Spontaneous development of drug resistance: mismatch repair and p53 defects in resistance to cisplatin in human tumor cells.
about
Azathioprine and UVA light generate mutagenic oxidative DNA damageSeparation of mutation avoidance and antirecombination functions in an Escherichia coli mutS mutantMutS inhibits RecA-mediated strand transfer with methylated DNA substrates.Systems biology of cisplatin resistance: past, present and futureDNA methylation changes in epithelial ovarian cancer histotypes.Msh-2 suppresses in vivo mutation in a gene dose and lesion dependent mannerPaclitaxel resistance in untransformed human mammary epithelial cells is associated with an aneuploidy-prone phenotype.Mismatch repair and treatment resistance in ovarian cancer.NSC109268 potentiates cisplatin-induced cell death in a p53-independent manner.Cellular responses to Cisplatin-induced DNA damage.Mismatch repair in correction of replication errors and processing of DNA damage.DNA-bound platinum is the major determinant of cisplatin sensitivity in head and neck squamous carcinoma cells.α-TEA cooperates with chemotherapeutic agents to induce apoptosis of p53 mutant, triple-negative human breast cancer cells via activating p73.Epigenomics and ovarian carcinoma.Anti-apoptotic brain and reproductive organ-expressed proteins enhance cisplatin resistance in lung cancer cells via the protein kinase B signaling pathway.Reduced expression of small GTPases and hypermethylation of the folate binding protein gene in cisplatin-resistant cells.MiRNA Transcriptome Profiling of Spheroid-Enriched Cells with Cancer Stem Cell Properties in Human Breast MCF-7 Cell Line.Mismatch repair deficiencies transforming stem cells into cancer stem cells and therapeutic implications.MLH1 expression sensitises ovarian cancer cells to cell death mediated by XIAP inhibition.Pharmaco(epi)genomics in ovarian cancer.MutS inhibits RecA-mediated strand exchange with platinated DNA substratesMolecular mechanisms of cisplatin resistance in bladder cancer.DNA damage and repair in human cancer: molecular mechanisms and contribution to therapy-related leukemias.The expression of p21 is upregulated by forkhead box A1/2 in p53-null H1299 cells.The death domain kinase RIP has an important role in DNA damage-induced, p53-independent cell death.Platinum agents in the treatment of osteosarcoma: efficacy of cisplatin vs. carboplatin in human osteosarcoma cell lines.Mechanisms of tolerance to DNA damaging therapeutic drugs.The ability to engage enterocyte apoptosis does not predict long-term crypt survival in p53 and Msh2 deficient mice.Asplatin enhances drug efficacy by altering the cellular response.[Expression and significance of IKBKB in pulmonary adenocarcinoma A549 cells and its cisplatin-resistant variant A549/DDP].DNA damage repair in ovarian cancer: unlocking the heterogeneity.
P2860
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P2860
Spontaneous development of drug resistance: mismatch repair and p53 defects in resistance to cisplatin in human tumor cells.
description
2000 nî lūn-bûn
@nan
2000年の論文
@ja
2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
2000年论文
@zh
2000年论文
@zh-cn
name
Spontaneous development of dru ...... isplatin in human tumor cells.
@en
type
label
Spontaneous development of dru ...... isplatin in human tumor cells.
@en
prefLabel
Spontaneous development of dru ...... isplatin in human tumor cells.
@en
P2093
P2860
P356
P1433
P1476
Spontaneous development of dru ...... isplatin in human tumor cells.
@en
P2093
P2860
P2888
P304
P356
10.1038/SJ.ONC.1203668
P407
P577
2000-06-01T00:00:00Z
P5875
P6179
1039458250