Enhanced cell killing by overexpression of dominant-negative phosphatidylinositol 3-kinase subunit, Deltap85, following genotoxic stresses.
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Therapeutic Implications of Targeting AKT Signaling in MelanomaThe Akt signaling pathway: an emerging therapeutic target in malignant melanoma.Functional and therapeutic significance of Akt deregulation in malignant melanoma.The PTEN-AKT3 signaling cascade as a therapeutic target in melanoma.Effects of Ultraviolet Radiation on Glioma: Systematic Review.Targeting protein kinase-b3 (akt3) signaling in melanoma.Rituximab inhibits the constitutively activated PI3K-Akt pathway in B-NHL cell lines: involvement in chemosensitization to drug-induced apoptosis.Similar effects of Brca2 truncation and Rad51 paralog deficiency on immunoglobulin V gene diversification in DT40 cells support an early role for Rad51 paralogs in homologous recombination.Antiapoptotic effects induced by different wavelengths of ultraviolet light.Dietary vitamin E modulates differential gene expression in the rat hippocampus: potential implications for its neuroprotective properties.Analysis of caveolin-1 and phosphoinositol-3 kinase expression in primary uveal melanomas.
P2860
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P2860
Enhanced cell killing by overexpression of dominant-negative phosphatidylinositol 3-kinase subunit, Deltap85, following genotoxic stresses.
description
2000 nî lūn-bûn
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2000年の論文
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name
Enhanced cell killing by overe ...... following genotoxic stresses.
@en
type
label
Enhanced cell killing by overe ...... following genotoxic stresses.
@en
prefLabel
Enhanced cell killing by overe ...... following genotoxic stresses.
@en
P2093
P2860
P1476
Enhanced cell killing by overe ...... following genotoxic stresses.
@en
P2093
P2860
P304
P356
10.1111/J.1349-7006.2000.TB00919.X
P577
2000-12-01T00:00:00Z