Hyperosmolarity reduces GLUT4 endocytosis and increases its exocytosis from a VAMP2-independent pool in l6 muscle cells.
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Insulin-induced cortical actin remodeling promotes GLUT4 insertion at muscle cell membrane rufflesGLUT4 translocation precedes the stimulation of glucose uptake by insulin in muscle cells: potential activation of GLUT4 via p38 mitogen-activated protein kinaseEHD2 and the novel EH domain binding protein EHBP1 couple endocytosis to the actin cytoskeletonClathrin-dependent and independent endocytosis of glucose transporter 4 (GLUT4) in myoblasts: regulation by mitochondrial uncouplingInsulin-regulated aminopeptidase is a key regulator of GLUT4 trafficking by controlling the sorting of GLUT4 from endosomes to specialized insulin-regulated vesiclesA dominant-negative p38 MAPK mutant and novel selective inhibitors of p38 MAPK reduce insulin-stimulated glucose uptake in 3T3-L1 adipocytes without affecting GLUT4 translocation.Focal Adhesion Kinase contributes to insulin-induced actin reorganization into a mesh harboring Glucose transporter-4 in insulin resistant skeletal muscle cellsKinetic evidence for unique regulation of GLUT4 trafficking by insulin and AMP-activated protein kinase activators in L6 myotubesExocytosis mechanisms underlying insulin release and glucose uptake: conserved roles for Munc18c and syntaxin 4A serum factor induces insulin-independent translocation of GLUT4 to the cell surface which is maintained in insulin resistance.Reciprocal regulation of endocytosis and metabolismInsulin-stimulated plasma membrane fusion of Glut4 glucose transporter-containing vesicles is regulated by phospholipase D1.The scavenging of superoxide radicals promotes apoptosis induced by a novel cell-permeable fusion protein, sTRAIL:FeSOD, in tumor necrosis factor-related apoptosis-inducing ligand-resistant leukemia cells.Insulin can regulate GLUT4 internalization by signaling to Rab5 and the motor protein dynein.Enhanced energy expenditure, glucose utilization, and insulin sensitivity in VAMP8 null mice.The many ways to regulate glucose transporter 4.Study on glucose transport in muscle cells by extracts from Mitragyna speciosa (Korth) and mitragynine.GLUT4 activation: thoughts on possible mechanisms.Push/pull mechanisms of GLUT4 traffic in muscle cells.Turning signals on and off: GLUT4 traffic in the insulin-signaling highway.Glucose transporter 4: cycling, compartments and controversies.Electrical stimuli release ATP to increase GLUT4 translocation and glucose uptake via PI3Kγ-Akt-AS160 in skeletal muscle cells.Acute hypertonicity alters aquaporin-2 trafficking and induces a MAPK-dependent accumulation at the plasma membrane of renal epithelial cells.Aerobic exercise training induces metabolic benefits in rats with metabolic syndrome independent of dietary changesSeparation of insulin signaling into distinct GLUT4 translocation and activation steps.Insulin and hypertonicity recruit GLUT4 to the plasma membrane of muscle cells by using N-ethylmaleimide-sensitive factor-dependent SNARE mechanisms but different v-SNAREs: role of TI-VAMP.Measuring GLUT4 translocation in mature muscle fibers.Akt inhibitors reduce glucose uptake independently of their effects on Akt.Depletion of mitochondrial DNA causes impaired glucose utilization and insulin resistance in L6 GLUT4myc myocytes.Maturation of the regulation of GLUT4 activity by p38 MAPK during L6 cell myogenesis.Insulin accelerates inter-endosomal GLUT4 traffic via phosphatidylinositol 3-kinase and protein kinase B.Indinavir uncovers different contributions of GLUT4 and GLUT1 towards glucose uptake in muscle and fat cells and tissues.GLUT4 distribution between the plasma membrane and the intracellular compartments is maintained by an insulin-modulated bipartite dynamic mechanism.A Crk-II/TC10 signaling pathway is required for osmotic shock-stimulated glucose transport.Next-generation Akt inhibitors provide greater specificity: effects on glucose metabolism in adipocytes.Insulin sensitivity and inhibition by forskolin, dipyridamole and pentobarbital of glucose transport in three L6 muscle cell lines.Copper stimulates trafficking of a distinct pool of the Menkes copper ATPase (ATP7A) to the plasma membrane and diverts it into a rapid recycling pool.Regulatory volume increase is associated with p38 kinase-dependent actin cytoskeleton remodeling in rat kidney MTAL.Extracellular hyperosmotic stress stimulates glucose uptake in incubated fast-twitch rat skeletal muscle.Cardiac glycogen accumulation after dexamethasone is regulated by AMPK.
P2860
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P2860
Hyperosmolarity reduces GLUT4 endocytosis and increases its exocytosis from a VAMP2-independent pool in l6 muscle cells.
description
2001 nî lūn-bûn
@nan
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
2001年论文
@zh
2001年论文
@zh-cn
name
Hyperosmolarity reduces GLUT4 ...... ndent pool in l6 muscle cells.
@en
type
label
Hyperosmolarity reduces GLUT4 ...... ndent pool in l6 muscle cells.
@en
prefLabel
Hyperosmolarity reduces GLUT4 ...... ndent pool in l6 muscle cells.
@en
P2093
P2860
P356
P1476
Hyperosmolarity reduces GLUT4 ...... ndent pool in l6 muscle cells.
@en
P2093
P2860
P304
22883-22891
P356
10.1074/JBC.M010143200
P407
P577
2001-04-10T00:00:00Z