Removal of the MDCK cell primary cilium abolishes flow sensing.
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A role for Alström syndrome protein, alms1, in kidney ciliogenesis and cellular quiescenceA method for measuring electrical signals in a primary ciliumA polycystin-2 (TRPP2) dimerization domain essential for the function of heteromeric polycystin complexesCilia in vertebrate development and diseaseSensory functions of motile cilia and implication for bronchiectasisEndothelial cilia are essential for developmental vascular integrity in zebrafishMouse models of ciliopathies: the state of the art.Regulation of calcium signaling by polycystin-2Mechanosensing by the primary cilium: deletion of Kif3A reduces bone formation due to loadingCholangiocyte primary cilia are chemosensory organelles that detect biliary nucleotides via P2Y12 purinergic receptorsCholangiocyte cilia detect changes in luminal fluid flow and transmit them into intracellular Ca2+ and cAMP signalingThe native TRPP2-dependent channel of murine renal primary ciliaPrimary cilia mediate mechanosensing in bone cells by a calcium-independent mechanism.The von Hippel-Lindau tumor suppressor protein controls ciliogenesis by orienting microtubule growth.Visualization of live primary cilia dynamics using fluorescence microscopy.Evolutionary origins of sensation in metazoans: functional evidence for a new sensory organ in spongesCilioplasm is a cellular compartment for calcium signaling in response to mechanical and chemical stimuli.The primary cilium functions as a mechanical and calcium signaling nexusTransepithelial pressure pulses induce nucleotide release in polarized MDCK cells.Primary cilia respond to fluid shear stress and mediate flow-induced calcium deposition in osteoblasts.Advances in the pathogenesis and treatment of polycystic kidney diseasePrimary cilia are highly oriented with respect to collagen direction and long axis of extensor tendonStructural and functional analyses of liver cysts from the BALB/c-cpk mouse model of polycystic kidney diseaseIntratubular hydrodynamic forces influence tubulointerstitial fibrosis in the kidney.Polycystic liver diseases.Primary cilium-dependent mechanosensing is mediated by adenylyl cyclase 6 and cyclic AMP in bone cells.Temporal relationship between primary and motile ciliogenesis in airway epithelial cells.Effects of biomechanical forces on signaling in the cortical collecting duct (CCD).Polycystic kidney disease: inheritance, pathophysiology, prognosis, and treatment.Direct demonstration of tubular fluid flow sensing by macula densa cells.Mechanotransduction in the renal tubuleThe versatile nature of the calcium-permeable cation channel TRPP2New insights into ciliary function: kidney cysts and photoreceptors.Cilia involvement in patterning and maintenance of the skeletonDeciphering physiological role of the mechanosensitive TRPV4 channel in the distal nephron.Function and regulation of primary cilia and intraflagellar transport proteins in the skeletonElectrical Signaling in Motile and Primary CiliaChronic fluid flow is an environmental modifier of renal epithelial function.Cilia-like structures and polycystin-1 in osteoblasts/osteocytes and associated abnormalities in skeletogenesis and Runx2 expression.Flow stimulated endocytosis in the proximal tubule.
P2860
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P2860
Removal of the MDCK cell primary cilium abolishes flow sensing.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
2003年论文
@zh
2003年论文
@zh-cn
name
Removal of the MDCK cell primary cilium abolishes flow sensing.
@en
type
label
Removal of the MDCK cell primary cilium abolishes flow sensing.
@en
prefLabel
Removal of the MDCK cell primary cilium abolishes flow sensing.
@en
P1476
Removal of the MDCK cell primary cilium abolishes flow sensing.
@en
P2093
Praetorius HA
P2888
P356
10.1007/S00232-002-1042-4
P577
2003-01-01T00:00:00Z