The stability of tristetraprolin mRNA is regulated by mitogen-activated protein kinase p38 and by tristetraprolin itself.
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The roles of TTP and BRF proteins in regulated mRNA decayZFP36L1 is regulated by growth factors and cytokines in keratinocytes and influences their VEGF productionMAPKAP kinase 2 blocks tristetraprolin-directed mRNA decay by inhibiting CAF1 deadenylase recruitmentProtor-2 interacts with tristetraprolin to regulate mRNA stability during stressNuclear localization of protein kinase C-alpha induces thyroid hormone receptor-alpha1 expression in the cardiomyocyteRegulation of tristetraprolin during differentiation of 3T3-L1 preadipocytesBrf1 posttranscriptionally regulates pluripotency and differentiation responses downstream of Erk MAP kinaseFunctional analysis of KSRP interaction with the AU-rich element of interleukin-8 and identification of inflammatory mRNA targets.Phosphorylation site analysis of the anti-inflammatory and mRNA-destabilizing protein tristetraprolin.Pterostilbene 4'-β-Glucoside Protects against DSS-Induced Colitis via Induction of Tristetraprolin.Enteropathogenic Escherichia coli inhibits type I interferon- and RNase L-mediated host defense to disrupt intestinal epithelial cell barrier functionmiR-375 inhibits differentiation of neurites by lowering HuD levels.Coordinated expression of tristetraprolin post-transcriptionally attenuates mitogenic induction of the oncogenic Ser/Thr kinase Pim-1.Expression, purification, and biochemical characterization of the antiinflammatory tristetraprolin: a zinc-dependent mRNA binding protein affected by posttranslational modifications.The p38/MK2-driven exchange between tristetraprolin and HuR regulates AU-rich element-dependent translation.Phosphorylation of tristetraprolin by MK2 impairs AU-rich element mRNA decay by preventing deadenylase recruitment.Mitogen-activated protein kinase-activated protein kinase 2 regulates tumor necrosis factor mRNA stability and translation mainly by altering tristetraprolin expression, stability, and binding to adenine/uridine-rich elementRNase L attenuates mitogen-stimulated gene expression via transcriptional and post-transcriptional mechanisms to limit the proliferative responseLong-acting β2-agonists increase fluticasone propionate-induced mitogen-activated protein kinase phosphatase 1 (MKP-1) in airway smooth muscle cells.An RNA binding protein promotes axonal integrity in peripheral neurons by destabilizing REST.Deciphering the role of RNA-binding proteins in the post-transcriptional control of gene expressionmiRNA regulation of cytokine genes.Control of mRNA stability by SAPKs.Tristetraprolin mediates anti-inflammatory effects of nicotine in lipopolysaccharide-stimulated macrophages.Glucocorticoids regulate tristetraprolin synthesis and posttranscriptionally regulate tumor necrosis factor alpha inflammatory signaling.Novel mRNA targets for tristetraprolin (TTP) identified by global analysis of stabilized transcripts in TTP-deficient fibroblasts.CREB targets define the gene expression signature of malignancies having reduced levels of the tumor suppressor tristetraprolinPost-transcriptional regulation of satellite cell quiescence by TTP-mediated mRNA decay.Protein phosphatase 2A (PP2A) is increased in old murine B cells and mediates p38 MAPK/tristetraprolin dephosphorylation and E47 mRNA instability.Expression of leukemia inhibitory factor in Müller glia cells is regulated by a redox-dependent mRNA stability mechanismDominant Suppression of Inflammation via Targeted Mutation of the mRNA Destabilizing Protein Tristetraprolin.Aging impairs murine B cell differentiation and function in primary and secondary lymphoid tissuesFunctional regulation of Zfp36l1 and Zfp36l2 in response to lipopolysaccharide in mouse RAW264.7 macrophagesAnalysis of turnover and translation regulatory RNA-binding protein expression through binding to cognate mRNAs.Regulation of p21/CIP1/WAF-1 mediated cell-cycle arrest by RNase L and tristetraprolin, and involvement of AU-rich elements.The role of tristetraprolin in cancer and inflammation.LARP4 Is Regulated by Tumor Necrosis Factor Alpha in a Tristetraprolin-Dependent Manner.Enhanced stability of tristetraprolin mRNA protects mice against immune-mediated inflammatory pathologies.Interaction with Pyruvate Kinase M2 Destabilizes Tristetraprolin by Proteasome Degradation and Regulates Cell Proliferation in Breast Cancer.LPS-induced production of TNF-α and IL-6 in mast cells is dependent on p38 but independent of TTP
P2860
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P2860
The stability of tristetraprolin mRNA is regulated by mitogen-activated protein kinase p38 and by tristetraprolin itself.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
@zh
2004年论文
@zh-cn
name
The stability of tristetraprol ...... and by tristetraprolin itself.
@en
type
label
The stability of tristetraprol ...... and by tristetraprolin itself.
@en
prefLabel
The stability of tristetraprol ...... and by tristetraprolin itself.
@en
P2093
P2860
P356
P1476
The stability of tristetraprol ...... and by tristetraprolin itself.
@en
P2093
Carmen R Tchen
Jeremy Saklatvala
Matthew Brook
P2860
P304
32393-32400
P356
10.1074/JBC.M402059200
P407
P577
2004-06-07T00:00:00Z