Progressive loss of a glial potassium channel (KCNJ10) in the spinal cord of the SOD1 (G93A) transgenic mouse model of amyotrophic lateral sclerosis.
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New Insights on Astrocyte Ion Channels: Critical for Homeostasis and Neuron-Glia SignalingElusive roles for reactive astrocytes in neurodegenerative diseasesNovel molecular biomarkers at the blood-brain barrier in ALSAstroglia in Thick Tissue with Super Resolution and Cellular ReconstructionAquaporins in the Spinal CordFree radical stress-mediated loss of Kcnj10 protein expression in stria vascularis contributes to deafness in Pendred syndrome mouse model.Kir4.1 channels regulate swelling of astroglial processes in experimental spinal cord edema.Sporadic ALS has compartment-specific aberrant exon splicing and altered cell-matrix adhesion biology.Spinal cord injury causes a wide-spread, persistent loss of Kir4.1 and glutamate transporter 1: benefit of 17 beta-oestradiol treatmentInfluence of methylene blue on microglia-induced inflammation and motor neuron degeneration in the SOD1(G93A) model for ALS.Changes in the astrocytic aquaporin-4 and inwardly rectifying potassium channel expression in the brain of the amyotrophic lateral sclerosis SOD1(G93A) rat model.Elevated GFAP induces astrocyte dysfunction in caudal brain regions: A potential mechanism for hindbrain involved symptoms in type II Alexander disease.Differential distribution of Kir4.1 in spinal cord astrocytes suggests regional differences in K+ homeostasis.Spatial and temporal correlation in progressive degeneration of neurons and astrocytes in contusion-induced spinal cord injuryRegulation of Kir4.1 expression in astrocytes and astrocytic tumors: a role for interleukin-1 βDysfunctional Calcium and Glutamate Signaling in Striatal Astrocytes from Huntington's Disease Model MiceFunctional implications for Kir4.1 channels in glial biology: from K+ buffering to cell differentiationDNA methylation functions as a critical regulator of Kir4.1 expression during CNS development.Aquaporins and neurodegenerative diseases.Impairments in Motor Neurons, Interneurons and Astrocytes Contribute to Hyperexcitability in ALS: Underlying Mechanisms and Paths to Therapy.The role of glial-specific Kir4.1 in normal and pathological states of the CNS.Hereditary primary lateral sclerosis with cone dysfunction.Links between electrophysiological and molecular pathology of amyotrophic lateral sclerosis.Inhibition of Inwardly Rectifying Potassium (Kir) 4.1 Channels Facilitates Brain-Derived Neurotrophic Factor (BDNF) Expression in Astrocytes.Diversity of astroglial responses across human neurodegenerative disorders and brain aging.Role and Therapeutic Potential of Astrocytes in Amyotrophic Lateral Sclerosis.Astroglial transcriptome dysregulation in early disease of an ALS mutant SOD1 mouse model.Aquaporin-4 as a potential marker of BBB disruption in ALS models.Kir4.1-Dependent Astrocyte-Fast Motor Neuron Interactions Are Required for Peak Strength.Oligodendrocytes control potassium accumulation in white matter and seizure susceptibility.Differential loss of KIR4.1 immunoreactivity in multiple sclerosis lesions
P2860
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P2860
Progressive loss of a glial potassium channel (KCNJ10) in the spinal cord of the SOD1 (G93A) transgenic mouse model of amyotrophic lateral sclerosis.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
2006年论文
@zh
2006年论文
@zh-cn
name
Progressive loss of a glial po ...... amyotrophic lateral sclerosis.
@en
type
label
Progressive loss of a glial po ...... amyotrophic lateral sclerosis.
@en
prefLabel
Progressive loss of a glial po ...... amyotrophic lateral sclerosis.
@en
P2093
P2860
P1476
Progressive loss of a glial po ...... amyotrophic lateral sclerosis.
@en
P2093
Bettina Holtmann
Clemens Neusch
Iris Maletzki
Mathias Bähr
Melanie Kaiser
Swen Hülsmann
Walter Schulz-Schaeffer
P2860
P304
P356
10.1111/J.1471-4159.2006.04131.X
P407
P577
2006-08-21T00:00:00Z