Glucagon-like peptide-1-responsive catecholamine neurons in the area postrema link peripheral glucagon-like peptide-1 with central autonomic control sites.
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Central and peripheral administration of secretin inhibits food intake in mice through the activation of the melanocortin systemInsights into the role of neuronal glucokinaseThe role of gut endocrine cells in control of metabolism and appetiteGLP-1, the gut-brain, and brain-periphery axesGLP-1 receptor stimulation depresses heart rate variability and inhibits neurotransmission to cardiac vagal neurons.Neuroendocrine circuits governing energy balance and stress regulation: functional overlap and therapeutic implicationsThe anorectic actions of the TGFβ cytokine MIC-1/GDF15 require an intact brainstem area postrema and nucleus of the solitary tractExendin-4 increases blood glucose levels acutely in rats by activation of the sympathetic nervous systemCardiovascular and hemodynamic effects of glucagon-like peptide-1.Complete rescue of obesity, diabetes, and infertility in db/db mice by neuron-specific LEPR-B transgenes.Transferrin fusion technology: a novel approach to prolonging biological half-life of insulinotropic peptidesPotential role of TCF7L2 gene variants on cardiac sympathetic/parasympathetic activityVagal innervation of intestine contributes to weight loss After Roux-en-Y gastric bypass surgery in rats.Distribution and characterisation of Glucagon-like peptide-1 receptor expressing cells in the mouse brainCharacterization of Kiss1 neurons using transgenic mouse modelsThe vagus nerve, food intake and obesity.Gene-environment interactions controlling energy and glucose homeostasis and the developmental origins of obesityThe role of incretins in glucose homeostasis and diabetes treatment.Preproglucagon neurons project widely to autonomic control areas in the mouse brain.Preproglucagon (PPG) neurons innervate neurochemically identified autonomic neurons in the mouse brainstemObesity surgery and gut-brain communication.CCK stimulation of GLP-1 neurons involves α1-adrenoceptor-mediated increase in glutamatergic synaptic inputsJejunal linoleic acid infusions require GLP-1 receptor signaling to inhibit food intake: implications for the effectiveness of Roux-en-Y gastric bypass.Short-term exenatide treatment leads to significant weight loss in a subset of obese women without diabetes.Pancreatic GLP-1 receptor activation is sufficient for incretin control of glucose metabolism in mice.Vagal innervation of the hepatic portal vein and liver is not necessary for Roux-en-Y gastric bypass surgery-induced hypophagia, weight loss, and hypermetabolism.Circulating GLP-1 and CCK-8 reduce food intake by capsaicin-insensitive, nonvagal mechanisms.GLP-1R and amylin agonism in metabolic disease: complementary mechanisms and future opportunitiesNeuroprotective and neurotrophic actions of glucagon-like peptide-1: an emerging opportunity to treat neurodegenerative and cerebrovascular disordersReview of the neuroanatomic landscape implicated in glucose sensing and regulation of nutrient signaling: immunophenotypic localization of diabetes gene Tcf7l2 in the developing murine brain.PPG neurons of the lower brain stem and their role in brain GLP-1 receptor activation.Suppression of food intake by glucagon-like peptide-1 receptor agonists: relative potencies and role of dipeptidyl peptidase-4Autonomic nervous system-dependent and -independent cardiovascular effects of exendin-4 infusion in conscious rats.Vagal and hormonal gut-brain communication: from satiation to satisfactionProtein engineering strategies for sustained glucagon-like peptide-1 receptor-dependent control of glucose homeostasis.Caudal brainstem processing is sufficient for behavioral, sympathetic, and parasympathetic responses driven by peripheral and hindbrain glucagon-like-peptide-1 receptor stimulation.Effects of glucagon-like peptide-1, yohimbine, and nitrergic modulation on sympathetic and parasympathetic activity in humans.Effects of glucagon-like peptide-1 and sympathetic stimulation on gastric accommodation in humans.Paying the price for eating ice cream: is excessive GLP-1 signaling in the brain the culprit?Limiting glucocorticoid secretion increases the anorexigenic property of Exendin-4
P2860
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P2860
Glucagon-like peptide-1-responsive catecholamine neurons in the area postrema link peripheral glucagon-like peptide-1 with central autonomic control sites.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
2003年论文
@zh
2003年论文
@zh-cn
name
Glucagon-like peptide-1-respon ...... ntral autonomic control sites.
@en
type
label
Glucagon-like peptide-1-respon ...... ntral autonomic control sites.
@en
prefLabel
Glucagon-like peptide-1-respon ...... ntral autonomic control sites.
@en
P2093
P1476
Glucagon-like peptide-1-respon ...... entral autonomic control sites
@en
P2093
Anthony N Hollenberg
Brian J Choi
Charlotte E Lee
Daniel J Drucker
Hiroshi Yamamoto
Joel K Elmquist
Toshiro Kishi
P304
P356
10.1523/JNEUROSCI.23-07-02939.2003
P407
P577
2003-04-01T00:00:00Z