Induction of cancer cell apoptosis by flavonoids is associated with their ability to inhibit fatty acid synthase activity.
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Molecular targets of nutraceuticals derived from dietary spices: potential role in suppression of inflammation and tumorigenesisLuteolin, a flavonoid with potential for cancer prevention and therapyChemistry and biological activities of flavonoids: an overviewComments on the mechanisms of action of radiation protective agents: basis components and their polyvalenceApigenin: a promising molecule for cancer preventionThe dietary bioflavonoid, quercetin, selectively induces apoptosis of prostate cancer cells by down-regulating the expression of heat shock protein 90.Diet components can suppress inflammation and reduce cancer risk.Tools for the identification of bioactives impacting the metabolic syndrome: screening of a botanical extract library using subcutaneous and visceral human adipose-derived stem cell-based assays.Biochemistry, molecular biology, and pharmacology of fatty acid synthase, an emerging therapeutic target and diagnosis/prognosis marker.Ginkgo biloba extract kaempferol inhibits cell proliferation and induces apoptosis in pancreatic cancer cellsMolecular targets of apigenin in colorectal cancer cells: involvement of p21, NAG-1 and p53.Bioactive food components and cancer-specific metabonomic profiles.The importance of GLUT3 for de novo lipogenesis in hypoxia-induced lipid loading of human macrophages.Modulation of PI3K-LXRα-dependent lipogenesis mediated by oxidative/nitrosative stress contributes to inhibition of HCV replication by quercetin.Autophagy inhibitor chloroquine enhanced the cell death inducing effect of the flavonoid luteolin in metastatic squamous cell carcinoma cells.Antibacterial, antioxidant and tyrosinase-inhibition activities of pomegranate fruit peel methanolic extract.Inhibition of fatty acid amide hydrolase by kaempferol and related naturally occurring flavonoids.Laser-induced hydrogen radical removal in UV MALDI-MS allows for the differentiation of flavonoid monoglycoside isomers.Butin (7,3',4'-trihydroxydihydroflavone) reduces oxidative stress-induced cell death via inhibition of the mitochondria-dependent apoptotic pathwayGenotoxic and oxidative damage potentials in human lymphocytes after exposure to terpinolene in vitro.Apigenin suppresses cancer cell growth through ERbeta.The polyphenols (-)-epigallocatechin-3-gallate and luteolin synergistically inhibit TGF-β-induced myofibroblast phenotypes through RhoA and ERK inhibitionFatty acid synthase as a potential therapeutic target in cancer.The flavonoid nobiletin inhibits tumor growth and angiogenesis of ovarian cancers via the Akt pathway.Luteolin Inhibits Angiotensin II-Stimulated VSMC Proliferation and Migration through Downregulation of Akt Phosphorylation.Kaempferol Inhibits Pancreatic Cancer Cell Growth and Migration through the Blockade of EGFR-Related Pathway In Vitro.Taxifolin suppresses UV-induced skin carcinogenesis by targeting EGFR and PI3K.Dose-dependent benefits of quercetin on tumorigenesis in the C3(1)/SV40Tag transgenic mouse model of breast cancer.Cytotoxicity and genotoxicity of zingiberene on different neuron cell lines in vitro.Taxifolin synergizes Andrographolide-induced cell death by attenuation of autophagy and augmentation of caspase dependent and independent cell death in HeLa cells.A Genetic Screen Reveals Novel Targets to Render Pseudomonas aeruginosa Sensitive to Lysozyme and Cell Wall-Targeting AntibioticsThe flavonoid quercetin inhibits pancreatic cancer growth in vitro and in vivo.A review of the dietary flavonoid, kaempferol on human health and cancer chemopreventionDietary flavonoids and colorectal adenoma recurrence in the Polyp Prevention Trial.Natural compounds as regulators of the cancer cell metabolismApigenin inhibits antiestrogen-resistant breast cancer cell growth through estrogen receptor-alpha-dependent and estrogen receptor-alpha-independent mechanismsFatty acid synthase inhibitors: new directions for oncology.EWS Knockdown and Taxifolin Treatment Induced Differentiation and Removed DNA Methylation from p53 Promoter to Promote Expression of Puma and Noxa for Apoptosis in Ewing's SarcomaMetabolic intervention on lipid synthesis converging pathways abrogates prostate cancer growth.Inhibition of fatty acid synthase by luteolin post-transcriptionally down-regulates c-Met expression independent of proteosomal/lysosomal degradation.
P2860
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P2860
Induction of cancer cell apoptosis by flavonoids is associated with their ability to inhibit fatty acid synthase activity.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年学术文章
@wuu
2004年学术文章
@zh-cn
2004年学术文章
@zh-hans
2004年学术文章
@zh-my
2004年学术文章
@zh-sg
2004年學術文章
@yue
2004年學術文章
@zh
2004年學術文章
@zh-hant
name
Induction of cancer cell apopt ...... fatty acid synthase activity.
@en
type
label
Induction of cancer cell apopt ...... fatty acid synthase activity.
@en
prefLabel
Induction of cancer cell apopt ...... fatty acid synthase activity.
@en
P2093
P2860
P356
P1476
Induction of cancer cell apopt ...... t fatty acid synthase activity
@en
P2093
Guido Verhoeven
Koen Brusselmans
Ruth Vrolix
P2860
P304
P356
10.1074/JBC.M408177200
P407
P577
2004-11-08T00:00:00Z