Abscisic acid controls calcium-dependent egress and development in Toxoplasma gondii - Wikidata - wikimedia - TriplyDB

Abscisic acid controls calcium-dependent egress and development in Toxoplasma gondii

Abscisic acid controls calcium-dependent egress and development in Toxoplasma gondii

http://www.wikidata.org/entity/Q40794407

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Hostile takeover by Plasmodium: reorganization of parasite and host cell membranes during liver stage egress What do human parasites do with a chloroplast anyway?Host cell autophagy is induced by Toxoplasma gondii and contributes to parasite growth Intracellular calcium channels in protozoa A Novel Method for Inducing Amastigote-To-Trypomastigote Transformation In Vitro in Trypanosoma cruzi Reveals the Importance of Inositol 1,4,5-Trisphosphate Receptor Distinct external signals trigger sequential release of apical organelles during erythrocyte invasion by malaria parasites T. gondii RP promoters & knockdown reveal molecular pathways associated with proliferation and cell-cycle arrest TgCDPK3 regulates calcium-dependent egress of Toxoplasma gondii from host cells Calcium-dependent permeabilization of erythrocytes by a perforin-like protein during egress of malaria parasites Blockage of spontaneous Ca2+ oscillation causes cell death in intraerythrocitic Plasmodium falciparum Phosphorylation of a Myosin Motor by TgCDPK3 Facilitates Rapid Initiation of Motility during Toxoplasma gondii egress Carotenoid biosynthesis in intraerythrocytic stages of Plasmodium falciparum Externally triggered egress is the major fate of Toxoplasma gondii during acute infection.Calcium-dependent protein kinase 1 is an essential regulator of exocytosis in Toxoplasma.Distinct signalling pathways control Toxoplasma egress and host-cell invasion Malaria parasites tolerate a broad range of ionic environments and do not require host cation remodelling A central role of abscisic acid in stress-regulated carbohydrate metabolism.PDR-type ABC transporter mediates cellular uptake of the phytohormone abscisic acid.Impact of the host on Toxoplasma stage differentiation Problems with using the normal distribution--and ways to improve quality and efficiency of data analysis The malaria parasite progressively dismantles the host erythrocyte cytoskeleton for efficient egress How apicomplexan parasites move in and out of cells.Gibberellin biosynthetic inhibitors make human malaria parasite Plasmodium falciparum cells swell and rupture to death.The toxoplasma apicoplast phosphate translocator links cytosolic and apicoplast metabolism and is essential for parasite survival.Toxoplasma: the next 100years.Multiple roles for Plasmodium berghei phosphoinositide-specific phospholipase C in regulating gametocyte activation and differentiation.Identification of genes expressed during Toxoplasma gondii infection by in vivo-induced antigen technology (IVIAT) with positive porcine sera.Calcium ions are involved in egress of Babesia bovis merozoites from bovine erythrocytes.Antidepressant effects of abscisic acid mediated by the downregulation of corticotrophin-releasing hormone gene expression in rats.Simultaneous administration of 2-aminoethyl diphenylborinate and chloroquine reverses chloroquine resistance in malaria parasites.Plant Hormone Salicylic Acid Produced by a Malaria Parasite Controls Host Immunity and Cerebral Malaria Outcome Mechanisms of Toxoplasma gondii persistence and latency.Malaria invasion ligand RH5 and its prime candidacy in blood-stage malaria vaccine design Novel N-benzoyl-2-hydroxybenzamide disrupts unique parasite secretory pathway.Nitric oxide stimulates early egress of Toxoplasma gondii tachyzoites from Human foreskin fibroblast cells.A genetic screen to isolate Toxoplasma gondii host-cell egress mutants Role of deleted in breast cancer 1 (DBC1) protein in SIRT1 deacetylase activation induced by protein kinase A and AMP-activated protein kinase.Apicomplexan parasites co-opt host calpains to facilitate their escape from infected cells.Nitric oxide-activated calcium/calmodulin-dependent protein kinase regulates the abscisic acid-induced antioxidant defence in maize.Lytic Cycle of Toxoplasma gondii: 15 Years Later.

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Abscisic acid controls calcium-dependent egress and development in Toxoplasma gondii

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2008 nî lūn-bûn

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Abscisic acid controls calcium-dependent egress and development in Toxoplasma gondii

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Blima Fux

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Eduardo N Chini

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Fabien Brossier

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Kisaburo Nagamune

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Leslie M Hicks

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P2860

Inhibitors of the nonmevalonate pathway of isoprenoid biosynthesis as antimalarial drugs

Calcium signalling: dynamics, homeostasis and remodelling

Tropical infectious diseases: metabolic maps and functions of the Plasmodium falciparum apicoplast

Bioluminescence imaging of Toxoplasma gondii infection in living mice reveals dramatic differences between strains.

The temperature-signaling cascade in sponges involves a heat-gated cation channel, abscisic acid, and cyclic ADP-ribose.

A G protein-coupled receptor is a plasma membrane receptor for the plant hormone abscisic acid.

Disruption of the Toxoplasma gondii bradyzoite-specific gene BAG1 decreases in vivo cyst formation.

Elucidation of the indirect pathway of abscisic acid biosynthesis by mutants, genes, and enzymes.

Calcium regulation in protozoan parasites.

Regulation of abscisic acid biosynthesis.

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10.1038/NATURE06478

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451

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1018535470

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18185591

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Toxoplasma gondii

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