Inhibition of NFkappaB increases the efficacy of cisplatin in in vitro and in vivo ovarian cancer models.
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TCEAL7, a putative tumor suppressor gene, negatively regulates NF-kappaB pathwayTranscriptional regulation of chemokine expression in ovarian cancerChemotherapy-enhanced inflammation may lead to the failure of therapy and metastasisNitrosylcobalamin potentiates the anti-neoplastic effects of chemotherapeutic agents via suppression of survival signalingOncoproteomic analysis reveals co-upregulation of RELA and STAT5 in carboplatin resistant ovarian carcinomaCystathionine beta-synthase (CBS) contributes to advanced ovarian cancer progression and drug resistanceNuclear IKK activity leads to dysregulated notch-dependent gene expression in colorectal cancer.Carboplatin-induced gene expression changes in vitro are prognostic of survival in epithelial ovarian cancerWNT5A-ROR2 is induced by inflammatory mediators and is involved in the migration of human ovarian cancer cell line SKOV-3Pathogenesis of ovarian cancer: clues from selected overexpressed genes.Reactive oxygen species mediate lysophosphatidic acid induced signaling in ovarian cancer cells.Chronic cisplatin treatment promotes enhanced damage repair and tumor progression in a mouse model of lung cancerVascular endothelial growth factor is a promising therapeutic target for the treatment of clear cell carcinoma of the ovary.Elevated TAK1 augments tumor growth and metastatic capacities of ovarian cancer cells through activation of NF-κB signaling.miR-141 regulates KEAP1 and modulates cisplatin sensitivity in ovarian cancer cells.The NF-kappaB activation pathways, emerging molecular targets for cancer prevention and therapyCalcitonin gene-related peptide inhibits chemokine production by human dermal microvascular endothelial cells.Moringa Oleifera aqueous leaf extract down-regulates nuclear factor-kappaB and increases cytotoxic effect of chemotherapy in pancreatic cancer cells.Genomics of the NF-κB signaling pathway: hypothesized role in ovarian cancer.The biphasic role of NF-kappaB in progression and chemoresistance of ovarian cancer.Glycogen synthase kinase 3β inhibitors induce apoptosis in ovarian cancer cells and inhibit in-vivo tumor growthPreclinical Efficacy for AKT Targeting in Clear Cell Carcinoma of the OvaryChemosensitization in non-small cell lung cancer cells by IKK inhibitor occurs via NF-kappaB and mitochondrial cytochrome c cascade.NF-kappa B--a target in the inflammation of bone destruction.Sensitization of tumor cells to cancer therapy by molecularly targeted inhibition of the inhibitor of nuclear factor κB kinase.Expression of NF-κB and PTEN in primary epithelial ovarian carcinoma and the correlation with chemoresistance.IKK inhibition increases bortezomib effectiveness in ovarian cancer.Fucoxanthin enhances cisplatin-induced cytotoxicity via NFκB-mediated pathway and downregulates DNA repair gene expression in human hepatoma HepG2 cells.Inflammation, cancer and chemoresistance: taking advantage of the toll-like receptor signaling pathway.NF-kappa B as a target for cancer therapy.Tissue transglutaminase protects epithelial ovarian cancer cells from cisplatin-induced apoptosis by promoting cell survival signaling.Transglutaminase 2 expression predicts progression free survival in non-small cell lung cancer patients treated with epidermal growth factor receptor tyrosine kinase inhibitorResistance to chemotherapy in cancer: a complex and integrated cellular response.Resistance to cisplatin does not affect sensitivity of human ovarian cancer cell lines to mifepristone cytotoxicity.NAC-1, a potential stem cell pluripotency factor, contributes to paclitaxel resistance in ovarian cancer through inactivating Gadd45 pathway.Mifepristone abrogates repopulation of ovarian cancer cells in between courses of cisplatin treatmentTriptolide avoids cisplatin resistance and induces apoptosis via the reactive oxygen species/nuclear factor-κB pathway in SKOV3(PT) platinum-resistant human ovarian cancer cells.mTOR is a promising therapeutic target both in cisplatin-sensitive and cisplatin-resistant clear cell carcinoma of the ovary.Potential role of mTORC2 as a therapeutic target in clear cell carcinoma of the ovary.rpL3 promotes the apoptosis of p53 mutated lung cancer cells by down-regulating CBS and NFκB upon 5-FU treatment
P2860
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P2860
Inhibition of NFkappaB increases the efficacy of cisplatin in in vitro and in vivo ovarian cancer models.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
@zh
2004年论文
@zh-cn
name
Inhibition of NFkappaB increas ...... in vivo ovarian cancer models.
@en
type
label
Inhibition of NFkappaB increas ...... in vivo ovarian cancer models.
@en
prefLabel
Inhibition of NFkappaB increas ...... in vivo ovarian cancer models.
@en
P2093
P2860
P356
P1476
Inhibition of NFkappaB increas ...... in vivo ovarian cancer models.
@en
P2093
Akiko Kimura
Hirohisa Kurachi
Jun Kawagoe
Kazuhiro Takahashi
Keiichi Tasaka
Maki Saito
Masahide Ohmichi
Masahiro Sakata
Namiko Yada-Hashimoto
Seiji Mabuchi
P2860
P304
23477-23485
P356
10.1074/JBC.M313709200
P407
P577
2004-03-16T00:00:00Z