The p55 tumour necrosis factor receptor TNFR1 contains a trans-Golgi network localization signal in the C-terminal region of its cytoplasmic tail.
about
Arf and Rho GAP adapter protein ARAP1 participates in the mobilization of TRAIL-R1/DR4 to the plasma membraneCaspase-resistant Golgin-160 disrupts apoptosis induced by secretory pathway stress and ligation of death receptorsThe cytoplasmic domain of Vamp4 and Vamp5 is responsible for their correct subcellular targeting: the N-terminal extenSion of VAMP4 contains a dominant autonomous targeting signal for the trans-Golgi networkIncreasing the expression of calcium-permeable TRPC3 and TRPC7 channels enhances constitutive secretionTargeting of tumor necrosis factor receptor 1 to low density plasma membrane domains in human endothelial cells.Pathogenesis of familial periodic fever syndromes or hereditary autoinflammatory syndromes.Tumour necrosis factor receptor trafficking dysfunction opens the TRAPS door to pro-inflammatory cytokine secretion.Abnormal disulfide-linked oligomerization results in ER retention and altered signaling by TNFR1 mutants in TNFR1-associated periodic fever syndrome (TRAPS)Methylation-controlled J protein promotes c-Jun degradation to prevent ABCB1 transporter expression.Dysregulation of innate immunity: hereditary periodic fever syndromes.Hepatitis C Virus Exploits Death Receptor 6-mediated Signaling Pathway to Facilitate Viral Propagation.Human cytomegalovirus infection inhibits tumor necrosis factor alpha (TNF-alpha) signaling by targeting the 55-kilodalton TNF-alpha receptor.Dynamics of RASSF1A/MOAP-1 association with death receptors.Intracellular application of TNF-alpha impairs cell to cell communication via gap junctions in glioma cells.Abnormal tumor necrosis factor receptor I cell surface expression and NF-kappaB activation in tumor necrosis factor receptor-associated periodic syndrome.Tumor necrosis factor receptor I from patients with tumor necrosis factor receptor-associated periodic syndrome interacts with wild-type tumor necrosis factor receptor I and induces ligand-independent NF-kappaB activation.Mutant forms of tumour necrosis factor receptor I that occur in TNF-receptor-associated periodic syndrome retain signalling functions but show abnormal behaviour.Targeting endoplasmic reticulum protein transport: a novel strategy to kill malignant B cells and overcome fludarabine resistance in CLL.
P2860
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P2860
The p55 tumour necrosis factor receptor TNFR1 contains a trans-Golgi network localization signal in the C-terminal region of its cytoplasmic tail.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年学术文章
@wuu
2002年学术文章
@zh-cn
2002年学术文章
@zh-hans
2002年学术文章
@zh-my
2002年学术文章
@zh-sg
2002年學術文章
@yue
2002年學術文章
@zh
2002年學術文章
@zh-hant
name
The p55 tumour necrosis factor ...... egion of its cytoplasmic tail.
@en
type
label
The p55 tumour necrosis factor ...... egion of its cytoplasmic tail.
@en
prefLabel
The p55 tumour necrosis factor ...... egion of its cytoplasmic tail.
@en
P2860
P356
P1433
P1476
The p55 tumour necrosis factor ...... region of its cytoplasmic tail
@en
P2093
Abigail Stewart
J Paul Luzio
P2860
P356
10.1042/BJ20020048
P407
P577
2002-08-01T00:00:00Z