta-amyloid in organotypic hippocampal slice cultures depends on physical state and exposure time

Common structure of soluble amyloid oligomers implies common mechanism of pathogenesis

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1594-1607

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6079172

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17760871

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Alzheimer's disease

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2527620

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